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terminator codon mutations and hypofunctional genes responsible for various Î±-thalassemia syndromes [6]. Î±-thalassemia can be suspected based on factors such as a family history of anemia and ethnic and geographic backgrounds, particularly if the patients
It is a disorder that is caused by a microdeletion in chromosome band 22q11.2. The key gene that is lost is Tbx-1, a control gene that regulates other genes required for the connection of the heart with the blood circulation. Tbx-1 also controls genes involved in the development of the parathyroid and thymus glands and the shape of the face. Babies with DiGeorge syndrome are born with hypocalcemia, congenital heart disease, cleft lip and/or palate, and are highly susceptible to infections.