Does granulocyte colony stimulating factor directly inhibit myocardial ischemia-reperfusion injury through Akt-endothelial NO synthase pathway?
These results suggest that G-CSF acts directly on the myocardium during ischemia-reperfusion injury and has acute nongenomic cardioprotective effects through the Akt-endothelial NOS pathway.
MRCPs are stable measures for interventional studies. However, they do not reflect clinical severity of dystonic symptoms or improvement after therapeutic interventions.