Scurvy These make up one of two major primate suborders, haplorrhini and this group includes humans. The strepsirrhini (non-tarsier prosimians) can make their own vitamin C, and these include lemurs, lorises, pottos, and galagos. Ascorbic acid is also not synthesized by at least two species of caviidae, the capybara and the guinea pig. There are known species of birds and fish that do not synthesize their own Vitamin C. All species that do not synthesize ascorbate require it in the diet. Deficiency causes scurvy in humans, and somewhat similar symptoms in other animals. Animals that can contract scurvy all lack the
Vitamin B12 deficiency complex, defined as "myelosis funicularis", consists of the following symptoms: Vitamin B deficiency can cause severe and irreversible damage, especially to the brain and nervous system. These symptoms of neuronal damage may not reverse after correction of hematological abnormalities, and the chance of complete reversal decreases with the length of time the neurological symptoms have been present. Elderly people are at a even higher risk of this type of damage. In infants a number of neurological symptoms can be evident due to maternal malnutrition or pernicious anemia. These include poor growth, apathy, having no desire for food, and developmental regression.