Neuromechanics of idiopathic scoliosis shoulders compensates the angular momentum of the pelvis (i.e. when the shoulders rotate clockwise, the pelvis rotates counterclockwise; when the pelvis rotates clockwise, the shoulders rotate counterclockwise). As scoliosis perturbs the architecture of the shoulders, spine and pelvis, gait also differs between patients and non-scoliotic people. Independently of the Cobb angle, the affected vertebra and the age, idiopathic scoliotic people have a larger rachis flexion range of motion and a narrower hips extension range of motion than non-scoliotic people. The range of motion for rachis extension, hips flexion, left and right lateral flexions are similar to non-scoliotic people. After arthrodesis,
Scoliosis is due to genetic factors, and 62% is due to the environment. The genetics are likely complex, however, given the inconsistent inheritance and discordance among monozygotic twins. The specific genes that contribute to development of scoliosis have not been conclusively identified. At least one gene, "CHD7", has been associated with the idiopathic form of scoliosis. Several candidate gene studies have found associations between idiopathic scoliosis and genes mediating bone formation, bone metabolism, and connective tissue structure. Several genome-wide studies have identified a number of loci as significantly linked to idiopathic scoliosis. In 2006, idiopathic scoliosis was linked with three microsatellite