Required for male meiotic cytokinesis through its involvement in the regulation of mitochondrial aggregation and fusion, astral spindle assembly and contractile ring formation. Mutants are viable and females are fully fertile. Males show premature mitochondrial aggregation and fusion in spermatocytes and spermatids, leading to an aberrant mitochondrial shell around premeiotic nuclei. Improper mitochondrial localization occurs in the testis associated with defective central astral spindles and a lack of contractile rings which leads to meiotic cytokinesis failure.