in the absence of the “incidental capacitor” effect, results in the fastest possible termination of the re-entry at the apex of the heart, before the vortex first rotation ever started. The re-entry termination time here is more than twice shorter than in the raw and “edited” MRI isotropic simulations shown in Fig. \[3D\_X\_iso\] and Fig. \[3D\_X\_shaven\_iso\], shorter than in the analogous simulation with the “incidental capacitor” effect shown in the Fig. \[3D\_X\_aniso\], and times shorter than in any of the simulations of the re-entry with the perpendicular initial location shown in the Fig. \[3D\_iso\], Fig. \[3D\_aniso\], Fig. \[3D\_shaven\_iso\], and Fig. \[3D\_shaven\_aniso\] .]{}

In Fig \[Table1\], we have summarized the results of the simulations shown in Fig. \[3D\_iso\], Fig. \[3D\_aniso\], Fig. \[3D\_X\_iso\], Fig. \[3D\_X\_aniso\], Fig. \[3D\_shaven\_iso\], Fig. \[3D\_shaven\_aniso\], Fig. \[3D\_X\_shaven\_iso\], and Fig. \[3D\_X\_shaven\_aniso\], with the re-entry termination time shown in arbitrary time units under each respected whole heart model and initiation cite panel. It can be seen that the realistic anisotropy of the heart causes at least twice faster termination of re-entry. It also can be seen that indeed the present in the raw DT-MRI model leftover piece of tissue connected to the apical region of the heart has served as 