Case 1: a seven-year-old girl, followed for 5 months for lymph nodes tuberculosis on anti-TB treatment, which was revealed by suppurating then fistulated jugular-carotid and supraclavicular lymph nodes and confirmed by ganglionic biopsy which objectified a tuberculous granuloma. Clinical evolution was marked by the appearance of generalized tonic-clonic seizures with intracranial hypertension syndrome made of treatment-resistant headaches and vomiting, all evolving in a context of fever and alteration of the general state. On admission, she was febrile (39 C), had lost 6kg in 5 months and had small cervical lymphadenopathies. Cerebral MRI objectified necrotic nodules in left hemisphere with edema and meningeal granulations (Figure 1). Biological examinations showed an accelerated sedimentation rate, microcytic hypochromic anemia, and neutrophilic leukocytosis. The intradermal reaction to the tuberculin was positive. The cyto-chemical and bacteriological analysis of the CSF objectified hypoglycorrhachic lymphocytic meningitis. The diagnosis of the cerebral tuberculoma was suggested in front of the clinical context and radiological criteria. The diagnosis of tuberculous abscess was also suspected. The surgical approach of the lesions was direct with complete excision. The histological study showed a brain parenchyma modified by a polymorphic inflammatory infiltrate with epithelioid and giant cell granuloma with caseous necrosis in favor of tuberculoma. The patient continued her anti-TB treatment. Short-course low-dose corticosteroid therapy (1mg/kg/d) was prescribed in front of perilesional edema and associated meningeal involvement. Clinical evolution was marked by the appearance of 3/5 spastic hemiparesis postoperatively recovered after 1 month of motor kinesitherapy with good clinical improvement.