Plays a critical role in cytoplasmic virus egress. Participates in the final step of tegumentation and envelope acquisition within the host cytoplasm by directly interacting with the capsid. Upon virion binding to target cell, a signaling cascade is triggered to disrupt the interaction with the capsid, thereby preparing capsid uncoating (By similarity). Additionally, antagonizes the viral DNA-triggered antiviral immune response by targeting host STING1 and preventing its dimerization and trafficking (PubMed:32238587). Interacts with cytoplasmic envelopment protein 3 and with the capsid (By similarity). Interacts with host STING1; this interaction prevents viral DNA-triggered antiviral immune response (PubMed:32238587). Localizes in the host nucleus up to 18 hours postinfection, but at later times localizes to punctate, cytoplasmic structures. UL94 deficiency mediates HCMV-induced transcription of type I IFNs and downstream antiviral effectors leading to impaired viral replication. Belongs to the herpesviridae cytoplasmic envelopment protein 2 family.