Is alkali-induced corneal neovascularization independent of CXCR2-mediated neutrophil infiltration?
CXCR2-mediated neutrophil infiltration contributes only marginally to the subsequent development of CNV.
This study provides a first indication of EIC as possible precursor lesion for SOC. This finding could have major clinical implications for future ovarian cancer management and underscores EIC as a possible target for early SOC detection and prevention.