Probable lipid transfer protein (LTP). Seems to control the flowering process and lignin synthesis. Together with DIR1, required for glycerol-3-phosphate- (G3P) and azelaic acid- (AA) induced systemic acquired resistance (SAR). Component of plant systemic immunity involved in priming defenses in a AA-dependent manner, by modulating production and/or translocation of a mobile signal(s) during SAR. Confers resistance to Botrytis cinerea and Pseudomonas syringae pv. tomato DC3000 and PmaDG3. May be involved in induced systemic resistance (ISR) mediated by non-pathogenic bacteria (e.g. P. fluorescens GM30). Prevents electrolyte leakage during freezing damage. Self-interacts and binds to DIR1 (PubMed:23602565). Interacts with PDLP1 (PubMed:27078071). Chloroplast location is observed in the absence of PLDP1 or PDLP5. Transient accumulation in response to a brief exposures to cold. Induced by glycerol-3-phosphate (G3P) and azelaic acid (AA) during systemic acquired resistance (SAR) and Pseudomonas fluorescens GM30 strain during induced systemic resistance (ISR). Reduced cutin accumulation due to lower cutin biosynthesis. Early flowering in long-day conditions. Compromised pathogen-induced (e.g. Pseudomonas syringae pv. tomato DC3000 and PmaDG3) glycerol-3-phosphate- (G3P) and azelaic acid-(AA) dependent systemic acquired resistance (SAR). Increased tendencies in cellular damage after freezing treatment. Belongs to the plant LTP family. PEARLI1 subfamily.