Does dietary alpha-linolenic acid inhibit proinflammatory cytokine production by peripheral blood mononuclear cells in hypercholesterolemic subjects?
Increased intakes of dietary ALA elicit antiinflammatory effects by inhibiting IL-6, IL-1beta, and TNF-alpha production in cultured PBMCs. Changes in PBMC ALA and eicosapentaenoic acid (derived from dietary ALA) are associated with beneficial changes in TNF-alpha release. Thus, the cardioprotective effects of ALA are mediated in part by a reduction in the production of inflammatory cytokines.
Indicators of masticatory ability and masticatory behavior have different implications in their association with dietary patterns in children.