Pathophysiology of heart failure caused by dysfunction or destruction of cardiac myocytes or their molecular components. In congenital diseases such as Duchenne muscular dystrophy, the molecular structure of individual myocytes is affected. Myocytes and their components can be damaged by inflammation (such as in myocarditis) or by infiltration (such as in amyloidosis). Toxins and pharmacological agents (such as ethanol, cocaine, doxorubicin, and amphetamines) cause intracellular damage and oxidative stress. The most common mechanism of damage is ischemia causing infarction and scar formation. After myocardial infarction, dead myocytes are replaced by scar tissue, deleteriously affecting the function of the myocardium. On echocardiogram, this is manifest
Face and neck development of the human embryo tendon, dermis, pia mater and arachnoid mater, sensory neurons, and glandular stroma. Pharyngeal arches are formed during the fourth week. Each arch consists of a mesenchymal tissue covered on the outside by ectoderm and on the inside by epithelium of endodermal origin. In human embryology, there are six arches which are separated by pharyngeal grooves externally and pharyngeal pouches internally. These arches contribute to the physical appearance of the embryo because they are the main components that build the face and neck. In addition, the muscular components of each arch have their own cranial nerve, and wherever the muscle cells