Rotator cuff tear oxidative stress induces tenocyte apoptosis causing further rotator cuff tendon degeneration. A neural theory also exists that suggests neural overstimulation leads to the recruitment of inflammatory cells and may also contribute to tendon degeneration. Tears of the rotator cuff tendon are described as partial or full thickness, and full thickness with complete detachment of the tendons from bone. Shoulder pain is variable and may not be proportional to the size of the tear. Neer promoted the concept of three stages of rotator cuff disease. Stage I, according to Neer, occurred in those younger than 25 years and involved edema and
Shoulder joint the biceps brachii on the anterior side of the arm, and tendons of the rotator cuff; which are fused to all sides of the capsule except the inferior margin. The tendon of the long head of the biceps brachii passes through the bicipital groove on the humerus and inserts on the superior margin of the glenoid cavity to press the head of the humerus against the glenoid cavity. The tendons of the rotator cuff and their respective muscles (supraspinatus, infraspinatus, teres minor, and subscapularis) stabilize and fix the joint. The supraspinatus, infraspinatus and teres minor muscles aid in abduction and