### RASL10A Gene Overview

The **RASL10A** gene encodes a protein that has been predicted to possess GTPase activity and is thought to play a role in small GTPase-mediated signal transduction. This gene is associated with cellular mechanisms related to proliferation inhibition and is localized within the nucleolus and plasma membrane.

### 1. Associated Genes
While the provided summaries do not mention specific genes associated with RASL10A, it is often useful to consider its relation to other genes within the same pathways or networks. Genes commonly associated with signaling pathways that include GTPases, like RASL10A, may include:

- **RAS** family genes (KRAS, NRAS, HRAS)
- **RHEB** (Ras homolog enriched in brain)
- **RAP1A** (Ras-related protein 1A)
- **RAN** (Ras-related nuclear protein)

To find more specific associations, extensive genome-wide studies or pathway analyses may reveal additional interacting genes.

### 2. Aging and Age-Related Diseases
Research related to RASL10A's role in aging or age-related diseases is limited, but the general functions of GTPases often intersect with mechanisms of cellular aging and proliferation:

- **Cell Proliferation Regulation**: The RAS family, including RASL10A, is known to be involved in pathways that regulate cell growth and differentiation, which can impact senescence and tissue regeneration.
- **Cancer Connection**: The inhibitory effect on cellular proliferation might implicate RASL10A in tumorigenesis, particularly in aging populations where cancer incidence is higher.

It would be prudent to monitor research literature for ongoing studies that explore its role in aging or specific age-related disorders.

### 3. Interacting Drugs and Drug Classes
As of my last training data, specific drugs that directly interact with RASL10A are not widely documented in the literature. However, due to its role as a GTPase and potential involvement in proliferation signaling pathways, it could be influenced by:

- **Chemotherapeutic Agents**: Drugs targeting RAS signaling may indirectly affect RASL10A behavior.
- **Small Molecule Inhibitors**: These may target related GTPases or broader signaling pathways affecting cell proliferation.

For precise drug interactions, pharmacological databases should be consulted as studies emerge.

### 4. Pathways and Biological Processes
RASL10A is predicted to be involved in several key biological processes and pathways:

- **Signal Transduction**: Involved in pathways mediating growth factor signaling and transduction through small GTPases.
- **Cell Proliferation**: Acts as an inhibitor, which can have implications in cancer biology.
- **Cellular Response to Stress**: As a GTPase, it may play roles in stress responses that influence cellular aging.
- **Nuclear-cytoplasmic Transport**: Its nucleolar presence indicates potential involvement in RNA processing and assembly, impacting gene expression regulation.

### Conclusion
In summary, the RASL10A gene has functional importance in regulating cellular proliferation and may be interlinked with various biological processes, including aging and cancer-related pathways. While specific drug interactions are not fully elucidated, its GTPase activity suggests a potential for pharmacological targeting in relevant contexts. Further research is needed to fully characterize its associations with other genes, aging mechanisms, and therapeutic implications.