Transcriptional activator activated by proteolytic cleavage through regulated intramembrane proteolysis (RIP) (By similarity). Transcriptional activator that promotes leaf senescence by up-regulating senescence-associated genes in response to developmental and stress-induced senescence signals. Functions in salt and oxidative stress-responsive signaling pathways. Binds to the promoter of NAC029/NAP and NAC059/ORS1 genes (PubMed:23926065). Localized primarily in plasma membrane or endoplasmic reticulum membrane as dormant form and, upon specific stress or signal, is processed into a transcriptionally active and nuclear form after a proteolytic cleavage through regulated intramembrane proteolysis (RIP). Expressed in roots, rosette leaves, shoot apex, stems and flowers. Induced during normal senescence. By cold, salt, drought stress and methyl methanesulfonate (MMS) treatment. The NAC domain includes a DNA binding domain and a dimerization domain. Stay-green phenotype during senescence, salt stress and oxidative stress.