Does conduction slowing contribute to spontaneous ventricular arrhythmias in intrinsically active murine RyR2-P2328S hearts?
We associate altered ventricular myocardial CV potentially resulting in arrhythmogenic substrate with arrhythmic properties associated with genetic RyR2 alterations for the first time.
The immunological consequences of hypertonic saline infusion seem to be modest and are unlikely to cause any clinical effects, at least in normovolaemic women.