Is kainic acid-induced microglial activation attenuated in aged interleukin-18 deficient mice?
Deficiency of IL-18 attenuates microglial activation after KA-induced excitotoxicity in aged brain, while the net effects of IL-18 deficiency are balanced by the enhancement of other cytokines, such as TNF-alpha, IL-6 and IL-10.
We demonstrated the significant association between proteinuria and HCV, but not HBV, infection in this HBV/HCV-endemic area.