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In addition , the antibody reacted with nuclei from three of 18 benign prostate hyperplasia ( BPH ) specimens , a non-cancerous enlargement of the prostate ; and with some adjacent , but non-cancerous cells in three of 18 tissue specimens . This " hold-harmless " approach is designed to prevent rate shock and disruption in state rate designs after implementation of the new mechanism . The excess ACTH causes enlargement of both adrenal glands , so in the past surgeons removed both glands to be certain the tumor was completely removed . He added , however , if the UV-B exposure increases any further in Punta Arenas or elsewhere , researchers " should begin long-term surveillance for UV-related disease in humans and animals . Previous studies have shown that a large proportion of psychiatric patients with certain affective disorders , such as depression and manic-depressive disorder , have antibodies specific for proteins of Borna disease virus . In order to prevent disruption in state rate design , the Commission also adopted a " hold-harmless " approach under which the amount of support provided by the new forward-looking mechanism will be no less than the amount of explicit support provided by the existing mechanism . Those patients with no abnormal signs in the emergency department but who turned out to be seriously ill were more likely to have a temperature of 103 degrees or more , as well as a higher white blood cell count , higher respiration rate , higher pulse rate and bacterial infections , according to the results . The two new plans include Local Unlimited , a monthly rate of $ 54.99 , which carries an additional $ 0.05 per kilobyte roaming fee when users are outside of markets where AT & T operates wireless IP service , and National Unlimited , a monthly rate of $ 64.99 with no roaming charges . Hopkins scientist Victor A. McKusick , M.D ., began establishing the clinical definitions of Marfan syndrome in the 1950s , but its molecular roots were not known until the late 1980s when Dietz and other researchers demonstrated that the primary defect was in the gene encoding fibrillin .