A 26-year-old Nepalese lady with no previous medical history presented with chief complaints of headache and left-sided monocular blurry vision for one week duration. The headache was located in the occipital region and was intermittent with no aggravating or relieving factors. There was no fever, nausea, vomiting or photophobia. She denied any sick contacts, animal exposure, history of recent infections, night sweats, weight loss, loss of appetite or change in bowel habits. Family history was negative, and her last international travel was to Nepal four months prior to presentation. She was married, had one child, and worked in catering department. Of note, there was no handling of pork meat as part of her job. Vital signs were normal and cranial nerve examination revealed left-sided ptosis and dilated left pupil with sluggish reaction to light. There was mild impairment in adduction, however other extraocular movements were preserved. There were no sensory or motor deficits in any of the extremities and meningeal signs were absent. Rest of the systemic examination was normal. Laboratory workup revealed normal complete blood count, renal and liver function tests. Her inflammatory markers including C-reactive protein and ESR were also normal. Computed tomography (CT) scan of the brain showed a focal hyperdense calcified lesion measuring 6 mm in the midbrain and a calcified focal lesion measuring 5 mm in the left frontal lobe. There was no midline shift or hydrocephalus, and the lesions were interpreted as calcified granulomas (Fig. 1). Magnetic resonance imaging (MRI) of the brain showed a midbrain ring-enhancing lesion with central nodular calcification and significant peri-lesional edema. Another ring-enhancing nodular calcified lesion without edema was reported in the left parasagittal frontal lobe giving an overall impression of granulomatous disease consistent with NCC (Fig. 2). Due to these findings, enzyme-linked immunoelectrotransfer blot (EITB) for anticysticercal antibodies was sent and was reportedly positive. Neurology and infectious disease teams were consulted, and the patient was started on a tapering regimen of dexamethasone for 14 days, in addition to albendazole and praziquantel. The patient was discharged four days later after improvement in ptosis and blurry vision. Unfortunately, post-discharge follow up was not established as the patient travelled back to her home country.