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In addition, evidence on their combined effect on all-cause mortality is conflicting (17, 18). To address the interaction of obesity and OSA, we used a large historical cohort of patients with suspected OSA and investigated whether obesity influences the effect of nocturnal hypoxemia on cardiovascular events.
Substantial evidences demonstrate that obesity is a chronic low-grade inflammatory disease [56]. An important initiator of the inflammatory reaction to obesity is adipose tissue, which is consisting of adipocytes, preadipocytes, endothelial cells and immune cells (e.g., macrophages and lymphocytes).