Electrophysiological experiments done by Spivak et al. (1980) on frogs showed that anatoxin-a is a potent agonist of the muscle-type (α1)2βγδ nAChR.
Research conducted by Spivak et al. (1980) in frogs revealed that anatoxin-a acts as a powerful stimulator of the muscle-type (α1)2βγδ nicotinic acetylcholine receptor.