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The reduction in the rate of systolic ejection following elevation of aortic pressure leads to changes in cardiac output opposite to alteration in peripheral resistance, thus tending to maintain a relatively constant arterial pressure.
Because the mechanisms of cardiac-output-reserve (an increase in heart rate, blood volume, and cardiac contractility) are already a maximal use, there is a direct correlation with reduced exercise capacity.yperdynamic circulation. The cardiac effects of hyperthyroidism commonly cause increased preload with low systemic vascular resistance, a high heart rate, and increased cardiac muscle oxygen consumption. In the short term hyperthyroidism these effects result in an increased heart rate and high cardiac output.