“leftover” piece “incidental capcitor” effect, there is just a bit faster repolarisation of the whole heart than it was in the presence of the “incidental capcitor” in the raw DT-MRI simulation shown in Fig. \[3D\_aniso\]. [For the comparison of the re-entry termination times, and the whole heart recovery times, see FIG. \[Table1\]]{}. ]{}

In the 3D whole heart “edited” MRI model simulations shown in the Fig. \[3D\_X\_shaven\_iso\]

![image](Fig9small.pdf)

and Fig. \[3D\_X\_shaven\_aniso\],

![image](Fig10small.pdf)

a counter-clockwise excitation vortex was initiated by the phase distribution method [@chaos], with the initial position of the transmural vortex filament (yellow line) at the prescribed location [*along the $y$ axis*]{} at $x_0=40, z_0=60$. [It can be seen in Fig. \[3D\_X\_shaven\_iso\] isotropic, and in Fig. \[3D\_X\_shaven\_aniso\] anisotropic 3D simulations, that at $t=0$, there was the identical initial location of the filament of the excitation vortex: that is transmurally, roughly in the middle through the ventricles of the foetal heart, perpendicular to the initial location of the vortex filament in the “edited” MRI simulations shown in Fig. \[3D\_shaven\_iso\] and Fig. \[3D\_shaven\_aniso\], and similar to the initial location of the vortex filament in the raw DT-MRI simulations shown in Fig. \[3D\_X\_iso\] and Fig. \[3D\_X\_aniso\] . ]{}

[Fig. \[3D\_X\_shaven\_iso\] shows the 