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The reduction in the rate of systolic ejection following elevation of aortic pressure leads to changes in cardiac output opposite to alteration in peripheral resistance, thus tending to maintain a relatively constant arterial pressure.
Blood pressure varies with changes in blood volume, TPR, and cardiac output, which are determined primarily by venous return and neural and hormonal controls. Short-term mechanisms include both (1) neural and (2) hormonal controls, which alter blood pressure by changing peripheral resistance and CO.