Does cardiomyocyte A Disintegrin And Metalloproteinase 17 ( ADAM17 ) be Essential in Post-Myocardial Infarction Repair by Regulating Angiogenesis?
This study highlights the key role of cardiomyocyte ADAM17 in post-MI recovery by regulating VEGFR2 transcription and angiogenesis, thereby limiting left ventricular dilation and dysfunction. Therefore, ADAM17 upregulation, within the physiological range, could provide protective effects in ischemic cardiomyopathy.
The Lanz ETT decreases tracheal mucosal injury in dogs.