Membrane anchor for FtsZ. Binds and recruits FtsZ polymers to membranes early in the cell cycle. May also improve the efficiency of cytokinesis through the regulation of cell wall hydrolysis. Interacts with FtsZ filaments. Associates with membranes directly (PubMed:27028265). Colocalizes with FtsZ at the division site (PubMed:20864042). Deletion mutant grows with normal rates, morphology and Z-ring organization (PubMed:20864042, PubMed:27028265). In cells lacking other non-essential division genes implicated in cell wall hydrolysis, such as dipM, ftsE or amiC, deletion of fzlC causes synthetic cytokinesis defects (PubMed:27028265).