Computed tomography of the thyroid attenuation (80–100 Hounsfield Units). The presence of thyroiditis can be suggested by a diffusely enlarged and hypo-attenuating (around 45 Hounsfield Units) thyroid gland. This is probably due to follicular cell destruction and reduced thyroid iodine concentration. Marked homogenous enhancement is typically expected. Therefore, moderate thyroid enhancement in a case of thyroiditis suggests a diffuse inflammatory process. It is essential to clinically correlate this with a thyroid function test and serum autoantibody levels. During embryogenesis, the bi-lobed thyroid migrates inferiorly from the foramen cecum of the tongue to the lower neck. Initially, the thyroid primordium passes anterior to the primordial hyoid
Thyroid disease in pregnancy and the placental transfer of maternal thyroxine to the fetus, the demand for thyroid hormones is increased during pregnancy. The necessary increase in thyroid hormone production is facilitated by high human chorionic gonadotropin (hCG) concentrations, which bind the TSH receptor and stimulate the maternal thyroid to increase maternal thyroid hormone concentrations by roughly 50%. If the necessary increase in thyroid function cannot be met, this may cause a previously unnoticed (imld) thyroid disorder to worsen and become evident as gestational thyroid disease. Currently, there is not enough evidence to suggest that screening for thyroid dysfunction is beneficial, especially since treatment