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The system exerts its influence via release or inhibition of vasopressin (antidiuretic hormone, ADH) which acts on the kidney to decrease water excretion. Deficiency of ADH is usually due to hypothalamic-neurohypophyseal lesions (central diabetes insipidus) or insensitivity of the kidney to ADH (nephrogenic diabetes insipidus). These patients, if untreated, have the predictable result of dehydration, hyperosmolality, hypovolemia, and eventual death in severe cases.
17 Rickets caused by renal disease (renal osteodystrophy) is caused by disturbances in calcium and phosphorus regulation and calcitriol production. Malabsorption syndromes such as celiac disease and cystic fibrosis can cause vitamin D deficiency.ITAMIN D DEPENDENT. Vitamin Dâdependent rickets, type I is secondary to a defect in the gene that codes for the production of renal 25(OH)D 3 â1-Î±-hydroxylase (Figure 125) . Vitamin Dâdependent rickets, type II is a rare autosomal disorder caused by mutations in the vitamin D receptor.