Do mast cell-derived proteases control allergic inflammation through cleavage of IgE?
We suggest that IgE cleavage by effector cell proteases is a natural mechanism for controlling allergic inflammation.
The changes in P27 amplitude observed in Experiments 1 and 2 reflect an intermodal mechanism of somatosensory gating dependent upon spatiotemporal properties of bimodal stimulation whereas the P50 reflects multisensory aspects of somatosensory processing. Changes in behavior may reflect the importance of these early influences to sensorimotor transformations.