Pulmonary alveolus thin fluid coating within all alveoli. The fluid coating is produced by the body in order to facilitate the transfer of gases between blood and alveolar air. The surfactant is produced by the type II cells which are the most numerous cells in the alveoli, yet do not cover as much surface area as the squamous alveolar cells (a squamous epithelium). Type II cells also repair the endothelium of the alveolus when it becomes damaged. Insufficient surfactant in the alveoli can contribute to atelectasis (collapse of part or all of the lung). Without pulmonary surfactant, atelectasis is a certainty; however,
Neoteny in humans humans) interacted strongly with other genes and was part of a gene regulatory network, the derived mutation that is unique to the modern human version of the gene knocked out the attachment to which RNA strains from other genes connected to it so that the gene was disconnected from its former genetic network. It is suggested that since the FOXP2 gene controls synapses, its disconnection from a formerly complex network of genes instantly removed many instincts including ones that drove sexual selection. It is also suggested that it allowed more genetic variants that affect the phenotype to accumulate in humans,