Is hyperhomocysteinemia associated with the presence of left atrial thrombus in stroke patients with nonvalvular atrial fibrillation?
High plasma homocysteine conveys an independent risk for LA thrombus formation in patients with stroke caused by nonvalvular atrial fibrillation. This finding further supports the thrombogenic role of high homocysteine in conditions associated with blood stasis.
Oxygen administration results in a lung pathology known as oxygen toxicity. This effect is usually not significant if the duration of exposure is limited to < 24 h. In the presence of acute inflammatory lung injury, exposure to hyperoxia results in lung damage in a shorter time. We demonstrate that sufficiently decreased lung antioxidant reserve capacity may be accountable for this early toxicity.