Does ingestion of quercetin inhibit platelet aggregation and essential components of the collagen-stimulated platelet activation pathway in humans?
This study provides new evidence of the relatively high systemic availability of quercetin in the form of quercetin-4'-O-beta-D-glucoside by supplementation, and implicates quercetin as a dietary inhibitor of platelet cell signaling and thrombus formation.
Our results suggest that WBC count and CRP levels both independently predict mortality in the oldest old.