Does magnolol attenuate sepsis-induced gastrointestinal dysmotility in rats by modulating inflammatory mediators?
Magnolol prevents sepsis-induced suppression of intestinal motility in rats. The potential mechanism of this benefit of magnolol appears to be modulation of self-amplified inflammatory events and block of oxidative stress in the intestine.
Visfatin serum concentration seems to be increased in preeclampsia as compared with uncomplicated pregnancy.