Is pathological but not physiological retinal neovascularization altered in TNF-Rp55-receptor-deficient mice?
Inhibition of TNFalpha via TNF-Rp55 can alter retinal development and angiogenesis in a model of oxygen-induced retinopathy. The data underscore the potential effectiveness of TNF-inhibitory treatments as modulators in oxygen-induced retinopathy.
The newborn blood spot transcriptome can serve as a platform for investigating distinctive gene expression patterns in children who later develop CP.