Is activation of the Wnt/beta-catenin signaling pathway during oral carcinogenesis process influenced by the absence of galectin-3 in mice?
Absence of galectin-3 does not interfere in the pattern of beta-catenin expression and therefore in the mediation of the Wnt signaling pathway.
The combination of high-energy and altered surface chemistry present on TiZr modSLA was able to influence macrophages to produce the greatest anti-inflammatory microenvironment and reduce extended pro-inflammatory factor release.