Electrophysiological experiments done by Spivak et al. (1980) on frogs showed that anatoxin-a is a potent agonist of the muscle-type (α1)2βγδ nAChR.
Spivak et al.'s (1980) experiments on frogs found that anatoxin-a is a potent activator of the α1 subunit-containing nicotinic acetylcholine receptors in skeletal muscle.