Rapid eye movement sleep resemblance to waking life), and incorporation of instinctual themes. Sometimes they include elements of the dreamer's recent experience taken directly from episodic memory. By one estimate, 80% of dreams occur during REM. Hobson and McCarley proposed that the PGO waves characteristic of “phasic” REM might supply the visual cortex and forebrain with electrical excitement which amplifies the hallucinatory aspects of dreaming. However, people woken up during sleep do not report significantly more bizarre dreams during phasic REMS, compared to tonic REMS. Another possible relationship between the two phenomena could be that the higher threshold for sensory interruption during REM sleep
Rapid eye movement sleep from unavailability of monoamine neurotransmitters (restraining the abundance of acetylcholine in the brainstem) and perhaps from mechanisms used in waking muscle inhibition. The medulla oblongata, located between pons and spine, seems to have the capacity for organism-wide muscle inhibition. Some localized twitching and reflexes can still occur. Pupils contract. Lack of REM atonia causes REM behavior disorder, sufferers of which physically act out their dreams, or conversely "dream out their acts", under an alternative theory on the relationship between muscle impulses during REM and associated mental imagery (which would also apply to people without the condition, except that commands to