A healthy 14-year-old boy with a history of contact with dogs presented with reduced vision in his right eye. Best-corrected visual acuity (BCVA) on first presentation was 6/36 in the right eye and 6/5 in the left eye. Fundoscopy revealed bilateral granulomatous chorioretinitis. Serological testing was subsequently conducted to identify the underlying cause for the condition and included analysis for toxoplasmosis, Toxocara, syphilis, sarcoidosis, tuberculosis, rheumatoid factor, antinuclear antibody, anti-neutrophil cytoplasmic antibodies, and Lyme disease. Serological Toxocara testing was performed using ELISA (enzyme-linked immunosorbent assay) technique. The results of these tests were all negative. Plain film chest X-ray and high-resolution chest computed tomography were also performed with no pathology identified, specifically demonstrating no lymphadenopathy to suggest sarcoidosis and no signs of pulmonary tuberculosis. The patient re-presented 2 weeks later with a reduction in vision in his left eye (BCVA was 6/12). Fundoscopy at this stage showed bilateral peripapillary subretinal hemorrhages with subretinal hemorrhage extending into the left macula (Figure 1). Optical coherence tomography scan of the left macula showed macular edema secondary to the subretinal hemorrhage (Figure 2). Fluorescein angiography demonstrated bilateral peripapillary choroidal neovascular membranes (CNVM), a rare complication of granulomatous cho-rioretinitis (Figure 2). The patient received treatment of monthly intravitreal injections of ranibizumab over a period of 3 months in each eye. BCVA in the left eye improved from 6/12 to 6/5 with regression of the CNVM and resolution of sub-macular blood clearly visible on clinical examination and repeated optical coherence tomography. BCVA in the right eye also improved from 6/36 to 6/18 post-treatment. As the clinical findings were highly suspicious of toxocariasis despite negative Toxocara serology we decided to investigate further by obtaining intraocular fluid samples from both the anterior and posterior chambers for Toxocara polymerase chain reaction (PCR). Since Toxocara PCR was not available in any pathology laboratory in Australia we sent the samples to a research laboratory at the Faculty of Veterinary and Agricultural Science, University of Melbourne, to perform the test. The test returned negative for Toxocara. The patient did not receive anti-helminthic treatment because there was no systemic involvement and no live larva was seen on fundoscopy. The inflammation in ocular toxocariasis is usually a result from the body's response to the dead worm.