Neuromechanics of idiopathic scoliosis shoulders compensates the angular momentum of the pelvis (i.e. when the shoulders rotate clockwise, the pelvis rotates counterclockwise; when the pelvis rotates clockwise, the shoulders rotate counterclockwise). As scoliosis perturbs the architecture of the shoulders, spine and pelvis, gait also differs between patients and non-scoliotic people. Independently of the Cobb angle, the affected vertebra and the age, idiopathic scoliotic people have a larger rachis flexion range of motion and a narrower hips extension range of motion than non-scoliotic people. The range of motion for rachis extension, hips flexion, left and right lateral flexions are similar to non-scoliotic people. After arthrodesis,
Neuromechanics of idiopathic scoliosis Besides, idiopathic scoliosis being more frequent in certain families, it is suspected to be transmitted via autosomal dominant inheritance. Estrogens could also play a crucial part in the progression of idiopathic scoliosis through their roles in bone formation, growth, maturation and turnover. Finally, collagen, intervertebral disc and muscle abnormalities have been suggested as the cause in idiopathic scoliosis, although these are perhaps results rather than causes. The patient’s age at presentation of scoliosis, the remaining growth, the menarche status and the magnitude of the curve are used to foresee the idiopathic scoliosis curve progression. However, the relative importance of each