Does circulating interleukin-1β promote endoplasmic reticulum stress-induced myocytes apoptosis in diabetic cardiomyopathy via interleukin-1 receptor-associated kinase-2?
Elevated circulating IL-1β contributed to promote ER stress- induced myocytes apoptosis by affecting IRAK-2/CHOP pathway in DCM.
In the appropriate clinical context we suggest molecular testing for C. belli or an empirical therapeutic trial.