The ADAM15 gene encodes a member of the ADAM (A Disintegrin And Metalloproteinase) family of proteins, which are characterized by their roles in cell adhesion, proteolytic processing of proteins, and cellular signaling. Below, I address your specific requests related to the ADAM15 gene:

### 1. Associated Genes
While the summaries primarily mention interaction with integrin beta chain, it has also been found to interact with several other genes and proteins, likely reflecting its involvement in broader cellular processes. Some notable associated genes include:
- **ITGA5** (integrin alpha 5): interacts with ADAM15 in integrin-mediated processes.
- **ITGB1** (integrin beta 1): involved in cell adhesion, particularly in airway smooth muscle cells.
- **SRC** (SRC proto-oncogene): a non-receptor tyrosine kinase actively engaged in signaling pathways that interact with ADAM15.
- **E-cadherin** (CDH1): cleavage by ADAM15 impacts cellular adhesion and signaling.
- **ERK1/2** pathway components: involved in ADAM15-mediated cell surface expression and signaling.

### 2. Aging and Age-Related Diseases
ADAM15 has been implicated in several age-related processes and diseases:
- **Wound Healing**: Its role in the wound healing process suggests potential involvement in tissue regeneration, which can be compromised with age.
- **Neovascularization**: ADAM15 plays a part in pathological neovascularization, relevant in conditions like age-related macular degeneration.
- **Cell Migration**: Its effects on cell migration positions it as a factor in tissue remodeling over time, which could contribute to aging-related changes in tissue architecture.
- **Cancer**: Since ADAM15 is involved in cellular signaling pathways, alterations in its function may be linked to age-related increases in cancer incidence, particularly by affecting tumor microenvironments.

### 3. Drug Interaction
Currently, there are no specific drugs that are directly linked to the modulation of ADAM15. However, in the context of cancer and inflammation:
- **Protease Inhibitors**: These might indirectly impact ADAM15's activity by inhibiting its metalloproteinase function.
- **Integrin Targeting Agents**: Some therapies targeting integrin signaling may have downstream effects on ADAM15 interactions, particularly in vascular and fibrotic diseases.
- **Cytokine Modulators**: Given its interaction with cytokines and involvement in cell signaling, certain immunomodulatory drugs could affect ADAM15 function in inflammatory contexts.

### 4. Pathways and Biological Processes
ADAM15 is involved in various biological pathways and processes, including:
- **Cell Adhesion**: Mediates interactions between cells, particularly in immune responses and tissue organization.
- **Proteolysis**: Acts as a metalloproteinase, cleaving various substrates including E-cadherin, which affects cellular adhesion.
- **Cell Signaling**: Interacts with Src kinases and integrins, influencing intracellular signaling pathways critical for cellular responses.
- **Neovascularization**: Implicated in the processes of angiogenesis and tissue remodeling.
- **Wound Healing**: Participates in the dynamic process of wound closure and tissue repair.

In summary, ADAM15 is a multifaceted gene with roles in cell signaling, adhesion, and proteolysis, influencing numerous biological processes relevant to development, injury repair, and possibly age-related conditions. While direct interactions with specific drugs or profound aging associations are still under exploration, it presents potential as a biomarker or therapeutic target in various diseases.