Exercise-associated hyponatremia sodium blood serum or plasma test. Hyponatremic encephalopathy may be detect using brain imaging studies and pulmonary edema may be confirmed by x-ray. Traditional prevention of EAH focuses on reducing fluid consumption to avoid fluid retention before, during, and after exercise. However, since this can risk dehydration, an alternative approach is possible of consuming a substantial amount of salt prior to exercise. It is still important not to overconsume water to the extent of requiring urination, because urination would cause the extra salt to be excreted. In a published statement of the Third International Exercise-Associated Hyponatremia Consensus Development Conference, researchers
Hyponatremia arrest, and non-cardiogenic accumulation of fluid in the lungs. This is usually fatal if not immediately treated. Symptom severity depends on how fast and how severe the drop in blood salt level. A gradual drop, even to very low levels, may be tolerated well if it occurs over several days or weeks, because of neuronal adaptation. The presence of underlying neurological disease such as a seizure disorder or non-neurological metabolic abnormalities, also affects the severity of neurologic symptoms. Chronic hyponatremia can lead to such complications as neurological impairments. These neurological impairments most often affect gait (walking) and attention, and can