Do clinical features associated with internal carotid artery occlusion correlate with MRA cerebropetal flow measurements?
Neurological deficit caused by (transient) hemispheric ischaemia is associated with low N-acetylaspartate/choline ratios, whereas prior clinical features are not associated with low cerebropetal blood flow, as measured with MR angiography. As a result, differences in cerebropetal flow cannot explain why patients with similar carotid artery disease experience different neurological features.
The after effects of motor cortex rTMS can be selectively modulated by the pattern of muscle contraction.