Does a moderate-intensity static magnetic field enhance repair of cartilage damage in rabbits?
[corrected] In this animal study, microscopic Mankin's scoring depicted histological improvement in cartilage of magnet group.
Actin is a necessary component of the cytoskeleton-based forward trafficking apparatus for Cx43. In cardiomyocytes, Cx43 vesicles spend a majority of their time pausing at nonsarcomeric actin rest stops when not undergoing microtubule-based transport to the plasma membrane. Deleterious effects on this interaction between Cx43 and the actin cytoskeleton during acute ischemia contribute to losses in Cx43 localization at intercalated discs.