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The major target end organs for parathyroid hormone (PTH) action are the kidneys, skeletal system, and intestine. The primary response to parathyroid hormone (PTH) by the kidney is to increase renal calcium resorption and phosphate excretion.
In the anabolic protocol, the increased MCP-1 expression was more pronounced than in the catabolic protocol, but transient, suggesting that a transient increase in bone resorption may be necessary to the osteonabolic effect of PTH [ 53 and 54 ].irect effects of PTH on osteoblasts and osteocytes, and indirect actions on osteoclasts, promote both bone formation and bone resorption, and the final effect on bone mass, either anabolic or catabolic, appear to depend on the duration and periodicity of the PTH exposure.