Consistent risk factors include: *Severity of seizures, increased refractoriness of epilepsy and presence of generalized tonic–clonic seizures: the most consistent risk factor is an increased frequency of tonic–clonic seizures. *Poor compliance. Lack of therapeutic levels of anti-epileptic drugs, non-adherence to treatment regimens and frequent changes in regimens are risk factors for<unk><unk> . *Young age and early age of seizures onset *Male sex *Being asleep during a seizure is likely to favour<unk>  occurrence. Genetic mutations have been identified that increase a person's risk for<unk>  (some are discussed below), but ultimately their genetic risk is determined by the function of multiple genes that is not yet well understood. Overlap is seen between these ion channel genes and the different<unk><unk>  disorders, including<unk> , SIDS,<unk><unk><unk>  (SUD), and<unk><unk><unk>  in Youth (SUDY). Many of the genes are involved in Long QT syndrome. * Mutations in the KCNQ1 gene that codes for the voltage-gated potassium channel KV7.1 have been implicated in cardiac arrhythmias, such as Long QT syndrome 1 (LQT1), and epilepsy. * Mutations in potassium channel gene KCNH2 have been identified with LQT2. * Mutations in sodium channel gene SCN5A have been identified with LQT3. * Mutations in potassium channel gene KCNJ2 have been identified with LQT7. * Mutations in calcium channel gene CACNA1C have been identified with LQT8. * The sodium ion channel genes SCN1A, SCN1B, SCN2A, and SCN8A and the potassium channel KCNA1 have been implicated in both epilepsy and<unk> .
Sudden unexpected death in epilepsy
Febrile infection-related epilepsy syndrome
Photosensitive epilepsy
Northern epilepsy syndrome
Musicogenic epilepsy
Hypertensive emergency
Generalized epilepsy
Post-traumatic epilepsy