Required for pathogen-induced salicylic acid (SA) accumulation and SA-mediated resistance to virulent and avirulent pathogens (e.g. P.syringae). By pathogenic bacteria (e.g. P.syringae) and jasmonic acid (MeJA). Hypersusceptiblity to both virulent and avirulent strains of the bacterial pathogen P.syringae associated with impaired pathogen-mediated induction of salicylic acid (SA) and reduced pathogenesis-related (PR) genes induction. These phenotypes are reversed by SA treatment. In the cv. No-0 but not the cv. Columbia background, defects in SA accumulation or signaling enhances resistance to necrotrophic pathogens such as the fungi B.cinerea and A.brassicicola, leading to small necrotic spots and minor chlorosis, as well as reduced fungal growth. Belongs to the plant acyltransferase family.