Does pretreatment of liver grafts in vivo by γ-aminobutyric acid receptor regulation reduce cold ischemia/warm reperfusion injury in rat?
A specific agonist demonstrated regulation of GABAR in vivo in the liver. This activation in vivo reduced OS after OLT via the ATM/H2AX pathway.
An increase in expression of myoglobin protein in skeletal muscle reduces the endogenous perfusion recovery following surgically induced hind-limb ischemia.