Does study of Ethanol-Induced Golgi Disorganization reveal the Potential Mechanism of Alcohol-Impaired N-Glycosylation?
Thus, we provide the mechanism by which EtOH-induced Golgi remodeling may significantly modify formation of N-glycans.
Our results suggest that SAS is a promising anti-inflammatory agent in RPE cells, and may be an effective therapeutic approach for controlling chorioretinal diseases.