Does infection with human rhinovirus 16 promote enhanced IgE responsiveness in basophils of atopic asthmatics?
Circulating basophils display increased IgE responsiveness 3 weeks after rhinovirus infection in atopic asthmatics. This observation, coupled with increased expression of Syk, implicates basophils in promoting, or else prolonging, rhinovirus-induced inflammation in atopic asthmatics.
ADMA attenuates the association of endogenous melatonin with night-time BP and dipping in the general elderly population.