Does nitric oxide increase hepatic arterial blood flow in rats with carbon tetrachloride-induced acute hepatic injury?
The results of this study suggest that the increased hepatic arterial blood flow in CCl4-induced acute hepatic injury is mediated by excessive NO production and up-regulated by inducible NOS, which plays a role in reducing hepatic injury.
Our data identify BMP2 as a key negative regulator for the emergence of the lymphatic lineage during vertebrate development.