    <button id="show">Show</button>
    <button id="hide">Hide</button>
    <button id="toggle">Toggle</button>
    <br />
    <br />


    <select id="automation" class="premiumSelect">
        <option value="0" selected="selected" data-skip="1">I'm automated</option>
        <option value="1">Option 1</option>
        <option value="2">Option 2</option>
        <option value="3">Option 3</option>
        <option value="4">Option 4</option>
    </select>
</form>
```

You could use the following javascript to have the buttons actuate the drop down
selector:

```javascript
$(document).ready(function(){

   $('button#show').click(function(evt){
      evt.preventDefault();
      evt.stopPropagation();
      $('select#automation').premiumSelect('show');
   });

   $('button#hide').click(function(evt){
      evt.preventDefault();
      evt.stopPropagation();
      $('select#automation').premiumSelect('hide');
   });

   $('button#toggle').click(function(evt){
      evt.preventDefault();
      evt.stopPropagation();
      $('select#automation').premiumSelect('toggle');
   });
});
```

# Creating a Theme
Theming the Premium Select interface is pretty easy.  The basic idea is to add
a subclass of the div.premiumSelect CSS class, that is named after the theme
I'm creating.

If I want to create the 'olive' theme, I would define a CSS class like so:
```css
div.premiumSelect.olive {}
```

Then through that class you can override the various elements of the display. 
For instance if I want to change the background color of the selectBox I would
define the following CSS
```css
div.premiumSelect.olive {}

div.premiumSelect.olive.selectBox {
    background-color: #0000FF;
}
```

Here's a rough outline of the CSS structure for the Premium Select interface:
```css
/* Display wrapper */
div.premiumSelect.theme_name {}

/* Select Box */
div.premiumSelect.theme_name.selectBox {}

/* Drop Down */
div.premiumSelect.theme_name.dropDown {}

/* Drop Down List Item */
div.premiumSelect.theme_name.dropDown li {}

/* Drop Down List Item - First */
div.premiumSelect.theme_name.dropDown li.first {}

/* Drop Down List Item - Last */
div.premiumSelect.theme_name.dropDown li.last {}

/* Drop Down List Item - Hover */
div.premiumSelect.theme_name.dropDown li:hover {}

/* Drop Down List Item - Active */
div.premiumSelect.theme_name.dropDown li.active {}

/* Drop Down List Item Line n 

    n = A line number starting at 1.  Add a CSS definition for each additional
        line.
*/
div.premiumSelect.theme_name.dropDown li span.line-n {}
```We are finding planned grazing with cattle,focusing on increasing animal impact is achieving GRT plant density reductions and increasing density and vigour of more desirable pasture over a very short period of time. Unfortunately council mandated spraying regimes does little to help,as it destroys soil biology and creates bare soil- enabling GRTto dominate.e0083 Membrane type 1 matrix metalloproteinase activation is enhanced by low shear stress through integrin pathway in ApoE-/- mice

Objective Low shear stress and matrix metalloproteinase are involved in atherogenesis and plaque stability. The aim of this study wad to explore the relation and possible mechanism of membrane type-1 matrix metalloproteinase (MT1-MMP) and low shear stress (LSS). Methods 80 male apoE-/- mice were fed a high-fat diet, and lesions in the left carotid artery were induced by perivascular placement of constrictive collars. Results Ultrasound-determined shear stress was significantly lower in the left carotid artery than in the right artery (5.61±0.72 vs 9.76±1.48 N/m2, p<0.01). Confocal microscopy and dual staining revealed higher MT1-MMP expression in left common carotid artery. Cultured human umbilical vein endothelial cells (HUVECs) exposed to 0.4 N/cm2 shear stress (LSS) and 1.2 N/cm2 shear stress (PSS). HUVECs subjected to LSS showed a time-dependent increase in MT1-MMP mRNA level. MT1-MMP mRNA level was downregulated under PSS at 3 and 5 h. MT1-MMP protein showed no change in expression at 1-h LSS, but 3-, 5-, 8- and 16-h treatment produced significantly increased expression and activity. NF-κB DNA-binding activity was stronger at 30-min and 1-h LSS exposure than with control treatment. Pretreatment with 18 μM SN50 efficiently inhibited NF-κB DNA binding activity. In the presence of SN50, MT1-MMP mRNA expression, protein level and activity were significantly attenuated (all p<0.01). MT1-MMP mRNA expression, protein level and activity were inhibited by PD98059 (all p<0.01). To determine the relation between ERK1/2 and NF-κB with MT1-MMP induced by LSS, HUVECs were incubated with PD98059 for 2 h before LSS. The level of phosphor-IKKα/β was reduced, whereas that of phosphor-IκBα was increased as compared with no PD98059 treatment. As well, NF-κB DNA-binding activity was decreased. HUVECs were preincubated for 48 h with FAK siRNA and then treated with LSS. The shear stress-induced increase in MT1-MMP mRNA and protein level and activity was significantly inhibited (all p<0.01). As well, LSS-induced ERK1/2 activation was inhibited with 5-min LSS exposure (p<0.01). To examine NF-κB DNA-binding activity, HUVECs were pretreated for 48 h with FAK siRNA before being exposed to LSS. NF-κB DNA-binding activity was inhibited with FAK siRNA pretreatment. HUVECs were preincubated for 48 h with integrin β1 siRNA and then underwent LSS. The shear stress-induced increase in MT1-MMP mRNA expression, protein level and activity was significantly inhibited (all p<0.01). As well, LSS-induced FAK and ERK1/2 activation was inhibited at 5-min exposure (p<0.01). Next, we examined NF-κB DNA-binding activity with 48-h integrin β1 siRNA pretreatment before exposure to LSS; NF-κB DNA binding activity was inhibited by siRNA integrin β1 pretreatment. Conclusions MT1-MMP induced by LSS is involved in atherosclerotic plaque, via the integrin β1-FAK-ERK1/2-NF-κB pathway.Quite a few of you have tipped us the link (directly or indirectly through post comments) about Stephen Elop being the 7th biggest share holder at Microsoft, whilst holding NO/ZERO/0 stocks at Nokia.This fired up a lot of suspicions that Elop had masterminded some kind of take-over of Nokia by MS, with Nokia effectively being under MS’ thumb as he seemed to stand to gain a lot from it from his previous employer.
Finnish site, YLE (TOP 10 Site in Finland, not just some random site) claims however that the reason Elop has no shares in Nokia is because ofÂ the “terms of security trading regulations” that had prevented him from acquiring Nokia shares due to “insider trading”. Further more, whilst Elop did have significant shares at Microsoft, he began divesting this when he had left the company and sold as much as 60% of it before moving to Nokia.
Elop will apparently divest ALL of his Microsoft shares as soon as restrictions/rules allow, and likewise, purchase Nokia stock when legally able to.
BBC: news or entertainment? Resolve the conflict of interest.
What is the BBC for? That’s the fundamental question thrown up by the disasters the corporation has got itself into. Was the cancellation of a news programme on Jimmy Savile’s reported abuse of young girls influenced by the BBC’s plan to air a celebratory review of his life which was bound to draw in large audience figures?
Quite possibly not, and in light of the BBC’s latest howler over mistaken identity, the journalists working on the Savile exposure may have been right to hold back if they were not sure. But the BBC clearly had a conflict of interests. Should it broadcast a positive programme about Savile because it was bound to be popular as entertainment? Or should it give journalism priority, even if that results in cancellation of the entertainment?
Journalists take it for granted that news has overriding importance. But that is special pleading. Many people are not that interested in the BBC’s news. They have other sources. They are quite