Conclusion
pain
Our results suggest that cathodal (inhibitory) cerebellar t-DCS increased pain perception and reduced endogenous pain inhibition while anodal (excitatory) t-DCS did the opposite. This is compatible with previous results on cerebellar influences on pain perception, and tentatively supports our hypothesis that the cerebellum may be involved in pain modulation via endogenous, including descending pain pathways. Although effects were small and unlikely to be clinically significant, results nonetheless contribute to the increasing understanding of the interactions between the cerebellum and pain perception, and encourage future studies, preferably using improved methods of cerebellar stimulation.
