In the beginning Taipoxin was thought to be only neurotoxic. Studies showed an increase in acetylcholine release, indicating a presynaptic activity. Further experiments showed that Taipoxin inhibited the responses to electrical stimuli greater than the reaction to additionally administered acetylcholine. This led to the conclusion that Taipoxin has pre- and postsynaptic effects. Additional to the increased acetylcholine release it inhibits the vesicular recycling. More recent studies showed that the toxin has a myotoxic effect as well. The injection of Taipoxin into the hind limbs of rats leads to oedema formation and muscle degeneration. The study also supports the findings by Fohlman, that the α subunit yields the PLA2 potency, which is similar to the potency of notexin. Even so, the full potential of the raw toxin is only reached by the combination of the α and γ subunits.