document: To test TrQβ@b-3WJsiRNA miRNA, we silenced two genes to downregulate NF-κB expression capable of inhibiting DNA repair after chemotherapy or radiotherapy. One of them, EGFR, promotes the activation of survival signalling pathways. Increased EGFR activation and overexpression is strongly associated with tumorigenesis, tumour progression, and tumour invasion.44 Recently, it has been reported that nuclear translocation of EGFR also promotes the repair of DSBs after IR. The possible mechanism is