Probably plays a role in ribosome assembly or function; overexpression suppresses cold-sensitive growth of a bipA deletion (Ref.10) (Probable). May be involved in resolution of branched DNA intermediates that result from template switching in postreplication gaps. Binds DNA at Holliday junctions. May be involved in the correct segregation of nucleoids (PubMed:31665437). Has ATPase activity, binds DNA non-sequence specifically; the presence of DNA does not change the ATPase activity (PubMed:16407313). Mutations in this gene cause an increase in RecA-independent precise excision of transposons and insertion elements, and also reduce bacteriophage Mu growth (PubMed:6294054, PubMed:16407313, PubMed:19948254). ATP + H2O = ADP + H(+) + phosphate ATPase activity inhibited by N-ethylmaleimide but not by vanadate. Associates with ribosomes. Present from early exponential to stationary phase in equal amounts (at protein level). The C-terminal domain (CTD) helps bind DNA, is required to complement a gene deletion. Increase in the frequency of precise transposon excision (PubMed:9139905, PubMed:16407313, PubMed:19948254). No visible growth phenotype at 37 or 18 degrees Celsius. A double bipA-uup deletion does not grow at 18 degrees Celsius, i.e. the uup deletion exacerbates the bipA deletion (Ref.10). Deletion leads to an increase in DNA crossing over, DNA repeat deletion and DNA repeat expansion; double uup-radD deletion increases the phenotypes. Although single uup deleted cells replicate normally, they are filamentous and lack defined nucleoids, again exacerbated by a radD deletion (PubMed:31665437). Belongs to the ABC transporter superfamily. ABCF family. Uup subfamily.