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The major target end organs for parathyroid hormone (PTH) action are the kidneys, skeletal system, and intestine. The primary response to parathyroid hormone (PTH) by the kidney is to increase renal calcium resorption and phosphate excretion.
And since calcitonin is antagonistic, does it inhibit osteoclast activity? (And would the opposite system apply to osteoblasts?) My reasoning is that PTH increases calcium levels in the blood, which means it induces osteoclast activity because osteoclasts break down bone and subsequently release calcium in the blood.