Does miR-31 Overexpression exacerbate Atherosclerosis by Targeting NOX4 in apoE ( -/- ) Mice?
The current data suggested that miR-31 contributed to macrophage apoptosis by regulating the expression of NOX4.
Parallel acceleration of sequences used for adrenal tissue visualization leads to a significant increase in diagnostic quality by significantly reducing breathing motion artifacts without sacrificing contrast indispensable for adrenal lesion characterization.