Does cD70 exacerbate Blood Pressure Elevation and Renal Damage in Response to Repeated Hypertensive Stimuli?
Our findings illustrate a previously undefined role of CD70 and long-lived T(EM) cells in the development of blood pressure elevation and end-organ damage that occur on delayed exposure to mild hypertensive stimuli. Interventions to prevent repeated hypertensive surges could attenuate formation of hypertension-specific T(EM) cells.
The coagulation system is activated in the left atrium of patients with mitral stenosis even during anticoagulation.