CONCLUSIONS
myocardial damage
MYOCARDIAL DYSFUNCTION
The concentrations and temporal changes of hs‐cTnT, NT‐proBNP, hs‐CRP, and GDF‐15 at the index MI and 1 month later suggest differences in the pathobiology between MINOCA and MI‐CAD. Our data indicate greater initial inflammatory activity, similar degree of myocardial dysfunction, and less pronounced myocardial damage in patients with MINOCA compared with MI‐CAD but also more transient processes in patients with MINOCA. Further analyses are needed to explore the underlying pathophysiological processes and potential implications on patient management.