A 38-year-old Jordanian lady, with no known chronic illness, presented the 3rd time to emergency department with worsening symptoms of high grade fever, productive cough, exertional shortness of breath and dizziness for 10 days. Patient had positive sick contact with her kids whom suffered from upper respiratory tract infection symptoms. On her previous two visits to the emergency departments, she was vitally stable and treated as a case of community acquired pneumonia with oral antibiotics - Amoxicillin / clavulanic acid on the first visit and azithromycin on the 2nd visit, however, she was unable to complete the treatment due to multiple vomiting episodes, thus, she presented for the 3rd time in 10 days to the emergency department with worsening symptoms. She looked septic with oral temperature of 38.5 degree celsius, blood pressure of 86/51 mmhg, heart rate of 108 beats/minute, tachypenic with respiratory rate of 26 breaths / minutes, peripheral oxygen saturation of 93% on room air. Chest auscultation was remarkable for bilateral diffuse crackles on middle and lower lobes with scattered wheezes, other systematic examination was unremarkable. Lab test results showed markedly elevated C-reactive protein of 295 mg/L with negative procalcitonin of 0.47 ng/ml (Table 1). X-ray of the chest - PA view (Fig. 1) showed worsening bilateral lower zones infiltrates more pronounced on the right lower zones as compared to her 1st ED visit. Patient was treated as a case of sepsis secondary to community acquired pneumonia with intolerance to oral therapy. Her blood pressure improved with fluid resuscitation and she was commenced initially on IV ampicillin/ sulbactum plus azithomycn with symptomatic treatment. Patient continued to spike high grade fever with no clinical improvement for which antibiotics escalated to IV piperacillin/tazobactam on day 4 of admission and further work up was requested. High resolution CT scan was done and revealed bilateral basal air space opacities with peri bronchovascular distribution and centrilobular nodules with branching pattern suggestive of tree in bud predominantly in lower lobes (Fig. 2). Tests for tuberculosis from the sputum and autoimmune disorders did not yield any positive results. Bronchoscopy (Fig. 3) was done on day 9 of admission and showed inflamed looking airways, especially right upper lobe secondary carina with inflamed submucosal and mucosal changes. Broncho-alveolar lavage (BAL) was positive for candida albicans but negative for TB work up. Her BAL cell count differential revealed 36% Neutrophils and 64% Macrophages with no eosinophil or Lymphocytes present. The endobronchial biopsies revealed a rare non-necrotizing granuloma. She had negative sputum and blood cultures apart from persistently positive candida albicans isolated from the sputum. She had no clinical response to 6 days of IV piperacillin/tazobactam on day 10 of admission, she was therefore started on anidulafungin to treat a probable case of candida pneumonia infection after a thorough discussion with the infectious disease team in the hospital. She fortunately improved dramatically, this was reflected in both her clinical and biochemical status within the first 24hrs of starting anidulafungin (Fig. 4) which she continued intravenously for 4 days then discharged on oral fluconazole for another 20 days based on the susceptibility results. She showed complete recovery upon her post discharge follow up both clinically and radiologically (Fig. 5). Notably, our patient has been previously healthy with no preceded history of acute infection or visits to healthcare facilities prior to her acute illness, she has 5 kids and all delivered via normal vaginal delivery and her last child is 10 months old. She has naive surgical history and had not been on any acute or chronic medications besides no prior history of candida infections.