Computed tomography of the thyroid attenuation (80–100 Hounsfield Units). The presence of thyroiditis can be suggested by a diffusely enlarged and hypo-attenuating (around 45 Hounsfield Units) thyroid gland. This is probably due to follicular cell destruction and reduced thyroid iodine concentration. Marked homogenous enhancement is typically expected. Therefore, moderate thyroid enhancement in a case of thyroiditis suggests a diffuse inflammatory process. It is essential to clinically correlate this with a thyroid function test and serum autoantibody levels. During embryogenesis, the bi-lobed thyroid migrates inferiorly from the foramen cecum of the tongue to the lower neck. Initially, the thyroid primordium passes anterior to the primordial hyoid
Thyroid disease of transport proteins, resulting in differing total and free thyroid hormone levels. There are differing guidelines for T3 and T4 measurements. Autoantibodies to the thyroid gland may be detected in various disease states. There are several anti-thyroid antibodies, including anti-thyroglobulin antibodies (TgAb), anti-microsomal/anti-thyroid peroxidase antibodies (TPOAb), and TSH receptor antibodies (TSHRAb). Many people may develop a thyroid nodule at some point in their lives. Although many who experience this worry that it is thyroid cancer, there are many causes of nodules that are benign and not cancerous. If a possible nodule is present, a doctor may order thyroid function tests