Do acute exercises induce disorders of the gastrointestinal integrity in a murine model?
In the murine exercise model there is evidence that after intense endurance exercise repair processes occur in small intestinal epithelial cells, which affect permeability, Gt, and Isc. The formation of lamellipodia to close the "leaky" tight junctions caused by apoptosis might be an underlying mechanism.
The notochord is abnormal in the region of the developing midgut, and this may account for the occurrence of atresias found in this region.