Burrill Bernard Crohn published paper was "Regional Ileitis: A Pathologic and Clinical Entity". Crohn's work helped correct the thinking that the ailment was actually a form of tuberculosis rather than an inflammatory disease of the gastrointestinal tract. Crohn's work built on the work of Dr. Kennedy Dalziel of Glasgow into regional enteritis twenty years earlier. Crohn always preferred the medically descriptive terms "regional ileitis" and "regional enteritis" to "Crohn's disease", but he was not able to prevent the appropriation of his name for the disease. Some of Crohn's initial research into the causes of the Crohn's disease was centered around his personal conviction
Extrapulmonary tuberculosis in the liver or spleen, splenomegaly, splenic or lymph node calcification, inflammatory thickening of the terminal ileum and caecum, patulous I-C junction and lowattenuation lymphadenopathy may help suggest tuberculosis as the etiology of diffuse peritoneal disease. Like tuberculosis, Crohn‟s disease also can result in granulomatous peritonitis13. Smooth and regular peritoneal thickening is more in favor of granulomatous peritonitis. Extrapulmonary tuberculosis Extrapulmonary tuberculosis is tuberculosis (TB) within another location in the body than the lungs. This occurs in 15–20% of active cases, causing other kinds of TB. These are collectively denoted as "extrapulmonary tuberculosis". Extrapulmonary TB occurs more commonly in immunosuppressed