Transcription factor that seems to function as negative regulators of virulence by affecting the capacity to damage host cells and to stimulate a pro-inflammatory response. Expression is regulated by the AcuK and AcuM transcription factors. Results in increased resistance to the chitin synthesis inhibitor nikkomycin Z and causes increased damage to pulmonary epithelial cells and vascular endothelial cells, as well as increased pulmonary epithelial cell cytokine gene expression.