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In type 1 diabetes the immune system erroneously attacks the beta cells that produce insulin in your pancreas. It causes permanent damage, leaving your pancreas unable to produce insulin. Exactly what triggers the immune system to do that isnât clear. Genetic and environmental factors may play a role.
Fat prevents insulin from doing its job, almost creating somewhat of an obstacle course for it to work. Whatever the specific cause, the function of insulin is impaired. Insulin resistance develops before type 2 diabetes is diagnosed. To make up for less effective insulin, the pancreas works overtime to increase insulin output. Eventually, some of the insulin works and blood sugar levels remain normal for a while. As insulin resistance worsens and the pancreas cannot keep up with the demand, glucose levels begin to rise and diabetes is diagnosed when levels get too high.