Introduction
post-stroke gait, stroke, gait deficits, gait asymmetries, post-stroke gait biomechanics impairments
STROKE
Hemiparesis following stroke causes unilateral deficits in gait kinematics and kinetics, contributing to slowed gait speed, gait asymmetries, and increased fall risk [Previous studies have demonstrated two major biomechanical gait variables that contribute to overall propulsion, ankle plantarflexor moment, and trailing limb angle [Real-time biofeedback has emerged as a promising post-stroke gait training strategy that can target specific gait deficits on the paretic limb [Here, we studied the effects of TLA biofeedback on post-stroke gait biomechanics. Moreover, to assess its use as a suitable and clinically applicable alternative to AGRF biofeedback, we compared the immediate biomechanical effects of TLA biofeedback to AGRF biofeedback. We hypothesized that a biofeedback paradigm targeting paretic TLA would elicit favorable improvements in paretic propulsion and other post-stroke gait biomechanics impairments that are comparable in magnitude to AGRF biofeedback.