Among the risk factors for aSAH, clearly attributable and modifiable risks are very low body mass index, smoking, and high alcohol consumption.31,39,40 Yet, despite marked improvements in the treatment of hypertension and hyper- lipidemia and the decrease in rates of smoking over time, the incidence of aSAH has not changed appreciably in 30 It is possible that diet increases the risk of stroke in general and aSAH in particular. In an epidemiological study of Finnish smokers who were monitored for 13 years, in- creased consumption of yogurt (but not all dairy products) was associated with a higher risk of aSAH.41 Greater vege- table consumption is associated with a lower risk of stroke and aSAH.42 Higher coffee and tea consumption43 and higher magnesium consumption44 were associated with reduced risk of stroke overall but did not change the risk of aSAH. Predicting the growth of an individual intracranial aneu- rysm and its potential for rupture in a given patient remains problematic. When followed up on magnetic resonance im- aging, larger aneurysms ( 8 mm in diameter) tended to grow more over time,45 which implies a higher risk of rupture. 