Conclusion
schizophrenia
PATHOLOGY, DYSFUNCTION
Given their genetic basis, their association with schizophrenia and their mechanistic implication suggested by the comparison with the pharmaco-fMRI dataset, we consider the here reported alterations of functional connectivity as potential endophenotypes of NMDAR dysfunction and E/I-imbalance in schizophrenia. These endophenotypes may help to identify patients with a common pathology of this molecular pathway, thus, providing a potential target for a personalized therapy.