Light skin of UVA can penetrate deeply into the dermis in persons with light skin pigmentation with little protective melanin pigment. The characteristic of fair skin, red hair, and freckling is associated with high amount of pheomelanin, little amounts of eumelanin. This phenotype is caused by a loss-of-function mutation in the melanocortin 1 receptor (MC1R) gene. However, variations in the MC1R gene sequence only have considerable influence on pigmentation in populations where red hair and extremely fair skin is prevalent. The gene variation’s primary effect is to promote eumelanin synthesis at the expense of pheomelanin synthesis, although this contributes to very little
Baumann Skin Types form of post-inflammatory hyperpigmentation (PIH). Most pigment changes can be prevented and corrected through the use of proper skincare ingredients. Age, ethnicity and lifestyle habits influence whether skin is wrinkle-prone or tight. There is still a genetic component to this parameter, but external factors that contribute to an aged appearance are controllable. Sun exposure is the primary cause of skin aging, destroying skin proteins like collagen and elastin. Each parameter is associated with particular skin conditions and challenges, and the specific combinations also experience particular tendencies. For example, DRPW types often have a history of sun exposure manifested by wrinkles