In this task, you are given a text of many news articles seperated by special token "|||||". Your task is to summarize them.
Q: Smokers who survive their habit into old age may hold the key to better lung health for all, according to a Medical Research Council-funded (MRC) study involving researchers at The University of Nottingham and the University of Leicester. 
  
 Using genetic data from participants in the UK Biobank*, the research team has identified genetic differences which affect the likelihood of whether or not a person will smoke, and the predisposition of both heavy smokers and non-smokers to suffer from poor lung health. 
  
 The new discoveries may one day help scientists develop better treatments for diseases such as chronic obstructive pulmonary disease (COPD), a life-threatening condition which affects almost one million people in the UK. The findings could also help improve interventions aimed at helping smokers to give up. 
  
 The findings are presented today (Monday) at this year's European Respiratory Society (ERS) meeting in Amsterdam and are published in the Lancet Respiratory Medicine. 
  
 The lead researchers, Professor Ian Hall at The University of Nottingham, and Professor Martin Tobin from the University of Leicester, studied the lung health of approximately 500,000 UK Biobank participants. 
  
 A subset of 50,000 of these participants were selected based on their lung health and whether or not they were heavy smokers or had never smoked. The researchers then compared these factors with 28 million genetic variants in each participant, making this study one of the most detailed of its kind. The team were able to find parts of the human genome never before associated with a person's lung health, as well as five sections of DNA shown for the first time to relate to being a heavy smoker. 
  
 The discoveries help to explain why some people can have relatively good lung health, despite smoking, and why some can suffer from lung conditions even if they have never smoked before. Knowing why they are more likely to develop lung disease or to become heavy smokers is important for developing treatments for these diseases and for helping smokers to quit. 
  
 Professor Ian Hall said: "The drugs we use to prevent or treat diseases target the proteins in our bodies, and our genes influence the production of proteins. Understanding how the genes are involved in disease or in addiction to tobacco, can help us design and develop better and more targeted treatments that are likely to be more effective and have fewer side effects. UK Biobank was a bold vision when it was set up and this study shows just how much can be achieved by using the resource. We hope to get much further detail when genetics information on all UK Biobank participants becomes available next year." 
  
 Professor Martin Tobin said: "Smoking is the biggest lifestyle risk factor for COPD. Many, but not all, smokers develop the disease. Genetics play a big part, as they do in smoking behaviour. Our research helps to tell us why, paving the way for improved prevention and treatment. Stopping smoking is the best way to prevent smoking-related diseases such as COPD, cancers and heart disease." 
  
 The scientists hope to expand the study to look at the genetics of all 500,000 participants when the data become available in 2016. 
  
 Dr David Crosby, who leads on experimental medicine at the MRC, said: "UK Biobank was set up by the MRC and other funders for just this type of important research. Thanks to the 500,000 people who have volunteered to make this resource the biggest in Europe, researchers will have access to a huge amount of information about the part that environment and genetics play in disease. The genetics work done by this group will be incorporated into UK Biobank for other researchers working in different areas to use in their own studies, bringing closer the promise of new treatments and drugs for a whole range of diseases and conditions." 
  
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 The study was conducted by the UK BiLEVE - UK Lung Exome Variant Evaluation team of researchers. 
  
 Paper: Novel insights into the genetics of smoking behaviour, lung function and chronic obstructive pulmonary disease in UK Biobank by Louise V Wain et al is published in the Lancet Respiratory Medicine. ||||| Image copyright Thinkstock 
  
 The mystery of why some people appear to have healthy lungs despite a lifetime of smoking has been explained by UK scientists. 
  
 The analysis of more than 50,000 people showed favourable mutations in people's DNA enhanced lung function and masked the deadly impact of smoking. 
  
 The Medical Research Council scientists say the findings could lead to new drugs to improve lung function. 
  
 Media playback is unsupported on your device Media caption Professor Martin Tobin is one of the researchers behind the findings 
  
 But not smoking will always be the best option, they say. 
  
 Many, but not all, smokers will develop lung disease. But so too will some who have never touched a cigarette in their lives. 
  
 The researchers analysed the huge amount amount of health and genetic data from volunteers to the UK's Biobank project. 
  
 Breathe easily 
  
 They looked at Chronic Obstructive Pulmonary Disease (COPD) which leads to breathlessness, coughing and repeat chest infections. 
  
 The condition is thought to affect three million people in the UK and includes diseases such as bronchitis and emphysema. 
  
 By comparing smokers and non-smokers as well as those with the disease and without they discovered sections of our DNA that reduce the risk of COPD. 
  
 So smokers with "good genes" had a lower risk of COPD than those with "bad genes". 
  
 Image copyright SPL 
  
 Prof Martin Tobin, one of the researchers at the University of Leicester, said the genes seemed to affect the way the lungs grow and respond to injury. 
  
 But he told the BBC News website: "There doesn't appear to be any kind of magic bullet that would give anyone guaranteed protection against tobacco smoke - they would still have lungs that were unhealthier than they would be had they been a non-smoker. 
  
 "The strongest thing that people can do to affect their future health in terms of COPD and also smoking-related disease like cancer and heart disease is to stop smoking." 
  
 Addiction 
  
 The habit also increases the risk of heart disease and cancers, which are not considered in this study. 
  
 The scientists also uncovered parts of the genetic code which were more common in smokers than non-smokers. 
  
 They seem to alter the brain's function and how easily someone can become addicted to nicotine, although that still needs to be confirmed. 
  
 Prof Tobin said the findings offered "fantastic new clues about how the body works that we really had little idea about before and it's those things that are likely to lead to some really exciting breakthroughs for drug development." 
  
 Their findings were presented at a meeting of the European Respiratory Society and published in the Lancet Respiratory Medicine journal. 
  
 Ian Jarrold, the head of research at the British Lung Foundation, said: "These findings represent a significant step forward in helping us achieve a clearer picture about the fascinating and intricate reality of lung health. 
  
 "Understanding genetic predisposition is essential in not only helping us develop new treatments for people with lung disease but also in teaching otherwise healthy people how to better take care of their lungs." |||||
A: 
Smokers could soon benefit from new drugs that improve the function of their lungs, owing to the lucky few who smoke for years with little consequence. UK researchers scanned 50,000 smokers and non-smokers, including some who suffer from chronic obstructive pulmonary disease, and identified gene mutations that enhance lung function. Basically, smokers with these "good genes" had a lower risk of COPD while those with "bad genes" were more likely to develop the disease. This helps explain why non-smokers sometimes get sick, reports the Guardian. A researcher tells the BBC the genes affect how lungs grow and react to damage. But "there doesn't appear to be any kind of magic bullet that would give anyone guaranteed protection against tobacco smoke," he adds. A smoker with good genes "would still have lungs that were unhealthier than they would be had they been a non-smoker." The study, which involved scanning 28 million genetic variants in each participant, also identified five sections of DNA more commonly found in smokers that may affect how easily a person can become addicted to nicotine. That finding, however, still needs to be confirmed. The study offers "fantastic new clues about how the body works that we really had little idea about before and it's those things that are likely to lead to some really exciting breakthroughs for drug development," says a researcher. More specifically, understanding how a person’s genes relate to disease and tobacco addiction can help scientists "design and develop better and more targeted treatments that are likely to be more effective and have fewer side effects," a scientist adds. Researchers hope to expand the study group to 500,000 in 2016, per a release. (A similar study found some smokers may have genes that repair damage.)