Is jNK and IKKβ phosphorylation reduced by glucocorticoids in adipose tissue from insulin-resistant rats?
The insulin-resistant status of rats induced by dexamethasone administration have PKB and IRS-1 activity attenuated in epididymal fat without increases in the phosphorylation of the proinflammatory signals JNK and IKKβ.
A prospective database provides important information that can lead to improvement in patient care by identifying specific complications. High-risk patients such as the elderly, the obese, and those with poor pulmonary function can safely undergo pulmonary resection and have a shorter hospital stay.