Dry rot treatment Dry rot treatment Dry rot treatment refers to techniques used to eliminate dry rot fungus and alleviate the damage done by the fungus to human-built wooden structures. Dry rot ("Serpula lacrymans") is considered difficult to remove, requiring drastic action. Remedial timber treatment and damp proofing companies typically recommend stripping out of building fabric beyond the visible extent of the infestation and the use of fungicide. More holistic approaches attempt to eradicate dry rot by controlling the local environment to reduce timber moisture levels and increase ventilation in order to promote drying. The first priority when treating dry rot is to
Serpula lacrymans of 13805 genes. "S. lacrymans"' genome encodes six annotated polyketide synthases (PKS1-PKS6), 15 nonribosomal peptide synthetases (NPS1-NPS4, NPS7, NPS13-NPS15, NPS17, and NPS18), and two hybrids thereof (NPS6, NPS8, and NPS16). Additionally, the genome encodes for various putative adenylate-forming reductases (NPS5, NPS9-NPS12) (Eastwood et al., 2011). "NPS3" was overexpressed in E. coli and characterized as an atromentin/quinone synthetase that catalyzes the formation of atromentin, similar to GreA; InvA1,2 and 5; and AtrA from Suillus grevillei, Tapinella panuoides, Paxillus involutus, respectively. "NPS3" and its adjacent clustered aminotransferase gene ("AMT1") were also found to be up-regulated during co-incubation with bacteria (Tauber et al.,