Short-term outcomes<unk>  is often associated with a poor outcome. The death rate (mortality) for<unk>  is between 40 and 50 percent, but trends for survival are improving. Of those that survive hospitalization, more than a quarter have significant restrictions in their lifestyle, and less than a fifth have no residual symptoms whatsoever. Delay in diagnosis of minor<unk>  (mistaking the sudden headache for migraine) contributes to poor outcome. Factors found on admission that are associated with poorer outcome include poorer neurological grade; systolic hypertension; a previous diagnosis of heart attack or<unk> ; liver disease; more blood and larger aneurysm on the initial CT scan; location of an aneurysm in the posterior circulation; and higher age. Factors that carry a worse prognosis during the hospital stay include occurrence of delayed ischemia resulting from vasospasm, development of intracerebral hematoma, or intraventricular<unk>  (bleeding into the ventricles of the brain) and presence of fever on the eighth day of admission. So-called "angiogram-negative<unk> ",<unk>  that does not show an aneurysm with four-vessel angiography, carries a better prognosis than<unk>  with aneurysm, but it is still associated with a risk of ischemia, rebleeding, and hydrocephalus. Perimesencephalic<unk>  (bleeding around the mesencephalon in the brain), however, has a very low rate of rebleeding or delayed ischemia, and the prognosis of this subtype is excellent. The prognosis of head trauma is thought to be influenced in part by the location and amount of<unk>  bleeding. It is difficult to isolate the effects of<unk>  from those of other aspects of traumatic brain injury; it is unknown whether the presence of<unk>  blood actually worsens the prognosis or whether it is merely a sign that a significant trauma has occurred. People with moderate and severe traumatic brain injury who have<unk>  when admitted to a hospital have as much as twice the risk of dying as those who do not. They also have a higher risk of severe disability and persistent vegetative state, and traumatic<unk>  has been correlated with other markers of poor outcome such as post traumatic epilepsy, hydrocephalus, and longer stays in the intensive care unit. More than 90 percent of people with traumatic<unk>  bleeding and a Glasgow Coma Score over 12 have a good outcome. There is also modest evidence that genetic factors influence the prognosis in<unk> . For example, having two copies of ApoE4 (a variant of the gene encoding apolipoprotein E that also plays a role in Alzheimer's disease) seems to increase risk for delayed ischemia and a worse outcome. The occurrence of hyperglycemia (high blood sugars) after an episode of<unk>  confers a higher risk of poor outcome. Long-term outcomes Autopsy of a case with<unk> . The arachnoid mater is left in place on the exterior surface, containing extensive<unk>  that also fills the sulci, as detailed in magnified image. Neurocognitive symptoms, such as fatigue, mood disturbances, and other related symptoms are common sequelae. Even in those who have made good neurological recovery, anxiety, depression, posttraumatic stress disorder, and cognitive impairment are common; 46 percent of people who have had a<unk>  have cognitive impairment that affects their quality of life. Over 60 percent report frequent headaches. Aneurysmal<unk>  may lead to damage of the hypothalamus and the pituitary gland, two areas of the brain that play a central role in hormonal regulation and production. More than a quarter of people with a previous<unk>  may develop hypopituitarism (deficiencies in one or more of the hypothalamic-pituitary hormones such as growth hormone, luteinizing hormone, or follicle-stimulating hormone).
Subarachnoid hemorrhage
Migraine
Subdural hematoma
Meningitis
Cerebral venous sinus thrombosis
Migraine 
ROSAH syndrome
Epidural hematoma