Does interindividual transcriptional regulation of the human biglycan gene involve three common molecular haplotypes?
Our results indicate that molecular haplotypes within the BGN promoter may contribute to the molecular basis of interindividually different transcriptional BGN regulation, possibly modulating the predisposition to cardiovascular disease-related phenotypes.
In patients with SLE, the presence of carotid atherosclerosis is not associated with low BMD or VF.