Transcriptional activator (By similarity). Shown to have a role in the negative regulation of exit from L1 arrest and dauer diapause dependent on IIS signaling (insulin and insulin-like growth factor (IGF) signaling). Hypodermal expression is regulated by IIS/daf-16 while neuronal expression is not under the control of IIS/daf-16. Expressed in hermaphrodite dopaminergic neurons (ADE, CEP, and PDE). Expressed throughout development and adulthood but abundance declines with age. Induced by daf-16. Faster exit from L1 arrest and IIS-dependent dauer diapause.