RNA-binding protein that acts as a regulator of alternative pre-mRNA splicing (PubMed:26404089, PubMed:28971960). Negative regulator of responses to abscisic acid (ABA), including in early development (PubMed:26404089). Specifically associates with functional splicing complexes (By similarity). Associates with exon junction complex (EJC) proteins (By similarity). Induced by abscisic acid both at transcriptional and at post-translational (pre-mRNA splicing) levels. The PWI domain binds nucleic acids with significant help from its N-terminal flanking basic region. It has an equal preference for binding to single- or double-stranded species, and it contributes to RBM25 role in modulation of alternative splicing. Phosphorylated; the phosphorylation level is repressed by abscisic acid (ABA). Increased sensitivity to growth inhibition by abscisic acid (ABA) (PubMed:26404089). Altered transcript accumulation profiles due to impaired pre-messenger RNA (pre-mRNA) splicing (e.g. HAB1 transcripts alternative splicing) (PubMed:26404089, PubMed:28971960). These phenotypes are partially suppressed by an ectopic expression of HAB1 (PubMed:26404089).