Does c-Abl contribute to glucose-promoted apoptosis via p53 signaling pathway in podocytes?
c-Abl contributes to high glucose-induced podocyte apoptosis via p53 signaling pathway.
Thyroid cancer subtypes present differential promoter methylation signatures, and nondifferentiated subtypes are characterized by aberrant promoter hypomethylation rather than hypermethylation. Additional studies are needed to determine the potential clinical interest of the tumor subtype-specific DNA methylation signatures described herein and the role of aberrant promoter hypomethylation in nondifferentiated thyroid tumors.