Inland taipan may include headache, nausea, vomiting, abdominal pain, diarrhoea, dizziness, collapse or convulsions leading to major organ effects: neurotoxicity, coagulopathy, rhabdomyolysis or renal failure/damage and finally death. Inland taipan snake venom contains potent presynaptic neurotoxins (toxins in venom that cause paralysis or muscle weakness). Also present are postsynaptic neurotoxins, which are less potent but more rapid acting than the presynaptic neurotoxins. Presynaptic neurotoxins disrupt neurotransmitter release from the axon terminal. This takes days to resolve and does not respond to antivenom. Postsynaptic neurotoxins competitively block acetylcholine receptors but the effect can be reversed by antivenom. Neurotoxic envenoming causes a progressive descending
Inland taipan the prey with a series of rapid, accurate strikes. It is known to deliver up to eight venomous bites in a single attack, often snapping its jaws fiercely several times to inflict multiple punctures in the same attack. Its more risky attack strategy entails holding its prey with its body and biting it repeatedly. This injects the extremely toxic venom deep into the prey. The venom acts so rapidly that its prey does not have time to fight back. In captivity it may also eat day-old chicks. Inland taipan produce clutches of between one and two dozen eggs. The eggs