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This is denoted in the table by " B > T - , P - " , indicating that the B group DHA is not significantly different from the T + and P + group DHA . For example , the expression profiles of tumors reflect the transgenic oncogene [ 5 ] ; moreover , those tumors induced by genes that activate the Ras signaling pathway ( for example , polyoma middle T antigen , ErbB2 / Neu , and H-Ras ) are more similar to each other than to tumors induced by components of the Wnt signaling pathway ( Wnt1 , β - catenin , and c-Myc ) with respect both to gene expression patterns ( [ 5 ] , S. Huang et al ., in preparation ) and to cellular composition and histology [ 6-9 ] . As noted earlier , US-SCAP did not assess patients ' symptomatology at the time of medication initiation , only at predetermined intervals . All bootstrap values > 50 have been reported except in cases where allelic inteins fall within a well-supported ( 95 – 100 % bootstrap ) group , when some values > 50 have been omitted for reasons of space . Treatment group comparisons were based on treatment episodes using 3 statistical approaches ( Kaplan-Meier survival analysis , Cox Proportional Hazards regression model , and propensity score-adjusted bootstrap resampling methods ) . This is indicated by the observation that the B group had significantly greater DHA concentration than the other groups , denoted in the table by " B > all " .