Does low-dose X-irradiation promote mineralization of fracture callus in a rat model?
The results indicated LDI promoted mineralization at the stage of hard callus formation in a rat fracture model.
Neurally derived NO is responsible for a major component of the ChBF increase caused by EW stimulation in pigeons. This represents the first demonstration in vivo that neuronally produced NO is an important factor in the control of ChBF by the parasympathetic nervous system. In particular, neuronally produced NO appears to play a role in rapid upregulation of ChBF in the pigeon, whereas endothelially produced NO plays a major role in control of resting ChBF.