Do high velocity transient visual processing deficits diminish ability of patients with schizophrenia to recognize objects?
The findings afford support for an early information processing deficit in schizophrenics. These data also support the neurophysiologic model that explains the poor performance of patients with schizophrenia as it relates to a transient channel deficiency.
In the present study, the protein extracted from human urine was identical to bikunin, which may be expressed mainly in the proximal tubules and the thin descending segment near the loop of Henle, and which prevents CaOx crystallization in vitro.