Mediates sodium-dependent uptake of acetylcholine at neuromuscular junctions during periods of increased synaptic activity, may also prevent spillover to adjacent synaptic sites. Not involved in the uptake of other neurotransmitters (GABA, glycine, proline and glutamate) and there was also no inhibition of uptake by adding an excess of other candidate substrates (GABA, glycine, taurine, creatine, proline, alanine, carnitine, glutamate and betaine). Required for muscle integrity; altered transport of acetylcholine due to loss of dystrophin-glycoprotein complex (DGC) function results in muscle degeneration. Interacts with stn-1; part of the DGC. The DGC is not necessary to establish snf-6 at the postsynaptic membrane, but it is required for maintenance or stabilization. Body wall, and vulval and enteric muscles. Belongs to the sodium:neurotransmitter symporter (SNF) (TC 2.A.22) family.