### RNU6-604P Gene Overview

**Gene Symbol**: RNU6-604P  
**Type**: Gene associated with non-coding RNA, specifically a member of the U6 small nuclear RNA family, which plays a crucial role in the splicing of pre-mRNA in the spliceosome.

#### 1. Associated Genes
RNU6-604P is part of a larger family of small nuclear RNAs, primarily involved in RNA splicing. While the specific association with other genes may not be extensively characterized due to its status as a pseudogene, genes within the U6 RNA family (e.g., RNU6-1) and associated splicing factors (such as SNRNP complexes) could be considered related in terms of functional pathways.

#### 2. Aging and Age-Related Diseases
There is limited specific data about RNU6-604P's direct involvement in aging or age-related diseases. Still, small nuclear RNAs, including U6, are fundamental to mRNA processing and hence can be indirectly linked to aging processes. Dysregulation of RNA splicing is associated with various age-related diseases, such as cancer and neurodegenerative disorders. Specific studies are required to elucidate any direct association with RNU6-604P.

#### 3. Drug Interactions
As of now, there are no specific drugs or drug classes known to directly target or interact with the RNU6-604P gene. However, drugs influencing overall RNA splicing mechanisms may impact the broader family of small nuclear RNAs, including U6 and its paralogs. 

#### 4. Pathways and Biological Processes
RNU6-604P primarily participates in:

- **mRNA Splicing**: Integral to the spliceosome's function, catalyzing the removal of introns from pre-mRNA.
- **Gene Regulation**: Non-coding RNAs are known to play roles in the regulation of gene expression at various levels.
- **RNA Processing**: Involved in the broader aspect of RNA processing and modification necessary for mature mRNA's functionality.

### Summary 
RNU6-604P functions within the context of RNA splicing alongside other small nuclear RNAs, with indirect implications for gene regulation and disease processes associated with misregulated splicing mechanisms. Further research would be needed to clarify its roles specifically in aging and drug interactions, as well as to elucidate any more precise relationships with other genes in the context of health and disease.