Understanding the interactions of microbial communities present on amphibians' skin with fungal species in the environment can reveal why certain amphibians, such as the frog "Rana muscosa", are susceptible to the fatal effects of "B. dendrobatidis" and why others, such as the salamander "Hemidactylium scutatum", are able to coexist with the fungus. As mentioned before, the antifungal bacterial species "Janthinobacterium lividum", found on several amphibian species, has been shown to prevent the effects of the pathogen even when added to another amphibian that lacks the bacteria ("B. dendrobatidis"-susceptible amphibian species). Interactions between cutaneous microbiota and "B. dendrobatidis" can be altered to favor the resistance of the disease, as seen in past studies concerning the addition of the violacein-producing bacteria "J. lividum" to amphibians that lacked sufficient violacein, allowing them to inhibit infection. Although the exact concentration of violacein (antifungal metabolite produced by "J. lividum") needed to inhibit the effects of "B. dendrobatidis" is not fully confirmed, violacein concentration can determine whether or not an amphibian will experience morbidity (or mortality) caused by "B. dendrobatidis". The frog "Rana muscosa", for example, has been found to have very low concentrations of violacein on its skin, yet the concentration is so small, it is unable to facilitate increased survivability of the frog; furthermore, "J. lividum" has not been found to be present on the skin of "R. muscosa". This implies that the antifungal bacterium "J. lividum" (native to other amphibians' skin, such as "Hemidactylium scutatum") is able to produce a sufficient amount of violacein to prevent infection by "B. dendrobatidis" and allow coexistence with the potentially deadly fungus.