Scoliosis polymorphisms in the "MATN1" gene (encoding for matrilin 1, cartilage matrix protein). Fifty-three single nucleotide polymorphism markers in the DNA that are significantly associated with adolescent idiopathic scoliosis were identified through a genome-wide association study. Adolescent idiopathic scoliosis has no clear causal agent, and is generally believed to be multifactorial. The prevalence of scoliosis is 1% to 2% among adolescents, but the likelihood of progression among adolescents with a Cobb angle less than 20° is about 10% to 20%. Congenital scoliosis can be attributed to a malformation of the spine during weeks three to six "in utero" due to a
Body shape image). This sometimes affects their walking style, resulting in hip sway; The upper limb in females have an outward angulation (carrying angle) at elbow level to accommodate the wider pelvis. After puberty, hips are generally wider than shoulders. However, not all females adhere to this stereotypical pattern of secondary sex characteristics. Both male and female hormones are present in the human body, and though only one of them is predominant in an adult, the other hormone has effects on body's shape to some extent. Widening of the shoulders occurs as part of the male pubertal process. Expansion of the ribcage