Component of an exocyst subcomplex specifically involved in autophagy-related, Golgi-independent membrane traffic to the vacuole. Regulates autophagosome formation and autophagy-related Golgi-independent import into the vacuole (By similarity). Positive regulator of defense responses to pathogenic bacteria (e.g. P.syringae pv. maculicola), to the biotrophic oomycete H.arabidopsidis and to fungi (e.g. B.graminis hordei), especially in cell wall apposition formation related to plant defense (PubMed:21199889, PubMed:23170036). Required for both immediate and later responses triggered by pathogen-associated molecular patterns (PAMPs) (PubMed:23170036). Positive regulator of abscisic acid (ABA)-independent mannitol (drought)-promoted stomatal closure (PubMed:27956469). Self interacts (PubMed:21199889). Interacts with EXO70B1 (PubMed:23944713). Interacts with the exocyst subunits EXO70H1, SEC5A and SEC15B. Binds to SNAP33 (PubMed:21199889). Subunit of the exocyst complex that mediates vesicle tethering during exocytosis (Probable). Binds to PUB22 (PubMed:23170036). In the cytoplasm, localized in a peri-nuclear pattern. Mostly expressed in leaves and, to a lower extent, in roots, cotyledons, internodes, flower buds, siliques and anthers. Induced by elicitor peptides elf18 and flg22, by the fungus B.graminis hordei and by the bacterium P.syringae. Target of the E3 ubiquitin-protein ligase PUB22 that mediates its ubiquitination and degradation via the 26S proteasome to attenuate pathogen-associated molecular patterns (PAMP)-induced signaling, especially is response to the bacterial elicitor flg22. No discernible phenotype in normal conditions (PubMed:16942608, PubMed:21199889, PubMed:27956469). Altered mannitol (drought)-promoted stomatal closure (PubMed:27956469). Enhanced susceptibility to the bacterial pathogen P.syringae pv. maculicola and abnormal papilla formation, with an unusual wide halo made of vesicle-like structures upon inoculation by the fungal pathogen B.graminis hordei (PubMed:21199889). Reduced sensitivity to pathogen-associated molecular patterns (PAMPs) (e.g. bacterial elicitor flg22, bacterial transcription factor elf18, Pep1 and fungal chitin) leading to a reduced production of reactive oxygen species (ROS) and impaired induction of several plant defense genes. Increased disease susceptibility to the biotrophic oomycete H.arabidopsidis (PubMed:23170036). Belongs to the EXO70 family.