Is the inverse association of incident cardiovascular disease with plasma bilirubin unaffected by adiponectin?
Bilirubin is related to adiponectin, but the association of bilirubin with CVD risk is largely unaffected by adiponectin.
HF-SEP increase probably reflects the hyperactivation of brain-stem arousal-related structures. During such a hyperactivation no EEG abnormalities were observed.