BrlA, abaA and wetA are pivotal regulators of conidiophore development and conidium maturation (PubMed:17030990). They act individually and together to regulate their own expression and that of numerous other sporulation-specific genes (By similarity). Binds promoters of target genes at brlA response elements (BREs) containing the conserved sequence 5'-(C/A)(A/G)AGGG(G/A)-3' (By similarity). Positively regulates expression of the gliotoxin biosynthetic gene cluster in actively growing vegetative cells, and likely bridges morphological and chemical development during the life-cycle (PubMed:26032501). Regulates (directly or indirectly) the ergot cluster genes (PubMed:18333504). Positively regulates expression of the fumiquinazoline C biosynthetic gene cluster (PubMed:24612080). Positively regulates expression of the melanin biosynthetic gene cluster (PubMed:24123270). Mediates repression of ribosomal protein gene expression in response to nitrogen depletion (PubMed:19028996). Expression increases dramatically during the synchronous asexual development (PubMed:18298443). Expression is positively regulated by hsp90 (PubMed:22822234). Expression is controlled by upstream regulators fluG and flbA (PubMed:17030990). Expression is controlled by the histone methyltransferase clrD (PubMed:18849468). Expression is decreased by 2',4'-Dihydroxychalcone (2',4'-DHC) (PubMed:26190922). Abolishes completely conidiation (PubMed:17030990). Results in production of bristle-like structures instead of conidiophores and conidia and fails to produce ergot alkaloids (PubMed:18333504). Reduces significantly expression of gliotoxin biosynthetic gene cluster including gliM, gliP, gliT, and gliZ (PubMed:26032501). Impairs down-regulation of genes encoding ribosomal proteins under nitrogen-limiting, but not carbon-limiting, conditions (PubMed:19028996). Abolishes the production of fumiquinazoline C (PubMed:24612080).