Sodium- and chloride-dependent glycine transporter which is essential for regulating glycine concentrations at inhibitory glycinergic synapses. Sodium- and chloride-dependent glycine transporter. chloride(out) + glycine(out) + 2 Na(+)(out) = chloride(in) + glycine(in) + 2 Na(+)(in) chloride(out) + glycine(out) + 2 Na(+)(out) = chloride(in) + glycine(in) + 2 Na(+)(in) Interacts with EXOC1; interaction increases the transporter capacity of SLC6A9 probably by promoting its insertion into the cell membrane (By similarity). Interacts with EXOC3 and EXOC4 (By similarity). Expressed in the brain (at protein level) (PubMed:27773429). At 11 dpc, expressed in the ventral part of the ventricular zone. At 15 dpc, also expressed in adjacent mantle tissue and the meninges. Strongly expressed in 12 dpc and 15 dpc liver. Expressed in the brain. Expression is present at low levels as early as 9 dpc and 10 dpc, but strongly increases at 13 dpc and remains at high levels up to 15 dpc. Also expressed in adult. Mice show severe motor and respiration deficits at birth and die during the first postnatal day (PubMed:14622582). Accumulation of glycine in the synaptic cleft results in over-activation of postsynaptic glycine receptors and death of the newborn animals due to respiratory and feeding problem (PubMed:14622582). Produced by alternative promoter usage. Produced by alternative promoter usage. Produced by alternative splicing of isoform GlyT-1C. Belongs to the sodium:neurotransmitter symporter (SNF) (TC 2.A.22) family. SLC6A9 subfamily.