Does dexamethasone suppress peripheral prostanoid levels without analgesia in a clinical model of acute inflammation?
The lack of an analgesic effect for dexamethasone while reducing both PGE(2) and TxB(2) at the site of injury in comparison to ketorolac analgesia accompanied by greater reductions in levels of these prostanoids suggests that glucocorticoids at this dose do not suppress PGE(2) release sufficiently to attenuate peripheral sensitization of nociceptors after tissue injury.
Control of dust mites growth could alleviate the asthmatic symptoms and decrease the attacks.