This is due to toxins, extracellular enzymes (exported by the type II secretion system), polysaccharides, lipopolysaccharides, a fatty acid-dependent cell-cell communication system, and proteins (secreted by the type III secretion system), for example. The genes in the bacterial genome that are responsible for such interactions include avirulence (avr) and hypersensitivity response and pathogenicity (hrp) genes.
The presence of toxins, extracellular enzymes, polysaccharides, lipopolysaccharides, a fatty acid-dependent communication system, and proteins is the cause. The bacterial genome contains genes that are responsible for these interactions, including avirulence (avr) and hypersensitivity response and pathogenicity (hrp) genes.