A 68-year-old male patient with a background history of hypertension presented with abdominal pain in the right upper quadrant associated with anorexia for 1-month duration. However, he did not report any fever, chills, altered bowel habits, melena, or per rectal bleeding. He had no previous abdominal surgeries. The general examination was unremarkable, devoid of any fever or tachycardia but the abdominal examination revealed a palpable liver extending beyond the costal margin. His basic blood biochemistry revealed an elevated leucocyte count of 16.8 x 103/microL with a C-reactive protein level of 9.4 mg/L and an elevated erythrocyte sedimentation rate of 119 mm/h. His liver and renal profiles were normal. Ultrasound abdomen revealed a hypoechoic lesion in the liver measuring 8.9 x 6.4 cm involving liver segments V, VI, and VII without internal vascularity suggestive of a liver abscess. Further imaging with triphasic CT of the chest, abdomen, and pelvis revealed a large irregular shaped loculated peripheral enhancing, centrally hypo-attenuated fluid density lesion measuring 8.3 x 10.8 x 6.1 cm in size involving segment IVA and IVB of the liver suggestive of a liver abscess with mass effect on the common hepatic artery branches (Figure 1). The CT did not show any features of intrahepatic or extrahepatic biliary obstruction. The collection was drained under ultrasound guidance with a pigtail catheter. Aspirated purulent fluid investigations confirmed a liver abscess. The full report revealed 17,600 mm3 of pus cells and protein of 9000 mg/dL with a significant growth of Escherichia coli, suggestive of the diagnosis of a liver abscess with cytology report confirming the diagnosis. The mycobacterial investigations for tuberculosis, amoebiasis, and Burkholderia pseudomallei, three important causes for liver abscess in South Asian region were negative. Upon further investigations, an elevated HbA1c of 6.4% in the pre-diabetic range was found although FBS was normal. Echocardiography was normal without any features to suggest endocarditis. The investigation for a cause continued with upper GI endoscopy, which was normal. However, interestingly, the colonoscopy revealed a denture impacted at the sigmoid colon adjacent to multiple diverticula. There was no endoscopic feature of diverticulitis. The patient was unaware of having swallowed a denture. The denture was removed carefully using a snare and foreign body forceps. There was superficial ulceration at the site of impaction but no obvious perforation seen on careful examination. It was thought that a microperforation would have spontaneously healed during the delayed personation. The patient was treated with intravenous cefotaxime for 2 weeks according to the sensitivity reports. The patients had an uneventful recovery. He discharged following 2-week hospital stay. Serial follow-up ultrasound scans revealed a resolving abscess. As the denture had only plastic attachments, it was difficult to identify on the previous CT scans; however, there was evidence of colonic perforation from a foreign body depicted in the CECT images retrospectively (Figure 2).