CONCLUSION
AD, cognitive deficits
CORTEX
In this study, our findings reveal the efficacy of rTMS in the treatment of AD patients at the target of the left angular cortex and further explore altered functional connectivity induced by rTMS using longitudinally measured rs‐fMRI. Our findings suggest that the mechanism by which rTMS ameliorates cognitive deficits is by regulating the restrictive relationships between DMN subsystems and between the large‐scale functional networks associated with the DMN. Furthermore, dynamic regulation of the intra‐ and inter‐DMN at baseline may serve as a potential predictor of favorable rTMS response.