broken strong sacrificial bonds $\phi_{strong,f}$ (i.e. bonds with $\lambda_S > \lambda^{*}_{S}$ that break) as a function of the distance from the BDT $\Delta\tilde{\alpha}$ for various amounts of disorder $\delta \lambda$. At low $\delta\lambda$ and far below the BDT, failure of strong sacrificial bonds is required in order to achieve macroscopic failure, since there are just not enough weak sacrificial bonds (Fig. \[fig:StressConc\](b)). However, upon approaching the transition at $\Delta\tilde{\alpha}=0$, $\phi_{strong,f}$ grows, even though a decrease could be expected based on the total concentration of strong sacrificial bonds (Fig. \[fig:StressConc\](c)). Interestingly, we observe a maximum in $\phi_{strong,f}$ before fully entering the ductile regime $\Delta\tilde{\alpha}>0$ where mostly weak sacrificial bonds control the fracture process. Strong bonds can only break if the force they carry exceeds the maximum force that is expected based on the macroscopic force balance. Thus, we conclude that the network structure plays a crucial role in (re)distributing the load during deformation, giving rise to stress concentration. The development of stress heterogeneity is tightly bound to disorder that is present in the initial structure. We expect that by including more disorder in the network (e.g. with inhomogeneities in the connectivity of the nodes or in the stiffness of the bonds) 