Is excess post-exercise oxygen consumption associated with mechanomyographic amplitude after incremental cycle ergometry in the quadriceps femoris muscles?
These results suggest that EPOC after exercise could not be exclusively attributed to elevated activity of the working muscles.
The visual loss associated with ethambutol may be mediated through an excitotoxic pathway, inasmuch as ganglion cells are rendered sensitive to normally tolerated levels of extracellular glutamate. Ethambutol perturbs mitochondrial function. Its toxicity may depend on decreased ATPase activity and mitochondrial energy homeostasis. Glutamate antagonists may be useful in limiting the side effects seen with ethambutol.