Receptor kinase involved in the perception of C-terminally encoded plant signaling peptide (CEP) and subsequent regulation of root and shoot development. Together with CEPR1, mediates systemic nitrogen (N)-demand signaling upon the perception of root-derived peptides (e.g. CEP1) via the up-regulation of genes involved in N uptake and assimilation pathways. ATP + L-tyrosyl-[protein] = ADP + H(+) + O-phospho-L-tyrosyl-[protein] Interacts with the root-derived peptide CEP1 (PubMed:25324386). Binds to the ammonium transporter AMT1-1 (PubMed:21423366). Expressed in mature leaves, primary roots, and the root tips of both primary and lateral roots. The double mutant cepr1 cepr2 is insensitive to CEP1 in a root growth regulation and exhibit pleiotropic phenotype characterized by pale-green leaves and enhanced lateral root elongation. At adult stage, smaller rosette leaves and shorter floral stems, accompanied by anthocyanin accumulation. Down-regulation of genes involved in N uptake and assimilation pathways (e.g. NRT1.1, NRT2.1 and NRT3.1) leading to impaired nitrate uptake activity. Altered systemic induction of genes involved in N uptake and assimilation pathways in N-depletion conditions (PubMed:25324386). Increased resistance to osmotic stress (e.g. mannitol) (PubMed:21431781). Belongs to the protein kinase superfamily. Ser/Thr protein kinase family.