Effector that suppresses plant defense responses during the early stages of pathogen infection (PubMed:21653195, PubMed:26163574, PubMed:30346270). Suppresses cell death induced by effectors and PAMPs in plant hosts (PubMed:26163574, PubMed:30346270). Interacts with host acetyltransferase TAP1 and causes TAP1 relocation into the nucleus where it acetylates histones H2A and H3 during early infection, thereby promoting susceptibility of host plant to P.sojae (PubMed:30346270). Interacts with host acetyl transferase TAP1. Expression is strongly up-regulated during the early stages of infection. The RxLR-dEER motif acts to carry the protein into the host cell cytoplasm through binding to cell surface phosphatidylinositol-3-phosphate. Residues 69 to 86 are required for the interaction with host TAP1 and its re-localization into nuclear speckles. The nuclear localization signal (NLS) is required for localization into nucleus. Leads to smaller lesions on soybean seedlings. Belongs to the RxLR effector family.