Does γ-Aminobutyric acid-mediated neurotransmission in cerebellar-hypothalamic circuit attenuate gastric mucosal injury induced by ischemia-reperfusion?
These results indicate that the protective effects of the FN stimulation against GI-R injury may be mediated by attenuation of the excessive GSN activation, gastric mucosal cell apoptosis, and Bax expression in GI-R rats.
The bioadhesive polymer conjugate described here seems to be a useful tool in overcoming enzymatic degradation by aminopeptidase N.