Required for the establishment and maintenance of stable cell fate in several lineages including V5.pa, T, Z1/Z4 and QR lineages probably by repressing the expression of cell fate determinants (PubMed:20181741, PubMed:24346701). Required to maintain non-distal tip cell (DTC) fate of somatic gonadal cells through the htz-1-mediated repression of transcription factor ceh-22. Regulates the subnuclear localization of histone variant htz-1 in somatic gonadal cells (PubMed:24346701). Plays a role in the attenuation of the let-60/ras pathway, probably by preventing expression of activators of the pathway (PubMed:24285704, PubMed:24349540). Involved in adult locomotion. Acts together with the sumoylation pathway to prevent muscle myosin depletion in aging adults probably by preventing myoblast growth factor receptor egl-15 overexpression (PubMed:24285704). May play a role in vulva development (PubMed:24349540). Interacts with acetylated histone H4 (PubMed:20181741). Interacts (via BROMO domain 2) with smo-1 and ubc-9 (PubMed:24349540). Localizes to granular structures in interphase and co-localizes with chromosomes during metaphase. Expressed in T-cells, Q-cells, V5-cells and their descendants such as somatic gonad and syncytium. The BROMO domain 2 is essential for the interaction with smo-1 and E2 enzyme ubc-9. Abnormal cell fate determination of several lineages. T.p daughter cells adopt a hypodermal cell fate instead of a neuronal cell fate resulting in the loss of the phasmid socket cell. Generation of ectopic PDE, AVM and PVM neurons, and distal tip cell (DTC) (PubMed:20181741, PubMed:24346701). In addition, ectopic egl-17 expression in multiple vulva precursor cells and ectopic che-22 expression in extra distal tip cells (PubMed:24349540, PubMed:24346701). Moderate increase in mpk-1 phosphorylation (PubMed:24349540). RNAi-mediated knockdown further suppresses the age-dependent paralysis phenotype of the spop-1 gk630214 or dr95 mutants (PubMed:34593637).