Transgender hormone therapy (male-to-female) orchiectomy still experience morning erections. Voluntary erections may or may not be possible, depending on the amount of hormones and/or antiandrogens being taken. Managing long-term hormonal regimens have not been studied and are difficult to estimate because research on the long-term use of hormonal therapy has not been noted. However, it is possible to speculate the outcomes of these therapies on transgender people based on the knowledge of the current effects of gonadal hormones on sexual functioning in cisgender men and women. Firstly, if one is to decrease testosterone in male-to-female gender transition, it is likely that sexual desire and
Fraternal birth order and male sexual orientation sexual orientation via the fraternal birth order effect (for example, homosexual men with no older brothers) owe their homosexuality to other causes such as certain polymorphic genes (colloquially called "gay genes") or prenatal hormone levels during fetal development. Both direct and indirect evidence have demonstrated that the mechanism by which the fraternal birth order effect operates is prenatal in timing and biological in nature, rather than postnatal in timing or psychosocial in nature. Indirect evidence was found first when it was discovered that the fraternal birth order effect interacts with birth weight. Then, Bogaert (2006) produced direct evidence that the