Does l-N-iminoethyl-lysine after experimental brain trauma attenuate cellular proliferation and astrocyte differentiation?
Our results confirmed that reactive glial cells dominated the proliferating cell population after TBI and suggested that NO-regulated mechanisms are relevant for post-traumatic cellular proliferation and differentiation, since NO inhibition decreased the number of proliferating cells in the SVZ and the proportion of proliferating cells expressing GFAP, a marker of glial proliferation.
Temp Bond and Provilink luting agents exhibited the lowest mean tensile bond strengths. Neo-Temp luting agent exhibited a tensile bond strength more than 3 times that of Temp Bond luting agent.