Is prazosin-stimulated release of hepatic triacylglyceride lipase from primary cultured rat hepatocytes involved in the regulation of cAMP-dependent protein kinase through activation of the Ca ( 2+ ) /calmodulin-dependent protein kinase-II?
These results suggested that prazosin-stimulated HTGL release from hepatocytes was caused by activation of PKA associated with stimulation of CaMK-II activity through a signal cascade from PLC.
Data indicate age-dependent motivational factors for exercise engagement that may hold important implications for exercise adherence and engagement strategies.