The MSL3 gene (Male-Specific Lethal 3) is a well-studied gene involved in chromatin remodeling and gene regulation, particularly related to dosage compensation on the X chromosome. Below is a detailed overview based on your requests.

### 1. Gene Associations
Besides the MSL3 gene, other genes associated with the MSL complex which may be involved in its regulatory pathways and functions include:
- **MSL1**: Part of the MSL complex, essential for H4K16 acetylation.
- **MSL2**: Another component of the MSL complex, which interacts with the X chromosome.
- **MSL4**: Involved in the dosage compensation complex, working alongside MSL3.
- **MOF**: The histone acetyltransferase that acetyates H4K16, crucial for the activity of the MSL complex.
- **H4**: The specific histone that is acetylated at lysine-16 by the MSL complex.

### 2. Age-Related Details
Research has indicated that chromatin remodeling and gene regulation play significant roles in aging processes and age-related diseases. While specific studies linking MSL3 directly to aging are limited, its role in transcriptional regulation has implications for cellular aging. Disruptions in dosage compensation mechanisms can affect gene expression patterns associated with age-related degeneration and diseases such as:
- **Cancer**: Aberrant expression of genes regulated by MSL3 could influence tumorigenesis.
- **Neurological disorders**: Epigenetic regulation involving MSL3 might affect neurodevelopment and neurodegenerative processes.

### 3. Drug Interactions
Currently, specific drugs or drug classes that directly interact with MSL3 have not been well-established. However, targeting chromatin modifying enzymes, including those that interact with acetylation processes, could indirectly affect MSL3 function. Drug classes that could theoretically influence MSL3 activity include:
- **Histone deacetylase inhibitors (HDAC inhibitors)**: These drugs inhibit the removal of acetyl groups from histones, which may result in altered function of the MSL complex and thereby influence MSL3-related pathways. Examples: Vorinostat, Romidepsin.
- **Selective Estrogen Receptor Modulators (SERMs)**: While not directly linked to MSL3, alterations in chromatin dynamics can have implications in estrogen receptor signaling.

### 4. Pathways and Biological Processes
MSL3 is implicated in several key biological pathways:
- **Dosage compensation**: Balances gene expression between sexes for X-linked genes.
- **Chromatin remodeling**: Involves modifying histones to alter the accessibility of chromatin for transcription.
- **Histone modification**: Specifically involved in the acetylation of histone H4 at lysine-16, contributing to chromatin structure and function.
- **Epigenetic regulation**: Plays a role in the heritable changes in gene expression that do not involve changes to the underlying DNA sequence.
- **X-inactivation**: Contributes to the process by which one of the two X chromosomes in females is transcriptionally silenced.

In summary, MSL3 is a critical player in chromatin dynamics and transcriptional regulation, particularly related to dosage compensation and X-inactivation. Its involvement in various regulatory processes underlines its potential connection to age-related mechanisms and diseases. Research continues to elucidate the broad effects of its activity and regulation.