Does hypoxia-up-regulated mitochondrial movement regulator contribute to the APP/PS1 double transgenic mouse model of Alzheimer 's disease?
These results indicate that HUMMR does not play a key role in mitochondrial dysfunction in the APP/PS1 double transgenic AD mouse.
Benchmark dose estimates indicated that BZ exposure at levels below the current occupational exposure standard can induce genotoxicity and hematotoxicity.