Transcription factor that functions as repressor of flowering by enhancing the expression of several genes that delay flowering including FLC, FLM/MAF1, MAF2 and SVP (PubMed:15659097). Also acts in the floral homeotic AGAMOUS (AG) pathway, specifically by processing the AGAMOUS pre-mRNA (PubMed:10198637, PubMed:12530963). Functions in association with HUA1 and HEN4 in AG pre-mRNA processing (PubMed:12530963). Involved in all three aspects of the AG functions, the specification of stamen and carpel identities, the control of floral determinacy, and the spatial restriction of AP1 expression (PubMed:10198637, PubMed:12530963, PubMed:15659097). Acts as transcription regulator that controls anthocyanin accumulation (PubMed:25425527). Expressed in the inflorescence meristem, floral primordia, inflorescence stem, and floral pedicels. Also detected in the shoot apical meristem, stems, leaves, embryos, and roots. Hua2 mutations enhance the phenotype of mild ag-4 and elf4 alleles and also induce early flowering by reducing the expression of several MADS genes that act as floral repressors like FLC, FLM/MAF1, MAF2 and SVP. In cv. Sy-0, a naturally occurring allele K525E, termed ART1, together with active FRI and FLC alleles causes delayed flowering of primary and axillary meristems, resulting in the distinctive Sy-0 morphology (PubMed:17764945).