Is inhibition of nitric oxide synthesis in ischemia/reperfusion of the rat liver followed by impairment of hepatic microvascular blood flow?
Our results provide further evidence that NO maintains postischemic hepatic microvascular perfusion and that inhibition of NO synthesis has detrimental effects on hepatic microhemodynamics during reperfusion.
In the light of the reduction in selenium status in a number of European countries in recent years, this study raises the question of whether a small increase in selenium intake might help prevent preeclampsia in susceptible women.