Bruxism more than the periodontal ligament is biologically designed to handle, and so inflammation may result. A tooth may become sore to bite on, and further, tooth wear may reduce the insulating width of enamel and dentin that protects the pulp of the tooth and result in hypersensitivity, e.g. to cold stimuli. The relationship of bruxism with temporomandibular joint dysfunction (TMD, or temporomandibular pain dysfunction syndrome) is debated. Many suggest that sleep bruxism can be a causative or contributory factor to pain symptoms in TMD. Indeed, the symptoms of TMD overlap with those of bruxism. Others suggest that there is no
Habitual be the introduction, and subsequent loss of habitual "do (be)" in America, which was also the problem with the first expansion of the diffusion hypothesis. Another hypothesis for the origins of habitual "be" in NWBE and afterwards into AAE is the most logical and the strongest of the lot. The decreolization theory for the emergence of habitual "be" in AAE is the most likely and most supported of the theories of its origin. It involves the decreolization process of Caribbean English creole, with the loss of "does (be)", which itself is a decreolization of a previous creole habitual marker and its