The NRBF2P3 gene is part of the NRBF2 gene family. Given the nature of the pseudogene designation "P3," it suggests that this gene may not encode a functional protein. Pseudogenes often arise from the duplication or mutation of functional genes but frequently lose the ability to code for proteins through the accumulation of mutations.

### 1. Associated Genes
While specific associations can vary by study and database, pseudogenes like NRBF2P3 may have homology or connections to functional genes in the NRBF2 family or other related genes involved in cellular processes. Unfortunately, the specific associated genes are not always detailed in standard databases, but it's advisable to check the latest genomic databases (such as Ensembl or NCBI) or literature searches for any evolutionary relationships or regulatory functions linked to other genes.

### 2. Aging or Age-Related Diseases
Scientific literature on the direct role of NRBF2P3 in aging or age-related diseases specifically is limited, given its status as a pseudogene. However, research into the NRBF2 gene has shown it could be involved in cellular maintenance processes. Alterations in cellular maintenance pathways (like autophagy or lipid metabolism) may indirectly suggest roles of similar pseudogenes in longevity or age-related conditions. The latest studies would need to be referenced to ascertain any established connections.

### 3. Drug Interactions
As of the latest information available, there are no known drugs or drug classes that specifically interact with the NRBF2P3 gene, given its status as a pseudogene. Drug interaction that involves the functional NRBF2 gene or related pathways may indirectly be useful in research, but direct pharmacological relevance tends to focus on functional genes.

### 4. Pathways and Biological Processes
Similar to other pseudogenes, NRBF2P3 may not directly participate in canonical metabolic or signaling pathways. The primary NRBF2 gene has been associated with autophagy and lipid metabolism pathways. If there were to be any influence by the NRBF2P3 pseudogene, it would likely be through regulatory mechanisms or non-coding RNA aspects. Research in genomics often highlights that pseudogenes may influence expression patterns or act in a regulatory capacity, but for specifics, direct studies must be consulted.

In conclusion, while NRBF2P3 is not expected to have direct functional contributions, the surrounding genes and pathways will likely have more relevance in research concerning aging and associated pathways. Further research literature should be consulted for the latest findings about the gene's biological implications, especially if any studies have been published since October 2023.