Index patient, a 34-year-old female, hailing from a middle socioeconomic status, was brought to the clinic with marked behavioral symptoms. History revealed that about 10 days back, she had an abrupt onset of blurring of vision, with tingling sensation on the left half of her body. This subsequently improved within 12 h after being given benzodiazepines. Prominent behavioral changes followed this. She became exceedingly talkative, started repeating the same phrases many times, and would often ask for food after eating. She was unable to remember the particulars of her son like which class he was studying in. Reportedly, she was unaware of date and day, which started improving after about 7 days of onset of her illness. Biological functions were within physiological limit. She was unable to perform her household activities during the period, though she was taking care of her basic personal care under supervision. There was no history of psychoactive substance use. History of past medical or surgical or psychiatric illness revealed hypertension and occasional headache but without any psychiatric illness. Her medication history revealed she was on oral contraceptive pills and antihypertensive. There was no past history of any mental illness. Family history revealed dementia in her paternal grandfather. On physical examination, no focal neurodeficit was observed. She was cooperative toward the examiner, yet appeared anxious, restless and had stereotyped repetition of certain phrases. Speech output was increased in volume, and she was answering the queries of the examiner in an occasional irrelevant manner. No psychotic symptom was elicited. Judgment and insight was maintained. Her hematological and biochemical profile on the day of visit was unremarkable [Table 1]. She was started on divalproex sodium 500 mg at night. Considering her acute onset blurring of vision, a neurological opinion was sought. She was advised for neuroimaging and multiplanar magnetic resonance images of brain through T2-weighted and fluid-attenuated inversion recovery sequences revealed acute nonhemorrhagic infarct in bilateral temporo-occipital region (PCA territory) [Figure 1]. At follow-up after 3 weeks, she was still more talkative and irritable, and there had been occasional aggressive outbursts without any new physical complaints. She was complaining of headache at times and was sleeping less than usual.