Post CABG VT/VF The incidence of sustained VT or VF early after CABG is low, but these VAs are associated with high in-hospital VF occurring very early (intraoperatively or within 24 hours postoperatively) may be due to the transient effects of reperfusion, electrolyte and acid base disturbances, and the use of inotropes. Patients who present with VF or polymorphic VT in the postoperative period more often have associated ischemia, while pa- tients presenting with monomorphic VT usually have an old infarct and ventricular scar.S7.1.1.2-2 Polymorphic VT/VF occurring after CABG warrants a therapeutic approach targeting treatment of myocardial ischemia, including a possible need for assessment of graft patency, as well as identification and treatment of mechanical compli- cations and acute electrolyte or acid base disturbances. Risk factors for occurrence of monomorphic VT early af- ter CABG include prior MI, ventricular scar, LV dysfunc- tion, and placement of a bypass graft across a noncol- lateralized occluded coronary vessel to a chronic infarct Unlike polymorphic VT and VF, sustained monomorphic VT is typically not due to acute ischemia. Many of these patients have inducible sustained VT at electrophysiological study. Management of symptomat- ic VA in the early period after CABG follows the recom- mendations for acute and ongoing management of VT detailed elsewhere in this document. 