Secreted immune-induced peptides induced by Toll signaling (PubMed:9736738, PubMed:34432851). Has a significant role in resistance to infection by the entomopathogenic fungus B.bassiana R444 and weak antifungal activity against M.rileyi PHP1705 (PubMed:34432851). In adult males, activity appears to be important for neuromuscular processes that mediate correct wing posture upon Toll activation (PubMed:34432851). Hemolymph (at protein level). Hemolymph (at protein level). Hemolymph (at protein level). Hemolymph (at protein level). Hemolymph (at protein level). Hemolymph (at protein level). Hemolymph (at protein level). Hemolymph (at protein level). By bacterial infection (at protein level) (PubMed:9736738, PubMed:16510152). Detected within 24 hours of infection (PubMed:9736738). Up-regulated in the fat body following infection with M.luteus, with expression levels increasing from 2 to 48 hours post infection (PubMed:34432851). In adults, also up-regulated in the head including the border of the eyes and the ocelli, and in the brain tissue including the region posterior to the central brain furrow (PubMed:34432851). Up-regulated in the wing veins, along the borders of the thoracic pleura, and in the spermatheca of females (PubMed:34432851). In larvae, induced in the brain tissue at the posterior of the ventral nervous system (PubMed:34432851). Proteolytically cleaved. Viable with no obvious morphological defects, however flies have an increased susceptibility to entomopathogenic fungi (PubMed:34432851). Displays increased fungal load 48 hours after infection with B.bassiana R444 spores resulting in a significant decrease in survival (PubMed:34432851). Survival is also slightly decreased following septic injury with M.rileyi PHP1705 (PubMed:34432851). Some adult males display an erect wing phenotype upon infection with Gram-positive bacteria, fungi, and to a lesser extent, Gram-negative bacteria and clean injury (PubMed:34432851). Males also display the erect wing phenotype after injury with heat-killed E.faecalis suggesting that the phenotype occurs due to Toll activation and is not the result of infection (PubMed:34432851). No effect on activation of the Toll or imd/NF-kappa-B signaling cascades in response to microbial infection (PubMed:34432851). No significant affect on lifespan, resistance to bacterial infection and survival following clean injury (PubMed:34432851). In some wild flies and common laboratory strains, the BaraA locus has undergone a duplication event to produce two identical paralogs, BaraA1 and BaraA2 (PubMed:34432851). However BaraA copy number varies within these strains indicating that the duplication event is recent and not fixed (PubMed:34432851). The name 'Baramicin' derives from the Japanese idiom Bara-Bara, meaning to break apart, and refers to the multiple peptides produced from the precursor protein.