Are nOD2 gene variants a risk factor for culture-positive spontaneous bacterial peritonitis and monomicrobial bacterascites in cirrhosis?
NOD2 variants increase the risk for culture-positive SBP and bacterascites in cirrhosis and may affect survival.
This work supports a role of L-type Ca2+ channels both in the electrical and ionic changes occurring during the early phases of O2/glucose deprivation.