Cardiac outflow obstruction may also result in sudden-onset syncope with little or no prodrome. One critical clue is the exertional nature, and the other is the presence of a cardiac murmur. Young athletes may present with this etiology for syncope.
SUMMARY. Summary: Flagellar and translocation-associated type III secretion (T3S) systems are present in most Gram-negative plant-and animal-pathogenic bacteria and are often essential for bacterial motility or pathogenicity.o date, T3S4 proteins have been described for translocation-associated T3S systems from several animal-pathogenic bacteria (YscP from Yersinia spp., Spa32 from Shigella spp., and InvJ from Salmonella spp.) and the plant-pathogenic bacterium X. campestris pv. vesicatoria (HpaC) (Table 6).
V. cholerae pathogenicity genes code for proteins directly or indirectly involved in the virulence of the bacteria. During infection, V. cholerae secretes cholera toxin, a protein that causes profuse, watery diarrhea.Colonization of the small intestine also requires the toxin coregulated pilus (TCP), a thin, flexible, filamentous appendage on the surface of bacterial cells.uring infection, V. cholerae secretes cholera toxin, a protein that causes profuse, watery diarrhea. Colonization of the small intestine also requires the toxin coregulated pilus (TCP), a thin, flexible, filamentous appendage on the surface of bacterial cells.