<unk>  <unk>  is described as a rare form of non-cirrhotic portal hypertension in the absence of a recognizable cause.<unk>  is associated with solid organ transplant (eg. renal transplants, bone marrow transplants) and chronic use of medications. it may be classified into 2 subtypes: Pre-sinusoidal and sinusoidal. The pathogenesis of<unk>  is characterized by arterial hypervascularity secondary to loss of hepatic vein radicles and loss of central venule in the hepatic lobule.<unk>  is a rare disease. The estimated incidence of<unk>  is approximately 0.34 cases per 100,000 individuals.<unk>  is more commonly observed among patients between 25 and 65 years old. The majority of patients with<unk>  may be initially asymptomatic. Early clinical features include fatigue, weight loss, and abdominal distension.  If left untreated, the majority of patients with<unk>  may progress to develop acute hepatic failure and death. The diagnosis of<unk>  is made with the following diagnostic criteria: Latency of more than 6 months, minimal or no elevations in serum alanine transaminase (ALT), clinical, radiologic or endoscopic signs of portal hypertension, and liver biopsy. Treatment includes cessation of offending agents and management of portal hypertension and variceal bleeding.
Nodular regenerative hyperplasia
Atypical small acinar proliferation
Nodular lesions
Fibroepithelial neoplasms
Bauxite fibrosis
Fat necrosis
Reticular formation
Granuloma