The **PRELID1P4** gene is a pseudogene that belongs to the **PRELID** (pre-baculo-virus replication initiation factor and lipid droplet-associated protein) family. Here is a detailed description based on the information you provided along with additional insights.

### Basic Description
- **Gene Symbol**: PRELID1P4
- **Type**: Pseudogene (non-coding)

### Additional Associated Genes
While the summaries from NCBI and UniProt may not explicitly list other genes associated with PRELID1P4, other genes in the PRELID family, including **PRELID1**, **PRELID2**, and possibly other related lipid metabolism genes, may functionally relate to PRELID1P4 through shared pathways or similar biological functions. 

### Age-Related Details
As of the latest data:
- There is limited direct evidence linking PRELID1P4 to aging or age-related diseases specifically. However, as a pseudogene, it may be involved in regulatory functions that indirectly affect gene expression related to metabolism or cellular stress responses, which are processes often associated with aging.
  
### Drug Interactions
- Currently, there is no specific information available on drugs or drug classes that interact directly with PRELID1P4. As it is a pseudogene, it may not have direct implications for pharmacological interactions typically observed in functional genes.

### Pathways and Biological Processes
- **Biological Role**: Although PRELID1P4 is a pseudogene, members of the PRELID family are involved in lipid metabolism and the biogenesis of lipid droplets, playing a role in cellular homeostasis and metabolism.
- **Pathways**: While specific pathways might not be well-characterized for PRELID1P4 due to its pseudogene status, the related active genes (like PRELID1) have been associated with lipid metabolic processes and potentially mitochondrial dynamics.

### Summary
PRELID1P4 is a pseudogene related to lipid metabolism, which suggests potential indirect influences on processes relevant to metabolic homeostasis and possibly aging, although current information does not connect it directly with age-related diseases or drug interactions. Further research may illuminate its role more clearly in relation to gene regulation and biological processes, particularly as they pertain to its active counterparts in the PRELID family. 

For comprehensive functional analyses, consulting additional databases (such as KEGG, Reactome) and current literature would be necessary as new associations are continually being uncovered in genetic research.