Does lipocalin-2 deficiency or blockade protect against aortic abdominal aneurysm development in mice?
Either Lcn2 deficiency or anti-Lcn2 antibody blockade limits AAA expansion in mice by decreasing PMN infiltration in the aorta. Lcn2 modulation may therefore be a viable new therapeutic option for the treatment of AAA.
Coupled pacing reversed the left ventricular remodeling caused by the tachycardia resulting from AF.