Does trypsin inhibit lipopolysaccharide signaling in macrophages via toll-like receptor 4 accessory molecules?
Trypsin inhibited LPS signaling PAR-independently via degradation of TLR4 accessory molecules. This observation provides a better understanding of the complicated immune response in acute pancreatitis.
Majority of NS+AFL-A do not seem to have undiagnosed asthma and may instead have airflow limitation caused by other risk factors.