Is regression of glomerular injury by kallikrein infusion in Dahl salt-sensitive rats a bradykinin B2-receptor-mediated event?
Long-term infusion of subdepressor dose of rat urinary kallikrein attenuates functionally and morphologically the progression of renal injury in Dahl rats susceptible to salt-induced hypertension, and that the protection is mediated by stimulation of bradykinin B2 receptor.
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