Are cdc42 and Rac1 major contributors to the saturated fatty acid-stimulated JNK pathway in hepatocytes?
Our results demonstrate that Cdc42 and Rac1, rather than ER stress, are important components of a SFA-stimulated signaling pathway that regulates MLK3-dependent activation of JNK in hepatocytes.
DAC should be differentiated from artifacts using cough test and the presence of DAC was significantly correlated to the presence of BOO.