Auxin response factors (ARFs) are transcriptional factors that bind specifically to the DNA sequence 5'-TGTCTC-3' found in the auxin-responsive promoter elements (AuxREs). Acts as a transcriptional activator of several tropic stimulus-induced (TSI) genes, including SAUR50. Formation of heterodimers with Aux/IAA proteins may alter their ability to modulate early auxin response genes expression. Required for differential growth responses of aerial tissues. Involved in ethylene responses. Regulates lateral root formation through direct regulation of LBD16 and/or LBD29 (PubMed:29184030). Functionally redundant with ARF19 (PubMed:29184030). Mediates embryo axis formation and vascular tissues differentiation. Functionally redundant with ARF5. Involved in cellular dedifferentiation during callus formation on callus-inducing medium (CIM) and in an ATXR2-dependent manner (PubMed:29184030). Homodimers and heterodimers (PubMed:10476078, PubMed:21734647). Interacts with the auxin-responsive proteins IAA1 and IAA12 (BODENLOS)(PubMed:14973283, PubMed:21734647). Interacts (via PB1 domain) with IAA17 (via PB1 domain) (PubMed:24706860, PubMed:21734647). Interacts with IAA19 (PubMed:14729917, PubMed:21734647). Interacts with ARF5 (PubMed:17259263, PubMed:21734647). Binds to JMJ30 (PubMed:29923261). Binds to ATXR2 in the nucleus (PubMed:29184030). Expressed in the whole plant. The PB1 domain provides both positive and negative electrostatic interfaces for directional protein interaction. The arf7-1 arf19-2 double mutant is defective in callus formation. May be due to a competing donor splice site. Incomplete sequence. May be due to exon skipping. Belongs to the ARF family.