Acts as a gating modifier on both Kv and Nav ion channels, and also acts on blood pressure (By similarity). Voltage-dependently inhibits voltage-gated potassium channels Kv3 (Kv3.1/KCNC1, Kv3.2/KCNC2 and Kv3.4/KCNC4) and slows inactivation of the voltage-gated sodium channel Nav1.7/SCN9A (By similarity). Inhibits all Kv3.1, Kv3.2 and Kv3.4 by about 50% when tested at a voltage of +40 mV (45%, 48% and 56%, respectively) (By similarity). May act by binding residues in voltage-sensing domains S3b and S4 of Kv3 (By similarity). On sodium channels, tests have been done on human Nav1.7/SCN9A (expressed in HEK293 cells) (EC(50)=3 nM) and rat SCG neurons that mostly carry Nav1.7 channels (EC(50)=300 nM) (By similarity). This toxin also reduces blood pressure (By similarity). Highly expressed in the ectodermal tissue from the distal and proximal tentacles, body wall, and oral disk. Is the most represented component of the BDS family in A.viridis. This protein sequence is identical to BDS-1 from A.sulcata (AC P11494). Belongs to the sea anemone type 3 (BDS) potassium channel toxin family. Opinions are divided on whether Anemonia viridis (Forsskal, 1775) and Anemonia sulcata (Pennant, 1777) are separate species.