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Because there are fewer strains, they are less replicative and transmissible.â. The CCR5 mutation is not the only form of genetic immunity that exists. For example, there are what researchers call âelite controllers,â patients who test positive for HIV but who remain symptom free.nother influential HIV researcher also believes the CCR5 gene mutation is the answer. Dr. Michael Farzan, the Harvard University professor famous for discovering the benefits of CCR5 mutation 16 years ago, told The Guardian heâs close to developing an HIV vaccine based on this genetic immunity.
We review how bacterial, protozoan and fungal pathogens from distant evolutionary lineages have evolved surprisingly similar mechanisms of antigenic variation to avoid eradication by the host immune system and can therefore maintain persistent infections and ensure their transmission to new hosts.echanisms involving DNA recombination. DNA recombination that leads to the movement of non-expressed coding regions into defined genomic expression sites is one of the most common molecular mechanisms used by both bacterial and eukaryotic pathogens to produce antigenic variation (Fig. 4a).