Does exogenous insulin enhance glucose-stimulated insulin response in healthy humans independent of changes in free fatty acids?
Insulin potentiates glucose-stimulated insulin response independent of FFA concentrations in healthy humans.
One of the consequences of interrupting mitochondrial culling by the PINK1-Mfn2-Parkin mechanism is an increase in mtDNA double-stranded breaks, which adversely impact mitochondrial function and DNA replication.