The introduction of fake zooms, parallax, sliding and other changes in Apple's new iPhone and iPad software has a very real effect on people with vestibular disorders 
 
 Apple's new mobile operating system for the iPhone and iPad, iOS 7, is stark and minimal, yet dynamic. It makes frequent use of zoom and slide animations; the home screen boasts parallax, with icons apparently floating above subtly animating wallpaper. And it's making people sick. 
 
 Triggers and symptoms vary, but TidePool mobile app developer Jenni Leder's experience is not uncommon. A self-professed power-user, she frequently switches apps; but on iOS 7, this has caused headaches and feelings associated with motion sickness. "I now have to close my eyes or cover the screen during transitions, which is ridiculous," she told The Guardian, adding that there's nowhere to hide: "It's not apps that affect me, but accessing them. Tap a folder and the view zooms in. Tap an app and it's like flying through the icon and landing in that app's micro world — and I'm getting dizzy on the journey there." 
 
 This wasn't the case under iOS 6. That system wasn't devoid of triggers (full-screen slide transitions being fairly common), but zooming was minimal and parallax was absent, as were gamified animation effects such as subtly shifting and sliding balloons in Messages. 
 
 The same minimal effect is true of stock Android and Windows Phone, which lack triggering animations and effects as dynamic and aggressive as those in iOS 7. 
 
 Jump up 
 
 The severity of the jump from iOS 6 to iOS 7 means some organisations dealing with such motion-sickness problems are recommending that people with such conditions don't upgrade their iOS devices. A number of affected users have reportedly switched iPhones that had already been upgraded to iOS 7 for models running iOS 6. 
 
 Reactions to screen-based systems — especially those utilising 3D effects — aren't new. Cynthia Ryan, executive director of the Vestibular Disorders Association, says 3D effects can cause "intense nausea, dizziness and vertigo", sometimes from general vision problems, but also from visual-vestibular conflict. She added symptoms "manifest more severely if a viewer already has a disorder of the vestibular system". 
 
 The vestibular system is what gives us our sense of balance and sense of spatial awareness; it's dependent on three mutually orthogonal fluid-filled canals in the inner ear. But when the vestibular system and visual system come into conflict, the effect can be distressing. 
 
 John Golding, professor of applied psychology at the University of Westminster, says visually-induced motion-sickness often arises from "the induction of perceived self-motion while at the same time the vestibular system and somatosensory systems signal that the body is in fact static". 
 
 The number affected is hard to pin down. Golding says that "visually induced motion sickness" has "affected a relatively small proportion" - less than 5% - of the population, compared with other stimuli such as from boats (sea sickness), cars/coaches (car sickness), etc. But prevalence may be much higher in some clinical grpoups with vestibular disease." 
 
 Ryan says: "These symptoms manifest even more severely if a viewer already has a disorder of the vestibular system. An estimated 69 million US adults aged 40 and over — 35% of all Americans — experience vestibular system dysfunction." 
 
 Dizzying change 
 
 Similar symptoms can also arise from neurological conditions that cause central dizziness. Matt Gemmell, an independent iOS developer, thought it made sense that those with such conditions "would find some parts of the new iOS 7 interface uncomfortable or disorienting," because it "makes more extensive use of animations — and those animations are more pronounced". 
 
 The problem for those suffering is twofold: first, many other people refuse to believe a problem exists; secondly, there's no fix. "We're often contacted by people affected by moving images on screens, but people are affected in different ways — what's a problem for one person may not be for another," explained Natasha Harrington-Benton, director of the Ménière's Society, a UK charity for peopel with disorders causing dizziness or balance disorders. "But these disorders can be extremely debilitating, despite there being no visible symptoms". 
 
 Marissa Christina, a podcaster and writer about hidden disabilities, suggests there was a "lack of awareness" about such issues: "The words 'dizziness' and 'vertigo' don't strike fear into people, but those living with severe cases are in ongoing angst awaiting the next unwarranted attack". 
 
 The lack of a solution is the bigger problem. Apple provides a "Reduce Motion" option within the iOS 7 Settings app, but it is poorly labelled; it merely disables the parallax effect, but doesn't stop zooming or sliding. Apple did not respond to requests for comment for this article. Which for now, leaves affected people on their own. 
 
 Golding's suggestions to those affected include to "rest often", avoid situations which reduce peripheral visual clues which give the correct information the person is static (in other words, don't hold your iPad to your nose), or "just not use the device". Those might suit some, but aren't practical if you're reliant on mobile devices. 
 
 Blind spot 
 
 What's surprising is Apple's apparent blind spot regarding balance and related concerns. Gemmell said: "Apple is more committed to accessibility than any other platform provider I know of." This is borne out by the software's otherwise plentiful aids for people with vision, hearing and motor-control issues. 
 
 However, technology writer Kirk McElhearn quesions whether Apple is ignoring people with vision and balance problems. He told the Guardian: "If Apple wants to truly cater to users with disabilities, it must look more closely at which features cause difficulties, make more effort to listen to users who find them hard to use, and enable them to be more comfortable". 
 
 Christina said Apple should start by acknowledging the issue and "initiating an open dialog with those affected, developers, and Apple's own accessibility team". The company must, she said, "narrow in on what's adversely affecting people and ensure features can be disabled". 
 
 Gemmell says he is "sure suitable options to more comprehensively disable motion will be forthcoming". In the meantime, he recommended those affected send an email to accessibility@apple.com, succinctly stating which animations are problematic and requesting the means to disable them. 
 
 Playing a part 
 
 In the meantime, developers can also do their part. With app creators often following Apple's lead, it's perhaps inevitable the short-term will see yet more animation in iOS 7. Gemmell urges developers to "consider all categories and ability levels of user, and design interfaces judiciously"; they should enable users to disable anything that "could be annoying or intrusive", and UI effects should "have a useful function, such as showing context, hinting about functionality, or demonstrating transition, rather than being purely decorative". 
 
 With the latest update to the popular calculator app PCalc, developer James Thomson has recognised the accessibility concerns, and provided the option to "remove full-screen transitions throughout the app". 
 
 But he says= he'd sooner see Apple add this at system level, :so individual developers don't have to". Gemmell hopes Apple and developers alike would react accordingly regarding animations and transitions, simply because it's the right thing to do: "Accessibility affects everyone, and devices should be usable in all situations, by as many people as possible. Designing for accessibility levels the playing field, and increases the utility of devices for everybody." ||||| A major change in Apple’s iOS 7 update was its sleek, minimal aesthetic; however, the amount of motion now found within is anything but minimal. 
 
 The screen zooms in and out as folders and apps are open and closed; the app switcher zooms and slides; full-screen slide transitions are commonplace; and a faux-3D parallax effect exists on home screens, with icons appearing to float and move independently of background wallpaper. 
 
 For many people (most of Stuff included), this might seem dynamic and exciting. But iOS users with motion-sickness have suddenly found their iPhones and iPads are causing them problems. 
 
 On his blog, The Tech Chat, Mark Coppock wrote that “iOS 7 makes me sick — literally�?, and thought the new animations were “all more aggressive�? than in iOS 6. 
 
 The net result: “It feels to me like the whole screen is moving, and it generates a sort of motion sickness. I feel dizzy and can feel the very beginnings of nausea kicking in.�? ||||| Migraine is one of the most debilitating chronic disorders in the United States. 
 
 It is almost as prevalent as hypertension (high blood pressure) and is more common than asthma and diabetes mellitus. More importantly, migraine strikes people during what are expected to be their most productive years: between ages 20 and 40 for most women, with a slightly higher age range for men. 
 
 Despite better diagnostic capabilities and efforts to improve public awareness and education, it is estimated that approximately 50% of migraineurs go undiagnosed or mismanaged to this day. Many self-treat, or are treated inappropriately for sinus or other non-migrainous types of headache.1 
 
 Often described as “sick headache,” migraine is typically characterized by unilateral onset of head pain, severe progressive intensity of pain, throbbing or pounding, and interference with the person’s routine activities. Accompanying symptoms of photophobia (sensitivity to light) or phonosensitivity (intolerance to noise), as well as nausea and/or vomiting, are common, and often leads to the inability to perform daily tasks. 
 
 Following is a video filmed by Dr. P. Ashley Wackym of Portland, Oregon's Ear & Skull Base Center, showing a patient who has suffered from vestibular migraine. When Dr. Wackym first started seeing this girl she had to be confined to a wheelchair due to her severe symptoms. View this and more videos by Dr. Wackym on his You Tube Channel. 
 
 Migraine and vestibular dysfunction 
 
 Approximately 40% of migraine patients have some accompanying vestibular syndrome involving disruption in their balance and/or dizziness at one time or another. This may be prior to, during, after, or totally independent of their migraine event. Some interesting parallels exist between migraine and non-migrainous vestibular dysfunction. Many of the food and environmental triggers for migraineurs (see box on page 2) are the same as those for patients with non-migrainous vestibular dysfunction. Hormonal fluctuations, foods, and weather changes (barometric-pressure variations) often exacerbate both conditions. Finally, diet modifications and certain medications used in migraine management may ameliorate or prevent the vestibular component of the migraine.2,3 Interestingly enough, some of the analgesic medications for the pain do not resolve the dizziness and medications for the dizziness often do not resolve the painful headache. 
 
 The clinical presentation of vestibular symptoms that often correlate with migraine includes—but is not limited to—dizziness; motion intolerance with respect to head, eyes, and/or body; spontaneous vertigo attacks (often accompanied by nausea and vomiting); diminished eye focus with photosensitivity; sound sensitivity and tinnitus; balance loss and ataxia; cervicalgia (neck pain) with associated muscle spasms in the upper cervical spine musculature; confusion with altered cognition; spatial disorientation; and anxiety/panic.4 
 
 While migraine is often associated with benign recurrent vertigo of adults or paroxysmal vertigo of childhood,5,6,7 some migraine patients also present with true benign paroxysmal positional vertigo (BPPV) even after the migraine headache event has ceased. This is thought to be caused by a combination of vascular events along with an alteration of neural activity associated with the migraine event.8,9 It is believed that these changes more commonly affect the utricle and/or the superior portion of the vestibular nerve and anterior vestibular artery, rather than the saccule and the inferior portion of the vestibular nerve and posterior vestibular artery.10,11 This may explain why results within the normal range are often obtained with vestibular-evoked myogenic potentials (VEMP) testing of migraine patients in the absence of true BPPV. Similarly normal findings have been reported in cases of migraine in the apparent absence of inferior vestibular neuritis, leading to the belief that if inflammation is in fact present as a result of the migraine, and is a cause for utricular BPPV, the local inflammation of the peripheral blood vessels and/or cranial nerve branches is more prevalent in those supplying the utricle rather than the saccule. However, VEMP also now can be helpful in differentiating the clinical presentation of migraine vs Meniere’s syndrome or BPPV. Usually following a migraine event, the VEMP intensity measures are commonly hyperresponsive, whereas with Meniere’s exacerbation the affected ear intensity response is hyporesponsive, and with BPPV the affected ear latency response is typically prolonged. 
 
 Triggers for migraine 
 
 Note: Some of the triggers below may also apply to other types of vestibular dysfunction. 
 
 Food triggers 
 
 Aged or ripened cheeses (examples: Cheddar, Gruyère, Emmenthaler, Stilton, Brie, Gouda, Romano, Parmesan, feta, bleu, Camembert) 
 
 Foods containing large amounts of monosodium glutamate (MSG). Asian foods often have large amounts of MSG. 
 
 Smoked, cured, or processed meats such as bacon, sausage, ham, salami, pepperoni, pickled herring, bologna, chicken livers, and hot dogs 
 
 Food prepared with meat tenderizer, soy sauce, vinegar (except white vinegar), or yeast extract; and food that has been fermented, pickled, or marinated 
 
 Pea pods and pods of broad beans such as lima and navy beans 
 
 Onions, olives, pickles 
 
 Alcohol (especially red wine, port, sherry, Scotch, gin, and bourbon) 
 
 Sour cream, yogurt, buttermilk 
 
 Hot fresh bread, raised coffee cake, doughnuts 
 
 Excessive aspartame (artificial sweetener) 
 
 Chocolate, cocoa, carob 
 
 Nuts, peanut butter 
 
 Certain fruits, including figs, avocados, raisins, red plums, passion fruit, papaya, banana, and citrus fruit 
 
 Excessive tea, coffee, cola 
 
 Other triggers 
 
 Hormonal fluctuations 
 
 Barometric-pressure variations 
 
 Sleep disturbance 
 
 Stress 
 
 Medications 
 
 Parts of this listing are adapted from Ronald J. Tusa, MD, PhD, “Diagnosis and Management of Neuro-otologic Disorders Due to Migraine,” chap. 12 in Vestibular Rehabilitation, ed. Susan J. Herdman, PhD, PT (Philadelphia: F.A. Davis Co., 1994). 
 
 Recognition of migraine syndromes 
 
 Most people associate migraine with severe head pain and a period of incapacitation. However, a large portion of people with migraine often have no accompanying pain, their predominant symptom instead being vertigo (a spinning sensation) or dizziness/ disequilibrium (balance loss), mental confusion, disorientation, dysarthria, visual distortion or altered visual clarity, or extremity paresis. This presentation may result in a visit to the emergency room and extensive laboratory, imaging, and other diagnostic evaluations—often with normal results, which lead to increased confusion and anxiety on the part of the patient. In addition, anti-emetic (anti-vomiting) medications are often given, which may have sedative side effects associated with increased postural instability and increased fall risks. 
 
 Clinicians are faced with the task of attempting to apply objective clinical testing methods to determine the etiology (cause) of a patient’s symptoms so as to optimize treatment. Often, a combination of etiologies exists, which can complicate or confuse the diagnostic process. 
 
 Physicians should be using the International Headache Society’s International Classification of Headache Disorders (2nd edition) in order to better diagnose patients with primary headache disorders. These criteria, used by neurologists and other headache specialists, are readily available in almost every library, either online or in print. 
 
 Migraine headaches (with or without aura), tension-type headache, cluster headache, paroxysmal hemicrania, and chronic daily headache constitute the vast majority of primary headache disorders. Variants of migraine, such as post traumatic headache from concussive injury, exertional migraine and benign orgasmic headaches, are becoming more frequently recognized. These variant presentations may also develop vestibular syndromes that are often more persistent and debilitating than the original headache. 
 
 Mechanisms of migraine 
 
 The emergence of new technologies, such as functional/dynamic imaging studies, has shown promise in documenting the evolution of the migraine processes. As a result, a better understanding of the vascular and neural processes of migraine has been developed. 
 
 The consensus is that the types of headache outlined above—especially migraine/vascular types—are related to a mixed pathophysiology, with cerebral spreading depression of Leão (a spontaneous spreading of an electrical charge along the cortex) followed by activation of pain receptors located in the brainstem, not far from the vestibular apparatus. The release of neurotransmitters then leads to the dilation of blood vessels near the scalp and other structures outside of the brain substance. 
 
 Migraine is also thought to be an inherited disorder giving rise to a “vulnerability” to an abnormal discharge of neurons (different from that seen in epilepsy) that preferentially affects brainstem regions and is triggered by a chemical event.9 
 
 The vascular theory has been long accepted (and is perhaps better understood), which may make it difficult for some practitioners to accept the neural components and associated vestibular manifestations. 
 
 The exact mechanisms of migraine are still not completely understood. But since migraine pathophysiology has been shown to be not solely vascular, and is now thought to be a combination of altered vascular and neural processes, migraine-related vestibulopathy is easier to accept and to treat.12 
 
 Evaluation and testing 
 
 Migraine and its variants must be addressed in the clinical setting by a combination of medical management and comprehensive testing and rehabilitation techniques that offer the most complete and lasting benefit to the patient. 
 
 Traditionally, patients with recurrent vertigo associated with migraine are seen in consultation by neurologists. Otolaryngologists and internists are now becoming more familiar with this condition, but there remains a huge gap between those who care for migraine patients (with or without associated vertigo) and those who have remained “old school”—that is, not recognizing the peripheral and central vestibular components of migraine. 
 
 Patients with migraine associated vertigo (MAV) are often seen by audiologists and vestibular rehabilitation therapists for evaluation and treatment. These paramedical specialists are frequently needed to help the primary care doctor make a diagnosis of MAV. 
 
 After an initial, thorough subjective history is obtained, including a recitation of ongoing symptoms and disruption of activities of daily living, a battery of tests is typically performed, to determine a plan of care for optimized therapy. There are a large number of methods available for testing patients with MAV, and an optimal testing protocol is yet to be determined for this population. Some combination of computerized audiological and vestibular-function tests is typically employed, including positional testing with video-oculography; oculomotor and VOR (vestibulo-ocular reflex) assessments with gaze stability and/or dynamic visual acuity testing; horizontal canal testing with vENG (video electronystagmography), with calorics or rotational chair testing (preferred); audiogram and ABR (auditory brainstem response test); functional balance and gait assessments with CDP (computerized dynamic posturography); and VEMP. 
 
 In our clinic, a review of results obtained from such tests with MAV patients reveals a combination of findings that are attributable to both central processes and peripheral vestibular functions. 
 
 An important component of the evaluation is reliable documentation of the degree of limitation of daily functional capacities. A number of questionnaires and inventories have been employed for this purpose, including the Jacobsen Dizziness Inventory, Dynamic Gait Index, Activities-Specific Balance Confidence Scale, Timed Up and Go test, and others.7,13 
 
 Treatment 
 
 The methodology believed to have the highest efficacy in the management of migraine dizziness is a combination of medications, vestibular rehabilitation, and lifestyle modifications that include limitation of the risk factors associated with migraine (those related to diet, sleep, stress, exercise, and environmental factors). 
 
 Medication 
 
 Medications may be prescribed to prevent migraines or to stop a migraine that has already started. Drugs used to prevent frequent migraine attacks include beta-blockers, tricyclic antidepressants, calcium channel blockers, and certain anticonvulsant medications (Depakote and Topamax). Over the last several years, venlafaxine (Effexor XR) has become one of the favored preventative drug treatments for patients with migraine related vertigo. Drugs commonly used to stop migraine are aspirin, ibuprofen, isometheptene mucate, and the triptans, such as Imitrex and Relpax. Some of these medications work by blocking the action of serotonin (a neurotransmitter that causes large blood vessels to contract) or prostaglandins (a family of chemicals stimulated by estrogen that cause blood vessels to expand and contract).14 Generally the differentiation of whether to use a daily preventive vs an abortive type (taken to stop the already started migraine event) is the frequency and severity of the events. This is best determined by the patient’s discussion of options with the treating Neurologist. 
 
 Vestibular rehabilitation 
 
 The benefits of vestibular rehabilitation are well documented to reduce symptoms and restore function for vestibular-related disorders.7,13 With MAV, it is often helpful for the patient to have started the prescribed medications prior to beginning the vestibular rehabilitation course. This may allow for better tolerance to the exercise regimen without exacerbating the symptoms. The intensity of the rehabilitation course in gradually increased to the patient’s abilities, yet still at a low enough level so as to not initiate another migraine event. 
 
 For patients who have alterations in oculomotor functions and VOR deficits giving rise to visual perceptual dysfunction, a concentrated rehabilitation program consisting of VOR and gaze-stability exercises that emphasize visual acuity is effective. Various eye tracking devices are commercially available which allow the examiner to monitor not only the ability of the patient to visually track objects, but also allow the “method” of eye tracking employed by the patient to be evaluated. Spatial awareness may be altered, and exercises emphasizing proprioception and visual perception are helpful. Isolating visual fields incrementally during visual tracking exercises may be helpful in stabilizing alterations in positional sense. Vestibulo-visual interaction exercises also improve eye tracking abilities. It has become evident that velocity specific exercises are most effective. The velocity of the exercises needs to be matched to the measured velocity deficits on test results. Performing visual retraining exercises at random speeds rather than at specific velocities may be less effective. In cases where BPPV exists, performing canalith repositioning maneuvers is effective, and followed with home habituation exercises. 
 
 Postural instability and gait alterations respond to balance and gait-training tasks and exercises, employing both static and dynamic type balance exercises. Dual tasking and exercises that combine hand-eye coordination, balance maintenance, and gaze stability are effective as well, and can be combined with general conditioning exercises to the extent tolerated by the patient’s general health. Performing exercises on various surface textures and variable stabilities also is recommended. 
 
 In patients with cervicalgia and cervical muscle spasms that limit range of motion, treatment may also include modalities and manual mobilization and stretching of the upper cervical segments, in order to diminish the muscle spasms and guarding and restore normal mobility to the neck. As an adjunct to therapy, greater occipital nerve block (GON) injections are often helpful in reducing symptoms and restoring motion. Some treating MD’s now use Botox for these injections for more lasting effect. 
 
 Lifestyle modifications 
 
 A consistent effort by the patient to adhere to necessary lifestyle modifications (including avoiding the migraine triggers mentioned above), medication usage as prescribed, and specific tasks and exercises performed independently at home are critical to the success of the overall rehabilitation program. Such adherence is essential for effective reduction of the symptoms and limitations of function caused by migraine associated vertigo (MAV).7 
 
 Vestibular test results commonly observed in migraine-related dizziness patients 
 
 During video-oculography, a prevalent feature is poor gaze stability with ocular “drift,” often accompanied by spontaneous up or downbeating directional nystagmus, which does not suppress with fixation-suppression testing added. Unilateral or bilateral gaze induced lateral nystagmus is commonly observed. There may also be a reduced ability to cancel or inhibit the vestibulo-ocular reflex (VOR) function, used for attaining simultaneous head and eye tracking maneuvers. These results may be due to the fact that the cerebellum, which is responsible for coordinating gaze-fixation functions, is thought to be involved in the vascular and neural changes associated with migraine. 
 
 Testing of other cerebellar functions (involving coordinated movements of the extremities) may give normal results, with no postural instability or ataxia/apraxia evident, but postural instability is often evident as well. Smooth pursuit tests often give abnormal results (although these must be distinguished from expected age-related changes). Thus, it may be that only those neural processes of the cerebellum associated with coordinated eye motions are affected in migraine, and not the neural connections involving postural stability. 
 
 Computerized dynamic posturography (CDP) may give positive results for postural instability, especially when used in combination with head motions for dual tasking and otolithic system involvement. Alterations in balance strategies are commonly measured, and need to be addressed with the specific balance exercises in accord with test measures. 
 
 Saccadic eye-motion testing is usually normal, but a rebound nystagmus may be present with hyperresponsive neural findings and presence of overshoot phenomenon. Directional gaze testing is usually abnormal, as is the Halmagyi head thrust test. HIT (head impulse test) may be helpful in documenting the objective findings of VOR and gaze stability deficit. With Hallpike-Dix positional testing (unless true BPPV presents), no rotational component nystagmus is usually evident. However in acute migraine event, bilateral torsional nystagmus may present with positional testing and gaze added. 
 
 With passive VOR assessment via autorotation methods, or with mechanical rotational chair, an abnormal gain value with accompanying phase shift is usually evident. The visual-vestibular interaction can be markedly abnormal and may provoke symptoms of increased dizziness, often with accompanying nausea. Optokinetic after-nystagmus (OKAN) is commonly symmetrically prolonged. Subjective Visual Vertical assessment often is abnormal with accompanying spatial disorientation altered postural positional sense. 
 
 Active autorotation testing, which may be limited by cervicalgia and cervical muscle spasms with limited range of motion (often the patient moves “en bloc” to avoid eliciting dizziness), gives sporadic results. Gaze stability testing and dynamic visual acuity testing—after cervicalgia is resolved with appropriate treatments—are typically abnormal. Vestibular-evoked myogenic potentials (VEMP) testing has proven quite useful in determining differential diagnoses. Regularly, hyperactive VEMP responses are found in patients with MAV. 
 
 Audiometric testing in cases of migraine associated vertigo (MAV) typically reveals no changes in function other than occasional hyperacusis or noise sensitivity, which usually is temporary and resolves shortly after the migraine event ends. Tinnitus (most commonly associated with labyrinthitis rather than migraine), if present at all, is temporary. In cases of prolonged problematic tinnitus, tinnitus retraining therapy (TRT) may be helpful. Tinnitus masking devices are also commercially available. 
 
 Summary 
 
 Migraine associated vertigo (MAV) afflicts a large percent of the population and continues to be a challenge to healthcare professionals. Technologies for measurement continue to expand and new medications continue to be manufactured for this affliction. Effective management of MAV necessitates a comprehensive effort and active participation of the patient, the treating physician, and the rehabilitation professionals. Proper identification, objective diagnostic measurements, and optimized treatment approaches net the best results. 
 
 By Jeffrey Kramer, MD, Chief of Neurology, Mercy Hospital & Medical Center, Chicago, Illinois and Jim Buskirk, PT, SCS, PEAK & Balance Centers of America, Chicago, Illinois 
 
 References 
 
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 Click here to download the "Migrane Associated Vertigo (MAV)" publication.
– Some Apple users are sick of the company's new iOS 7 operating system. Literally. The new zoom and slide animations, and the 'floating" icons on the home screen, are causing motion sickness in many people with vestibular disorders—conditions that affect balance and vision, the Guardian reports. "I now have to close my eyes or cover the screen during transitions, which is ridiculous," says one app developer, who reports headaches and other symptoms associed with motion sickness. "Tap an app and it's like flying through the icon and landing in that app's micro world—and I'm getting dizzy on the journey there." Other symptoms from exposure to this kind of animation can include "intense nausea, dizziness and vertigo," says a spokesperson for the Vestibular Disorders Association. One developer affected by motion sickness has already built an app to disable the offending features, reports Stuff. But others say Apple itself needs to step up. Tech writer Kirk McElhearn says Apple was once "at the forefront of providing accessibility options," but now needs to demonstrate more sensitivity to users like him. "Their apparent disregard is disturbing, and shows a trend toward paying lip service to accessibility, but not thinking it through," he writes on his blog Kirkville.