Does heart failure determine the myocardial inflammatory response to injury?
Heart failure patients show a clear myocardial inflammatory response upon reperfusion, probably explained by degranulation of infiltrated inflammatory cells. Results in controls indicate that they better withstand cardioplegic arrest and reperfusion without an explicit myocardial inflammatory response.
Methocarbamol inhibits sucrase activity and its carbamate group plays a main role in the binding of drug to sucrase.