Are mice deficient in tumor necrosis factor receptors p55 and p75 , interleukin-4 , or inducible nitric oxide synthase susceptible to endotoxin-induced uveitis?
Mice deficient in TNFR p55 and TNFR p75, IL-4, or iNOS retain the susceptibility to EIU, but TNF-alpha and IL-4 influence the influx of inflammatory cells to the eye during this disease.
FLT3 signaling attenuates ALI by regulating the accumulation and maturation of pulmonary cDCs, indicating a potential pharmacotherapy for ALI.