DISCUSSION
PAD, peripheral artery disease
PAD, PERIPHERAL ARTERY DISEASE
The current study assessed the acute effects of a mitochondrial targeting antioxidant on metabolic function during a single exercise bout in physically inactive females. Contrary to our hypothesis, MitoQ supplementation did not improve metabolic function during exercise. However, both maximal aerobic exercise capacity (i.e., 
Interestingly, the lower MitoQ has been shown to accumulate within the brain, skeletal muscle, and kidneys (Smith et al., Our current findings have concluded that there was no acute impact of the supplement on submaximal exercise capacity or efficiency, yet there was a significant decrease in maximal exercise capacity. A study analyzing mtROS production and scavenging efficiency at high levels of exercise intensity concluded that maximal exercise performance is linked to the scavenging of antioxidant Exerkines released due to a multitude of redox signaling cascades caused by high mtROS production (Sawada et al., To our knowledge, the acute effect of MitoQ on exercise capacity has only been examined in patients with peripheral artery disease (PAD) (Park et al., 