Part of the ABC transporter FtsEX involved in asymmetric cellular division facilitating the initiation of sporulation. Interacts with FtsE. Vegetative bacilli mutant cells exhibit a distinct morphology in which individual bacilli appear kinked with frequent curves and sharp angles among chains of bacilli. Grows more slowly than wild-type bacteria. Affords resistance of vegetative B.anthracis bacilli to human chemokine CXCL10 regardless of their growth phase and to murine Cxcl9-mediated bactericidal activity. CXCL10 treated spores fail to germinate and resume vegetative growth indicating that FtsX is not required for the ability of CXCL10 to target the spore form of B.anthracis. In the absence of FtsX, CXCL10 is unable to localize to its presumed site of action at the bacterial cell membrane. No statistically significant differences are observed between wild-type and deletion mutant bacilli susceptibility to cationic antimicrobial peptide protamine. Potential therapeutic target of chemokine-mediated antimicrobial activity against B.anthracis to treat infection. Belongs to the ABC-4 integral membrane protein family. FtsX subfamily.