Muscle hypertrophy
edema, P., muscle motor units, myosin, muscle enlargement, muscle fatigue
EDEMA, INFILTRATION, CONTRACTION, STRETCHES
Diagnostic musculoskeletal ultrasound can be used as a tool for the rapid and effective measurement of quadriceps circumference (cross-sectional area). In this experiment, musculoskeletal ultrasound was used to measure the sum of the thickness of the rectus femoral muscle and the middle femoris muscle before and after the intervention, as well as to determine the variation in the extensor knee muscle before and after the experimental intervention. After the intervention, the sum of the thickness of rectus femoris and vastus intermedius was improved to a certain extent in the control group, but the difference was not statistically significant. The experimental group showed significant improvements in the rectus femoral and intermediate femoral girths after 8 weeks of intervention. As the level of compression increased, the increase in thickness of rectus femoris and vastus intermedius continued to increase.The expected outcomes of the experiment were that 8 weeks of compression training would increase the patient’s quadriceps thickness and that the level of compression would affect individual circumference values. The experimental results and research of Ryan et al. [Schoenfeld [Metabolic stress is another well-known mechanism of muscle enlargement. It usually occurs when anaerobic glycolysis, a process that occurs when the body uses glycogen faster than oxygen consumption, causes lactic acid and other metabolites to accumulate inside the cell. In the case of resistance training, if the patient receives high-frequency, short-interval training, this mechanism is activated. This is also how BFR training works to promote muscle growth. The increased muscle circumference in the experimental group was associated with the accumulation of metabolites, such as lactate and hydrogen ions, as well as the osmotic effect that promotes the infiltration of water molecules into the cells, resulting in cell edema. This edema stretches the muscle cell membrane, leading to an increase in the thickness of the direct and intermediate femoral muscles.Post-training metabolite accumulation can also cause muscle fatigue by reducing the release of calcium ions from the sarcoplasmic reticulum, as well as reducing the sensitivity of actin and myosin to calcium ions. Changes in calcium ions increase the activity of muscle motor units with higher activation thresholds; these are often associated with larger fibers that produce more tension, so the cell membrane is stretched, thereby increasing the muscle circumference. Compression training allows muscle fatigue, which reduces the rate of contraction, while providing sufficient time to promote bridging formation so that more tension is generated, thereby allowing more cells to be stretched. An increase in metabolic stress, which leads to an increase in mechanical stress, therefore promotes the growth of muscle girth. Experimental results and studies by Jeremy P. Loenneke et al. [