We present a model for HIV replication in which the viral protease depletes the infected cells of bcl-2 , leading to oxidative stress-dependent activation of NF kappa B , a cellular factor required for HIV transcription , and ultimately to cell death .
we present a model for hiv replication in which the viral protease depletes the infected cells of bcl-2 , leading to oxidative stress-dependent activation of nf kappa b , a cellular factor required for hiv transcription , and ultimately to cell death .
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