Does spinal mitochondrial-derived ROS contribute to remifentanil-induced postoperative hyperalgesia via modulating NMDA receptor in rats?
These findings indicated that ROS-dependent activation of NMDA receptor in the spinal cord might be a potential mechanism underlying remifentanil-induced postoperative hyperalgesia.
CK level was the most sensitive index of PM/DM and was highly correlated with other serum muscle enzyme levels. An equation capable of predicting potential liver damage according to the correlation of CK and ALT was produced.