Does gliclazide protect human islet beta-cells from apoptosis induced by intermittent high glucose?
Therefore, gliclazide protected human beta-cells from apoptosis induced by intermittent high glucose, and this effect was likely to be due, at least in part, to the anti-oxidant properties of the molecule.
RIPC may protect the liver from IRI by induction of HO-1/p38-MAPK-dependent autophagy.