Exercise-associated hyponatremia sodium blood serum or plasma test. Hyponatremic encephalopathy may be detect using brain imaging studies and pulmonary edema may be confirmed by x-ray. Traditional prevention of EAH focuses on reducing fluid consumption to avoid fluid retention before, during, and after exercise. However, since this can risk dehydration, an alternative approach is possible of consuming a substantial amount of salt prior to exercise. It is still important not to overconsume water to the extent of requiring urination, because urination would cause the extra salt to be excreted. In a published statement of the Third International Exercise-Associated Hyponatremia Consensus Development Conference, researchers
Serum sickness proteins to form immune complexes. These complexes precipitate, enter the walls of blood vessels, and activate the complement cascade, initiating an inflammatory response and consuming much of the available complement component 3 (C3). The result is a leukocytoclastic vasculitis. This results in hypocomplementemia, a low C3 level in serum. They can also cause more reactions resulting in typical symptoms of serum sickness. Serum sickness can be developed as a result of exposure to antibodies derived from animals. These sera or antitoxins are generally administered to prevent or treat an infection or envenomation. Some of the drugs associated with serum sickness