Does inhibition of p38 MAPK activity fail to attenuate contractile dysfunction in a mouse model of low-flow ischemia?
The p38 MAPK activation that accompanies short-term hibernation does not appear to contribute to the contractile deficit.
Caution should be exercised when treating patients with oral sodium phosphate who are considered to be cellularly depleted of potassium. These patients are at risk of hypokalaemia after this treatment.