Is defective expression of HRK associated with promoter methylation in primary central nervous system lymphomas?
Our findings suggest that transcriptional repression of HRK is caused by promoter hypermethylation in PCNSL, and that the loss of HRK associated with the methylation profile of other genes is a potential step in the modulation of cellular death by apoptosis during PCNSL tumorigenesis.
CPU-86017 is more potent to suppress Ca2+ entry than intracellular calcium mobilization and Lev enhances both.