Neutrophil oxidative burst, adhesion molecules, and MPO (Figs. 
trauma
ADHESION
Neutrophil oxidative burst capacity from trauma patients expressed in median fluorescence intensity (MFI). (A) Unstimulated cells. (B) PMA stimulated cells. (C) fMLP stimulated cells. Lines represent the median and interquartile range. Red open circle = Trauma-Sham. Blue open circle = Trauma-RIC. Green open circle = Healthy Controls. Neutrophil cell surface adhesion molecule expression from trauma patients expressed in median fluorescence intensity (MFI). (A) unstimulated CD11b expression. (B) PMA stimulated CD11b expression. (C) fMLP stimulated CD11b expression. (D) unstimulated CD62L expression. (E) PMA stimulated CD62L expression. (F) fMLP stimulated CD62L expression. (G) Plasma levels of Myeloperoxidase (MPO) in trauma patients. Red open circle = Trauma-Sham. Blue open circle = Trauma-RIC. Green open circle = Healthy Controls. Lines represent the median and interquartile range. Unstimulated neutrophil ROS production was lower at Admission in the Sham (P < 0.05) and RIC groups (P = 0.079) and remained significantly lower at 1 and 3 h compared to healthy controls. By 24 h, ROS did not differ from healthy controls (Fig. The cell surface expression of neutrophil CD11b was not significantly different between Sham or RIC-treated trauma patients and healthy controls under unstimulated, PMA, or fMLP conditions (Fig. Cell surface CD62L expression was increased in trauma neutrophils at Admission in both Sham and RIC groups, compared to healthy controls and gradually returned towards levels in healthy control by 24 h post intervention (Fig. Sham-treated trauma patients had a significant increase in MPO (Fig. 