Recently, studies in malaria-endemic areas suggest that placental malaria (PM) may be associated with a dysregulation in angiopoietins. Increased levels of angiopoietin-1 appear to be associated with a decrease in placental weight and placental barrier thickness in women infected with Plasmodium (the causative agent of malaria).
Recent studies in areas where malaria is prevalent propose that placental malaria (PM) could be connected to a disturbance in the regulation of angiopoietins. Elevated levels of angiopoietin-1 are associated with decreased placental weight and increased thickness of the placental barrier in women infected by Plasmodium.