Does sesamin ameliorate oxidative stress and mortality in kainic acid-induced status epilepticus by inhibition of MAPK and COX-2 activation?
Taken together, it suggests that sesamin could protect KA-induced brain injury through anti-inflammatory and partially antioxidative mechanisms.
Larger air bubbles of 80% anterior chamber volume decrease the risk of graft detachment after DMEK with no detrimental effect on the outcome and risk for pupillary block.