FCH human metabolites limit unbalanced metabolism upon inflammatory stress
To ensure the physiological relevance of our As expected and shown in Fig. 
Primary human chondrocytes were incubated in the presence or absence of rhIL-1b (1ng/ml) in combination with either human naive serum (H-NAIVE) or human serum enriched with metabolites derived from FCH ingestion (H-FCH). Glycosaminoglycans (GAGs) (