C. albicansis the most prevalent human fungal pathogen, and can be especially dangerous to immunocompromised individuals. One key aspect ofC. albicansvirulence is morphological plasticity.C. albicanscan undergo a number of distinct morphological changes and these changes are controlled by complex transcriptional networks. The transcription factor Ume6 is an important member of these networks, playing an essential role mediating filamentation.C. albicans, however encodes a secondUME6homolog,UME7.UME7is highly conserved in the CTG fungal clade, but the role ofUME7inC. albicansbiology is unknown. Here we truncate and deleteC. albicans UME7. We find Ume7 is dispensable for growth and filamentation. We also find that deletion does not have major consequences on virulence or white opaque switching. Our results suggest that under standard laboratory conditions deletion ofUME7does not have large effects onC. albicansphenotype leaving its role inC. albicansbiology undefined.