Exercise-associated hyponatremia cells in one area begin to increase in size, swelling occurs in the affected area. Swelling is commonly observed in hands, legs, and feet. Sodium is also important in regulating the amount of water that passes through the blood-brain barrier. Decreased sodium blood levels result in increased permeability of water across the blood-brain barrier. This increased influx of water causes brain swelling which leads to severe neurological symptoms. EAH is categorized by having a blood serum or plasma sodium level below normal, which is less than 135 mmol/l. Asymptomatic EAH is not normally detected unless the athlete has had a
Hypotonic hyponatremia ADH secretion. As a result, the kidneys of such patients recover water and produce a fairly concentrated urine. Treatment is simple (if not without risk) — simply restore the patient's blood volume, thereby turning off the stimulus for ongoing ADH release and water retention. It is worth considering separately, the hyponatremia that occurs in the setting of diuretic use. Patients taking diuretic medications such as furosemide (Lasix), hydrochlorothiazide, chlorthalidone, etc., become volume depleted. That is to say that their diuretic medicine, by design, has caused their kidneys to produce more urine than they would otherwise make. This extra urine represents