### LRRC10 Gene Overview

**Gene Symbol:** LRRC10  
**Function:** Predicted to enable actin binding activity and is associated with cardiac muscle cell development. It is expected to be located in the myofibril, active in the cytoskeleton and sarcomere, indicating its role in cardiac structure and function.

### 1. Associated Genes

While the provided summary does not include specific genes associated with LRRC10, it is important to note that gene interaction networks often include different genes related to muscle function and cardiac development. Some genes commonly studied in this context include:

- **ACTC1** (Actin alpha cardiac muscle 1): Involved in cardiac contractility.
- **MYPBC3** (Myosin-binding protein C, cardiac): Associated with familial hypertrophic cardiomyopathy.
- **TNNT2** (Troponin T type 2): A key regulator of cardiac muscle contraction.
- **MYH7** (Myosin heavy chain 7): Related to the structure of cardiac muscle.

These genes can be interconnected with LRRC10 due to their roles in sarcomere structure and cardiac function.

### 2. Known Details Related to Aging or Age-Related Diseases

While specific age-related studies focusing solely on LRRC10 are limited, it can be inferred that any disruptions in genes involved in cardiac development, structure, or function (like cardiac myofibrils) can lead to aging effects related to heart disease. Aging is associated with changes in cardiac physiology, including increased stiffness, hypertrophy, and other age-related cardiovascular diseases. For instance, altered gene expression may impact the overall efficiency of the cardiac muscle, increasing susceptibility to conditions such as heart failure, cardiomyopathy, or ischemic heart disease.

### 3. Drug Interactions

Currently, there is no specific information about drugs or drug classes that directly interact with LRRC10. However, given its involvement in cardiac function, it may be of interest to investigate the effects of common cardiovascular drugs such as:

- **Beta-blockers:** Used to manage heart conditions, potential impact on gene expression related to cardiac function.
- **ACE inhibitors:** Known for altering the mechanical and structural properties of the heart.

Further research would help elucidate any direct interactions.

### 4. Pathways and Biological Processes

LRRC10 is involved in several biological processes and pathways, including but not limited to:

- **Cardiac muscle contraction:** Key for maintaining heart function.
- **Developmental processes of cardiac muscle cells:** Important during embryogenesis and heart morphogenesis.
- **Cytoskeletal organization:** The role LRRC10 plays in actin binding suggests involvement in maintaining cellular structure.

Additionally, involvement in the **Regulation of Actin Cytoskeleton** may implicate it in broader pathways related to muscle development and function. The study of protein-protein interactions could further reveal relevant pathways where LRRC10 participates in cardiac health and diseases.

### Conclusion

The LRRC10 gene is essential for cardiac function, with predicted roles in muscle development and the maintenance of structural integrity in myofibrils. Exploring its interactions, biological pathways, and implications related to aging and heart diseases will help in comprehensively understanding its importance in cardiovascular health. Further research is necessary for more precise associations with drugs and other genes directly related to this gene.