The mechanism of<unk>  is a result of an abnormal induction of metanephric mesenchyme. This could be a result of a formation difficulty of the mesonephric duct. Some mutations in genes associated with renal dysplasia (in syndromes) have been determined. The mutations in question occur at EYA1 or SIX1 genes (branchio-oto-renal syndrome). The PAX2 gene is also thought to play a role in<unk> . The contralateral kidney often undergoes hypertrophy. This is believed to be a compensatory mechanism to the non-functional<unk> . About 90% of patients with an<unk>  will have contralateral hypertrophy into adulthood. The impact of contralateral hypertrophy on long-term renal outcomes is unknown.
Multicystic dysplastic kidney
Renal segmental hypoplasia
Page kidney
Horseshoe kidney
Crossed renal ectopia
Ectopic kidney
Supernumerary kidney
Medullary sponge kidney