The **SNORD36A** gene (also known as small nucleolar RNA, C/D box 36A) encodes a specific type of small nucleolar RNA (snoRNA), which is predominantly involved in the modification of ribosomal RNA (rRNA) and other non-coding RNAs through methylation.

### 1. Associated Genes
While specific interactions for SNORD36A are not heavily documented, snoRNAs often participate in the regulation of the expression or function of multiple RNA species. Other genes that may be associated with SNORD36A in functional or regulatory contexts include:
- **H/ACA snoRNA genes**
- **Other C/D box snoRNAs** (e.g., SNORD42, SNORD68)
- **rRNA genes**, specifically those associated with ribosome biogenesis.

### 2. Aging or Age-related Diseases
Current literature provides limited information directly linking SNORD36A to aging or specific age-related diseases. However, snoRNAs in general have been implicated in:
- The regulation of cellular processes that can affect aging, such as ribosome biogenesis and the maintenance of RNA integrity.
- Some studies suggest that disruptions in snoRNA expression may be connected with age-related diseases like cancer, but direct evidence for SNORD36A specifically is sparse and necessitates further research.

### 3. Drug or Drug Classes Interaction
As of the current knowledge up to October 2023, there are no well-documented drugs or drug classes specifically known to interact with SNORD36A. This snoRNA's activity does not directly guide drug interactions as would be typical with coding genes. However, general research into snoRNA functions could eventually lead to insights into therapeutic strategies.

### 4. Pathways and Biological Processes
The **SNORD36A** gene is primarily involved in:
- **Ribosome biogenesis**: It plays a role in the modifications of rRNA during the assembly of ribosomes.
- **Gene regulation**: Necessary for the proper maturation of rRNA and possibly influencing the transcription of other genes through effects on rRNA processing.

It is important to note that the snoRNA gene family functions as a broader network, and specific experimental data may elucidate detailed pathways involving SNORD36A as research progresses. 

### Conclusion
In summary, while SNORD36A is an important component of cellular machinery concerning rRNA modification, direct associations with aging, specific diseases, and drug interactions require further exploration. Ongoing research into the roles of snoRNAs will continue to clarify their biological significance and potential therapeutic implications.