Here we investigate the hypothesis that low shear-induced activation of NF-kappaB is responsible for enhanced expression of vascular cell adhesion molecule ( VCAM-1 ) resulting in augmented endothelial cell-monocyte ( EC-Mn ) adhesion and that this activation is dependent on intracellular oxidant activity .
here we investigate the hypothesis that low shear-induced activation of nf-kappab is responsible for enhanced expression of vascular cell adhesion molecule ( vcam-1 ) resulting in augmented endothelial cell-monocyte ( ec-mn ) adhesion and that this activation is dependent on intracellular oxidant activity .
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