Is the anemia of `` haemoglobin-deficit '' ( hbd/hbd ) mice caused by a defect in transferrin cycling?
Our results indicate that heme biosynthesis, DMT1, and the mitochondrial iron handling machinery are all normal in hemoglobin-deficit mice, while transferrin cycling is deficient. Therefore, the product of Sec15l1 is directly involved in vesicular trafficking, docking, fusing, and/or cargo delivery in erythroid precursors.
Understanding what motivates veterans to use dual care and influences their satisfaction with care received will enable the VHA to implement policy that improves the quality of care provided to rural veterans.