Does low glucose but not galactose enhance oxidative mitochondrial metabolism in C2C12 myoblasts and myotubes?
Our results show that using galactose to increase oxidative metabolism may not be applicable to every cell line, and the changes in mitochondrial respiratory parameters associated with treating cells with galactose are mainly due to glucose deprivation. Moderate concentrations of glucose (1 g/l) in a growth medium are optimal for mitochondrial respiration in C2C12 cell line while supraphysiological concentrations of glucose cause mitochondrial dysfunction in C2C12 myoblasts and myotubes.
We have no evidence that bone marrow derived dendritic cells from patients with MM are infected with KSHV.