Inland taipan may include headache, nausea, vomiting, abdominal pain, diarrhoea, dizziness, collapse or convulsions leading to major organ effects: neurotoxicity, coagulopathy, rhabdomyolysis or renal failure/damage and finally death. Inland taipan snake venom contains potent presynaptic neurotoxins (toxins in venom that cause paralysis or muscle weakness). Also present are postsynaptic neurotoxins, which are less potent but more rapid acting than the presynaptic neurotoxins. Presynaptic neurotoxins disrupt neurotransmitter release from the axon terminal. This takes days to resolve and does not respond to antivenom. Postsynaptic neurotoxins competitively block acetylcholine receptors but the effect can be reversed by antivenom. Neurotoxic envenoming causes a progressive descending
Taipan taipans, with the central ranges taipan being an offshoot of the common ancestor of the inland and coastal taipans. Their diets consist primarily of small mammals, especially rats and bandicoots. Species of this genus possess highly neurotoxic venom with some other toxic constituents that have multiple effects on victims. The venom is known to paralyse the victim's nervous system and clot the blood, which then blocks blood vessels and uses up clotting factors. Members of this genus are considered to be among the most venomous land snakes based on their murine , an indicator of the toxicity on mice. The