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terminator codon mutations and hypofunctional genes responsible for various Î±-thalassemia syndromes [6]. Î±-thalassemia can be suspected based on factors such as a family history of anemia and ethnic and geographic backgrounds, particularly if the patients
This gene encodes a member of the short-chain nonmetalloenzyme alcohol dehydrogenase protein family. The encoded enzyme is responsible for the metabolism of prostaglandins, which function in a variety of physiologic and cellular processes such as inflammation.Mutations in this gene result in primary autosomal recessive hypertrophic osteoarthropathy and cranioosteoarthropathy. Genetic variants in HPGD encoding 15-PGDH, appear to modulate colorectal risk. 2  The present study appears to be the first to evaluate possible associations between genetic heterogeneity in HPGD and CRC risk. 3  Observational study of gene-disease association and gene-environment interaction.