Does salidroside protect against Homocysteine-induced Injury in HUVECs via The Regulation of Endoplasmic Reticulum Stress?
Taken together, these findings implicate that SAL could regulate ER-stress pathway on the viability of endotheliocyte induced by Hcy in vitro. Our findings provide the first evidence that SAL plays an important role in endotheliocyte protection via suppressing ER-stress pathway in HUVEC cells, and that it may be a promising therapeutic target for atherosclerosis and cardiovascular disease. This article is protected by copyright. All rights reserved.
PRL production is suppressed in leiomyomas but not in myometrium after treatment with onapristone in vitro. This suppression may serve as a marker for the clinical effectiveness of agents used in the treatment of leiomyomas.