Negative regulator of reactive oxygen-induced cell death, cold stress-induced cell death, pathogen-induced hypersensitive response (HR), basal disease resistance. May be involved in the induction of the copper/zinc superoxide dismutase CSD1 and CSD2 that detoxify accumulating superoxide before the reactive oxygen species (ROS) can trigger a cell death cascade. LSD1 and LOL1 have antagonistic effects on CSD1 and CSD2 accumulation to regulate oxidative stress-induced cell death. Antagonizes the function of BZIP10, a positive regulator of cell death, by interacting in the cytoplasm and preventing its nuclear localization. Controls lysigenous aerenchyma in hypocotyls under root hypoxia. Required for leaf acclimation in response to excess excitation energy. Interacts with BZIP10 and AMC1 (via N-terminus). Binds to BZIP63. Interacts with CAT1, CAT2 and CAT3 in a zinc-finger-dependent manner (PubMed:23958864). Interacts (via N-terminus) with GILP (PubMed:21526181). Expressed in cotyledons, roots, rosette leaves, stems, inflorescences and flowers. By methyl viologen. No visible phenotype under normal growth condition, however cold treatment induces the development of yellowish leaves with necrosis, and treatment with salicylic acid or infection with avirulent pathogen causes a runaway cell death and plant death. When expressed in Arabidopsis, Pisum sativa LSD1 interacts with importin alpha via the LSD1-type zinc finger motifs, suggesting that the nuclear import of LSD1 may rely on the interaction between its zinc finger motifs and importin alpha. Intron retention.