The **NDUFB1P1** gene is a pseudogene associated with the mitochondrial complex I, specifically related to the NADH:ubiquinone oxidoreductase component. Below I have compiled the requested details based on known literature and available databases.

### 1. Associated Genes
While specific interactions or associations with other genes may not be extensively documented for **NDUFB1P1**, it shares similarities with genes associated with mitochondrial function and oxidative phosphorylation. Notably, its counterpart, **NDUFB1** (NADH:ubiquinone oxidoreductase core subunit B1), may be the most relevant gene in this context, as both are involved in the respiratory electron transport chain.

Additionally, it may have indirect associations with other genes involved in mitochondrial processes such as those in the OXPHOS (oxidative phosphorylation) pathway, including:
- **NDUFA1**
- **NDUFA8**
- **NDUFC1**

### 2. Aging and Age-Related Diseases
The **NDUFB1P1** gene, being a pseudogene of the mitochondrial complex, raises interest in studies associated with mitochondrial dysfunction, which has been linked to several aging-related diseases, including:
- **Neurodegenerative Disorders:** Conditions such as Alzheimer's disease and Parkinson's disease show an imbalanced mitochondrial function that can be linked to mutations or anomalies in complex I components.
- **Metabolic Disorders:** Impaired mitochondrial function is also implicated in metabolic syndromes that often correlate with aging.
- **Mitochondrial Myopathies:** Abnormalities in mitochondrial genes can lead to muscle weakness and exercise intolerance, prominent in aging populations.

While direct causal links between **NDUFB1P1** and these diseases might be less documented due to its pseudogene status, its association with **NDUFB1** and the broader context of mitochondrial function suggests a potential relevance.

### 3. Drug Interactions
There are no specific drugs known to interact directly with **NDUFB1P1** due to its pseudogene designation and limited functional activity. However, drugs that target mitochondrial function and modulate oxidative phosphorylation might indirectly relate to conditions where **NDUFB1** and its pathways are relevant. Classes of such drugs might include:
- **Mitochondrial inhibitors:** Such as rotenone, which affects complex I.
- **Antioxidants:** Drugs that aim to alleviate oxidative stress in mitochondria may also indirectly influence pathways associated with this component of the electron transport chain.

### 4. Pathways and Biological Processes
The **NDUFB1P1** gene itself is not widely involved in specific pathways due to its nature as a pseudogene. However, it can be associated with the following biological processes due to its relationship with mitochondrial function:

- **Oxidative Phosphorylation:** It is a critical component in the respiratory electron transport chain.
- **Mitochondrial Biogenesis:** Involved in pathways that regulate the generation and maintenance of mitochondria.
- **Cellular Energy Metabolism:** Plays a role in ATP generation via oxidative phosphorylation.
- **Mitochondrial Electron Transport:** Participates in the transfer of electrons from NADH to ubiquinone, leading to proton gradient formation.

In summary, while **NDUFB1P1** is a pseudogene and does not express a functional product, its association with mitochondrial biology and its peers encapsulate considerable biological significance, particularly concerning aging and associated disorders.