MFS transporter; part of the gene cluster that mediates the biosynthesis of cercosporin, a light-activated, non-host-selective toxin (PubMed:17250832, PubMed:17462021, PubMed:26938470). The perylenequinone chromophore of cercosporin absorbs light energy to attain an electronically-activated triplet state and produces active oxygen species such as the hydroxyl radical, superoxide, hydrogen peroxide or singlet oxygen upon reaction with oxygen molecules. These reactive oxygen species cause damage to various cellular components including lipids, proteins and nucleic acids (PubMed:11701851). Responsible for secretion and accumulation of cercosporin, but does not play any roles in self-protection against the toxicity of cercosporin (PubMed:17250832). Expression is positively regulated by the cercosporin cluster-specific transcription factor CTB8 (PubMed:17462021). Expression is also affected by nitrogen and carbon sources and pH, and is also controlled by another transcription activator, CRG1, previously shown to regulate cercosporin production and resistance (PubMed:17462021). Reduces cercosporin toxin accumulation and fungal virulence. Belongs to the major facilitator superfamily. CAR1 family.