Is vascular endothelial cell growth-activated XBP1 splicing in endothelial cells crucial for angiogenesis?
These results suggest that XBP1 can function via growth factor signaling pathways to regulate endothelial proliferation and angiogenesis.
These results indicate that serotonin is a likely endogenous mediator that can increase cilia-driven transport independent from acetylcholine during activation of mast cells and platelets.