Monooxygenase that converts the endogenous (and likely the host) jasmonate (JA) to its hydroxylated derivative 12-hydroxyjasmonate (12OH-JA), also known as tuberonic acid, a compound that attenuates or disables jasmonate-based host innate immunity and which is essential for proper initiation and elaboration of the blast disease in rice (PubMed:26258762). ABM, together with a polyketide synthase MGG_04775 and the esterase MGG_04774, share the secondary metabolism gene cluster with ABC transporter ABC3, and therefore may also be involved in the synthesis of other important metabolites such as the ABC3 transporter efflux substrate (ATS) and/or additional polyketides (Probable). H(+) + jasmonate + NADPH + O2 = (1R,2R)-12-hydroxyjasmonate + H2O + NADP(+) Associates with the cortical endoplasmic reticulum (ER) network during pre-invasive pathogenic development on the host surface pathogenesis and remains associated with the cortical ER in the invasive hyphae untill the later stages of host colonization (PubMed:26258762). Associates also, albeit weakly, with the biotrophic interfacial complex (BIC), a specialized secretory organelle present on the invasive hyphae in M.oryzae (PubMed:26258762). Secreted specifically during invasive growth (PubMed:26258762). Fails to develop blast disease on the rice host but does not affect vegetative growth, asexual development and appressorium formation (PubMed:26258762). Significantly reduces the host penetration ability (PubMed:26258762). Leads to the accumulation of a methylated form of jasmonate (MeJA) resulting in a strong induction of the host defense response in an otherwise susceptible rice plant (PubMed:26258762). Does not affect the ability to cause blast disease in barley (PubMed:26258762).