A 15-month-old child, born to non-consanguineous parents, presented to us with complaints of regression of milestones since the last 3 months. No history of fever, vomiting, seizure, weakness of any limbs, ear discharge, feeding difficulty or choking episodes, cough, or weight loss was observed. There was no history of any episode of loss of consciousness or hospitalization. There was a history of contact with pulmonary TB. General examination was essentially normal. On examination of the CNS, the child looked drowsy with right-sided third and seventh cranial nerve palsy, tone and power in all four limbs were grade 4/5 with brisk deep tendon reflexes and bilaterally extensor plantar response. No signs of meningeal irritation were observed with a normal fundus examination. Non-contrast computed tomography (NCCT) of brain carried out elsewhere before admission showed gross hydrocephalus with multiple hypoechoic lesions in bilateral parietal regions and around the brain stem. We kept our provisional diagnosis as brain abscess or intracranial space-occupying lesion. Ventriculoperitoneal (VP) shunting was carried out by the neurosurgery department but abscess drainage could not be performed due to the possibility of ventriculitis owing to the proximity of the abscesses to the ventricular system. The child was started on broad-spectrum antibiotics in antimeningitic doses, and simultaneously TB workup was carried out. Cerebrospinal fluid (CSF) study (conducted during the placement of VP shunt) neither showed any pleocytosis nor high protein and low sugar. Gastric aspirate for cartridge-based nucleic acid amplification test (CBNAAT) and acid-fast bacilli (AFB) was negative but Mantoux test was positive (12 x 13mm). Chest X-ray showed a miliary pattern. Contrast-enhanced magnetic resonance imaging (CEMRI) of the brain showed multiple thick-walled irregular lesions in the cerebrum, cerebellum, and brain stem [Figure 1A and B]. Magnetic resonance spectroscopy (MRS) showed a high lipid lactate peak. Parental screening for TB confirmed pulmonary TB in the mother too. Hence, ATT along with steroids was started with the diagnosis of multiple TBAs causing obstructive hydrocephalus. After few days of starting ATT, the child developed isoniazid-induced hepatitis, and she was given modified ATT regimen with rifampicin, pyrazinamide, and ethambutol with levofloxacin. Subsequently, she tolerated feeds, gained weight, and attained milestones. Right-sided ptosis and weakness also improved gradually. On follow-up after 5 months, the child was walking with support, achieved pincer grasp, uttering disyllables, feeding normally, and there was no focal neurological deficit. Repeat NCCT of brain [Figure 2] also showed resolution of brain abscesses.