[lemma=mutation] [lemma=breed | tag="," | tag=□] □
[lemma=mutation] [lemma=breed | tag="," | tag=WDT] □
[lemma=mutation] [lemma=breed | tag="," | tag=","] □
The result of an illegal experiment in genetic mutation , Experiment # 626 , is so horrifyingly hostile that it 's been locked up by its inventors . Because of the numerous CFTR mutations it is not clear at this time whether the patients with only a single identified mutation , in fact , have a second unrecognized CFTR mutation or whether heterozygosity for CFTR contributes to susceptibility to pancreatitis . If we were to seize the reigns of genetic mutation , in essence to jump evolution ahead , say by turning off or eliminating genes which cause disease , or adding ones from dogs which will make us cooler , so be it . The molecular biology of GSD IIIa and IIIb is an extremely active area of research ; several quite different mutations , including different types of mutation , in the debrancher gene can produce GSD IIIa.Patients with GSD IIIb are deficient in debrancher activity in the liver but have normal enzyme activity in muscle . Provided : Each GeneCard may include the following information : official gene name and symbol , synonyms or alternative names , ID numbers assigned to the gene in other databases , chromosomal location , chromosome map showing where the gene is found , domains and protein families associated with the gene 's protein product , links to sequence records , expression patterns in human tissues , links to similar genes in other organisms , SNPs and variants , disorders and mutations , links to citations in Medline , and links to other related resources . Three of the patients with one viral load " blip " had sequence data , two of which showed wild-type virus and one that demonstrated the M184V mutation , in which the amino acid valine has been substituted for the wild-type methionine at codon number 184 in the reverse transcriptase gene . The molecular biology of GSD IIIa and IIIb is an extremely active area of research ; several quite different mutations , including different types of mutation , in the debrancher gene can produce GSD IIIa . But this may not be the case after mutation , independently of whether the mutation is performed by setting the string value directly or by changing the Attr child nodes . The most likely reason why the rate of divergence has been quite uniform in all these " radically different " lineages is that cytochrome c does the exact same thing in all these lineages ( it transports electrons in the fundamental cellular process of oxidative phosphorylation ) , and the mutations that do not destroy or change the function of cytochrome c are necessarily neutral .