P granule component, which acts redundantly with P granule component meg-1 to promote P granule segregation during embryogenesis, and germ cell proliferation and differentiation in larval stages (PubMed:18202375, PubMed:21305687, PubMed:25535836). Together with phosphorylated meg-1, promotes the disassembly of zygotic P granules in the anterior cytoplasm of pre-gastrulation embryos (PubMed:25535836). Together with dephosphorylated meg-1, promotes the assembly and accumulation of zygotic P granules in the posterior cytoplasm of pre-gastrulation embryos (PubMed:25535836). Required for fertility (PubMed:18202375, PubMed:21305687). Localizes to P granules in embryogenesis. Expressed in the P lineage during embryogenesis (PubMed:18202375). Expressed at the 4- to 8-cell stage, and expression remains throughout the remaining P cell divisions in P2, P3 and P4 cells, and subsequently in primordial germ cells Z2 and Z3 (PubMed:18202375). RNAi-mediated knockdown results in sterility, and in P granule segregation defects (PubMed:18202375). RNAi-mediated knockdown increases the sterility phenotype of meg-1 vr10 and meg-1 vr11 mutants from 15 to 100% and from 4 to 93%, respectively, and results in a larger proportion of under-proliferated gonads than in the single meg-1 mutants (PubMed:18202375, PubMed:21305687, PubMed:25535836). Simultaneous RNAi-mediated knockdown of meg-1 and meg-2 results in defects in P granule disassembly in the anterior cytoplasm of the P1 blastomere causing some P granules to be mis-segregated to somatic blastomeres (PubMed:25535836).