Inland taipan venom procoagulants known. Though mild coagulopathy has been reported for inland taipan envenomation (Sutherland and Tibballs, 2001). No nephrotoxins (kidney toxins) have so far been isolated from inland taipan snake venoms, but renal (kidney) impairment or acute renal failure can occur secondary to severe rhabdomyolysis. Taipan snake venom does contain myotoxins that cause myolysis (rhabdomyolysis, muscle damage); the urine of a bite victim often turns reddish-brown as their muscles dissolve and are passed through the kidneys (Myoglobinuria). The kidneys are often badly damaged by filtering so much tissue debris out of the blood, and kidney failure is a common complication
Inland taipan Cook University, the majority of components in the venom have not been characterized and little molecular research has been undertaken on taipan ("Oxyuranus") species at large. As of 2005, the amino acid sequences of only seven proteins from inland taipan have been submitted to SWISS-PROT databases. The mortality rate is high in untreated cases: Clinically, envenomation may represent a complex scenario of multiple organ system poisoning with neurotoxic symptoms typically dominating. Acute renal failure, rhabdomyolysis, and disseminated coagulopathy may also complicate the setting. The first local and general symptoms of a bite are local pain and variable non-specific effects which