Are pathophysiological consequences of TAT-HKII peptide administration independent of impaired vascular function and ensuing ischemia?
Our findings challenge the notion that the effects of TAT-HKII are attributable to impaired vascular function and ensuing ischemia, thereby lending further credence to the role of mitochondria-bound HKII as a critical regulator of cardiac function, ischemia-reperfusion injury, and cardioprotection by ischemic preconditioning.
Health literacy mediates the relationship between age and health outcomes in adults with HF.