A 65-year-old lady presented with cough, night sweats and exertional breathlessness of two months' duration. There was no history of weight loss or fever. Her past history was significant for hypertension and non-productive cough two years before that was evaluated at another center. CT had shown right paratracheal and bilateral hilar lymphadenopathy along with upper lobe-predominant micronodules. Tuberculin skin testing was negative and endobronchial ultrasound-guided needle aspiration (EBUS-FNA) and transbronchial lung biopsy (TBLB) were performed. EBUS-FNA showed few non-caseating granulomas and TBLB was non-contributory. A presumptive diagnosis of tuberculosis was made and four-drug anti-tuberculosis treatment (ATT) was administered for six months with improvement in cough. Examination was unremarkable during current evaluation. There was no exposure to pets, drugs or occupational dusts. Chest radiography was normal. Spirometry was normal and diffusion capacity showed mild reduction. HRCT showed profuse random micronodules without lymphadenopathy and an upper-lobe and peripheral predominance that was visualized better with maximum intensity projection (MIP, Figure 1). Renal function tests, serum calcium and angiotensin-converting enzyme (ACE) levels were normal. Fibreoptic bronchoscopy (FOB) was done with bronchoalveolar lavage (BAL) and TBLB; BAL Fluid was negative for acid-fast bacilli and Xpert MTB/RIF and TBLB showed interstitial inflammation only. Surgical lung biopsy was performed and showed extensive perivascular non-caseating granulomas (Figure 2). BAL mycobacterial cultures were subsequently reported as negative. She was started on steroids with resolution in cough and breathlessness by second month of treatment. She remains asymptomatic and off steroids after one year of treatment.