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And Edited by Noman Islam Nirob.\nPharmacology Viva Question for MBBS Students in Bangladesh\nProfessional MBBS viva Question Board 1\nSubject:pathology (Bacteriology + Virology)\nBranches of microbiology, essential and non essential components of bacteria, functions of capsule, name some capsulated bacteria, differences between gram positive and gram negative, define toxin, define colony, examples of indicator media, Mac Conkey’s agar media te kon bacteria culture kori, cell wall synthesis inhibiting antibiotics, mechanism of penicillin,Corynebacterium diptheriae’r morphology, kon disease kore?\nWindow period, importance, Elaboration of HIV, opportunistic infections in AIDS, complications of mumps.\nSee : Lange Review of Medical Microbiology & Immunology\nProfessional MBBS viva Question Paper 2\nWhat is prokaryotes ; eukaryotes;difference ki?\nNucleoid ki? Antigenic shift;drift; orthomyxo virus family er member ke??segmented virus ki ki?HIV window period r dx.\nProfessional MBBS viva Question Paper 3\nProfessional MBBS viva Question Paper 4\nKi xm dite ascho… microbiology defi..\nBranch of microbiology… primitive nucleus ki…etak ki bole…nucleoid meaning ki… classification of s.aureus…culture finding of s.aureus….\nVirus defi…light microscope e ki ki r dekhte pabo(pox virus and inclusion)… examples of inclusion….Rabies virus shape kmn, structure, transmission, management,ek jaigai ig r vaccine dile ag-ab reaction hobe…\nProfessional MBBS viva Question Paper 5\nKi xm dite asxho , Define bacteria , eukaryote , prokayote,essential structure ,function of them, cell wall component , importance ,spore , steps , importance , gene transfer , sterilization , method ,autoclave details ,antimicrobials , cell wall inhibiting er mechanism ,\nH.ducrei ,chancroid , dx ,culture\nDefine virus , why acellular,morphology/ criteria,difference bet virus bacteria , structure of virus , replication site , step , viral interference with eg , hepatitis B virus er sob , dengue er strain ,\nProfessional MBBS viva Question Paper 6.\nProfessional MBBS viva Question Paper 7\nBacteria-difference btwn prockaryotic and eukaryotic.classification of bacteria.kon bacteria dekte kmon.staphylococcus keno clusster form e thake. Streptococci keno chain form a thake.streptococci er classification. Madam typable nontypable ei classification shunte chaice.streptococcus pyogens er disease gula.\nVirus-interference ki.example. Recrudence ki.antigenic drift,shift.kontar vaccine tairi kora zay na.\nProfessional MBBS viva Question Paper 8\nbranch of microbiology\nphase and elaborately sob.\nBacteria dekhar jonno ki korbo?\nPCR kora hocche ekhn khuv emn ekta disease?\nki dhoroner virus?\nr kon virus pandemic kore?\ncorona virus vaccine ki basis kore banacche?\noncogenic virus kongula?\nHerpes virus kon ta ki kore?\nProfessional MBBS viva Question Paper 9\nMicroorganism def,microscope e ki ki dekha jay,culture media ki ki ase, selective +indicator media er example, kon bacteria culture kora hoy, selective agent r indicator agent ki, lactose fermenter r non fermenter guli ki clr produce kore, kno kore,salmonella shigella proteus alada korbo kivabe,virus er def, virus kivabe ber hoy cell theke (budding r cytolysis),koyta ber hoy(numerous), staphylococcus er diseases,staph cluster r strpto chain kno,measles er complications +kader hoy, mumps complication.\nProfessional MBBS viva Question Paper 10\nKi xm dite ascho?ata ki define kra jay? Branches\nStreptococcus type..strep pneuminia diseases.. Culture..kicabe bujhbo..colony kmn hbe..pyogen er disease, cultute..\nN.gonorrhoea ki krbe..culture..specimen..\nCytopathic effect..ki hbe..multinucleated giant cell kivabe hoy. Kon virus e pabo…inclusion body..type..virus..negri bodies..kon virus e pabo..ata ki disease kre..wound management..r mne nei\nProfessional MBBS viva Question Paper 11\n1.Define sterilization, Disinfectant, Antiseptic.\n2.Classify sterilization. Moist heat দিয়ে sterilize করা হয় এরকম Method এর নাম বলো.\n3.What is Tyndalization.\n4.Salmonella virus নাকি Bacteria😑.কী Disease করে.Lab diagnosis of Enteric fever.\n5.WIDAL test কেন 2nd week এ করবো.Blood culture কেন 1st week এ করবো.\n4.What is virus.Virus এর replication কোথায় হয়.\n5.Type of Influenza virus.কোনটা বেশী Notorious & কেনো.\n6.What is antigenic Shift.What is reassortment.\n7.Replication procedure of Influenza virus.\nProfessional MBBS viva Question Paper 12\ndetonation, prokaryotic, eukaryotic definition+criteria, non essential components of bacteria, flagella in details, sterilization in details, spore defination\nSystemic bacteriology :\nSyphilis ke kore, lab diagnosis, tests, vdrl vs tpha konta kno kora hoy (in details)\nPolio virus (everything)\nMechanism + diagnosis\nProfessional MBBS viva Question Paper 13\n1) Procaryotic cell বলতে কি বোঝো\n2) Bacterial cell এর essential components, periplasmic space এর importance, এখানে থাকে এমন একটা enzyme এর নাম বলো\n3) Culture, media definition. Both selective & indicator হিসাবে কাজ করে কোন media, কোনটা selective হিসাবে কাজ করে, কোনটা indicator হিসাবে কাজ করে, Mac conkey’s agar কি কালার দেবে? কোন গুলা দেবে, Lactose non farmenters এর নাম গুলা, proteus কিভাবে চিনবো( swarming growth শুনতে চায়)\n4) কোন কোন Hepatitis এর ভ্যাক্সিন আছে? Hepa B এর ভ্যাক্সিন নিলে এটা আর কোন hepatitis virus কে prevent করবে? কেনো করবে?\n5) HIV এর diagnosis\n6) Treponema pallidum এর diagnosis, VDRL এর value কি 0 হইতে পারে?\nProfessional MBBS viva Question Paper 14\nculture media ki,aita kore koi rakhbo(incubator e)aita temperature koto,culture koraer por treatment deoar aghe ki korbo(antibiotic sensitivity test),sterilization er defi,tyndalization ki,kun ta te beshi temperature(hot air oven),koto minute dore,aita kise use kora hoi,autoclave kise kora hoi.streptococcus er classification,ki ki diseases kore,post streptococcal disease.\nhuman body te kibabe infect kore,HHP-8 er name ki,kaposis sarcoma kiser sathe rltd,HIV te kokkhan habe,imunnosupression kokhan habe.\nProfessional MBBS viva Question Paper 15\nBacteria component.,different Between gram positive & negetive cell wall,periplasmic space r important,mesosome r function,b lactame r function,cornebacteria diagnostic full.,\nVirus def, virus interference, measles complications 15 yr e konta hoi.,vaccines polio vaccine r dose\n3.bacteria r essential structure\n7.staphylococcus দেখতে কেমন?\n8. Streptococcus ki disease করে,\nRheumatic fever mechanism?\n9.neisseria gonorrhea ki disease? ,\nChildren ki kore?\n10. N gonorrhea lav diagnosis\nWorld wide konta?\nProfessional MBBS viva Question Paper 17\nki xm dite ashcho? microbiology ki, branches, virus defi, rabies virus, dekhte kemon ,type, management of bite,\nbacteria ki, structure, function, dekhte kemon , staph disease.\nma’am akta ans dile link kore onek choto choto ques kortese. maximum i mone nai. but shob i superficial.\nProfessional MBBS viva Question Paper 18\nMicrobiology te use kora jinishpotro kivabe jibanumukto kore, sterilization def, filtration k ki sterilization bola jabe kina( na, karon spore kill korte parena), spore killing procedure gular nam, konta ideal, hot air oven er temperature, kongula sterilize kora jai, media def, selective and indicator both kon media +selectivity maintainer r indicator er nam, lactose fermenting, non fermenting konta ki color dei, shigella salmonellar difference, proteus kamon dekha jabe blood agar e.\nVirus definition, inclusion body def, owl eye appearance er inclusion body kothay pawa jai(CMV), CMV kon group er moddhe pore, Kaposi’s sarcoma kore kon herpes virus, eta kon condition e hoy, opportunistic infection bolte ki bujho, HIV te ki hoy jar jonno opportunistic infection hoy?(CD4 cell count kome) koto kome? (<200/cumm of blood), HIV te ki infection houar por por e ki AIDS hoy?, stages, ARC r symptom gular nam, HIV diagnosis.\n#General bact:defi culture media,,media prepare korar por kothay rakhba, incubator er temp. koto,,Blood culture kivabe hypersensitivity kora hoy?blood culture kise kora hoy? Enteric fever kon bacteria kore? salmonella kon media te culture korba, Mac Conkey’s a kI color dibe,,pale color r kon bacteria dey,,tahole salmonella theke shigella, proteus k alada korba kivabe?\nvirus er structure, envelope kotha theke ase,,replication kothay hoy,,pseudovirus ki,importance ki,,,,viral disease a drug dileo 7din na dileo 7 din kno bola hoy(bolchilam virus decline hoy,,kno kivabe hoy?)\nProfessional MBBS viva Question Paper 19\nKi xam dite ascho? Virus Bacteria microorganism naki na?\nBasic difference between virus and bacteria.\nKonta light microscope e dekha jay konta dekha jay na?\nVirus ki diye dekhe? Light microscope e kon virus dekha jay?\nSymmetry of capsid? Complex kontay thake( Small Pox)\nViral replication e enzymatic protein age hoy naki structural protein? Keno enzymatic protein age hoy?\nSTD transmitted by bacteria ki ki ase?\nTreponema pallidum diagnose kmne korbo? VDRL er importance ki? Eitar value ki zero hoite pare?\nTPHA er value kmn thake(Once positive always positive)\nHow will you diagnose HIV?\nLearn: Pharmacology Viva Question for MBBS Students in Bangladesh\nProfessional MBBS viva Question Paper 20\nStaining ki ki ase,negative staining ki diye kore,kar khetre kore,culture media def,koto temperature e culture kore,culture plate ta ready korar por kothay rakhte hobe (incubator e) ,,culture kora ses hole next step ki,ki korba (antibiotics sensitivity test kore treatment shuru korte hobe),, N.gonorrhoeae er lab diagnosis,kno endocervical swab neya hoy, Herpes virus koyta,kongula carcinoma kore,kaposi’s sarcoma kothay hoy,AIDS er diagnosis.\nProfessional MBBS viva Question Paper 21\nMicrobiology er classification.\nPlasmid ki, function.\nCulture sensitivity test keno kora hoy.\nGrowth er defintion. Growth curve er phase, stationary phase e ki hy.\nIndicator r selective media konta, name of indicator.\nMcconkey media te ki culture korbe.\nMeasles kader hy, complications.\nShingles, ki, ekhane ki hy(nerve pain).\nProfessional MBBS viva Question Paper 22\nKi ki topic er xm dite ashso ei board e…bacterial growth ki..binary fission ki…bacterial growth curve,koita phase,ki ki,log phase er importance,Cell wall acting antibiotic kivabe ekhane kaj kore….bacterial spore er definition,Germination er stage,kon stage e ki hoy…spore forming bacteria…\nStructure of virus,kothai theke envelop toiri hoy,\nAnti viral drugs.\nProfessional MBBS viva Question Paper 23\nAutoclave ar temp, pressure, time, use\nSharp cutting instrument keno autoclaving a kora hoy na\nSporicidal process ar nam..\n*virus- defi, criteria, keno obligate intracellular, structure, envelop ar sathe replication ar ki somporko(budding ar maddome release hoy)\nBudding kothai theke hoy, replication ar steps,\nViral protein at nam, replication ar kon stage a toiri hoy,\nHBV ar type, kn kn hepatitis ar vaccine ache..\nProfessional MBBS viva Question Paper 24\n-What are the essential structures of Bacteria?\n– Bacteria Nucleoid ki diye toiri? (Both DNA & RNA)\n– Virus er nuclear substance?? (Only DNA or RNA)\n– Is the cell wall of Gram Positive and Negative bacteria same in thickness? (Gram positive Thick)\n– Difference between them\n– Periplasmic space a ki thake?? Beta lactamase enzyme ki kore? (Breaks down Beta Lactam Ring of Beta Lactam antibiotics) tahle ki hoy? (Bacterial Drug Resistance)\n– What is Spore??\n– Suitable environment a spore convert hoye ki hoy? (Vegetative state of bacteria)\n– Germination er steps (Activation, Initiation, Out growth)\n-Activation ki ki diye hoy (Heat, Low pH)\n-What are the characteristics of Herpes?\n– Congenital kon gulo kore? (HSV-2, CMV)\n– What is shingle? (Reactivation of Chicken Pox)\n-Kader hoy? (Adult)\n– Keno reactivation hoy? ( Due to latency of HHV)\n– Shingles a ki paoa jay? (Maculo-papular Rash, Nerve pain)\n– Criteria of Nerve Pain? (The pain is so severe that the patient sometimes attempt suicide.. so, Suicidal Pain)\n– Kon Herpes virus kono disease korena?? (HHV-7)\n– HHV-7 er arekta nam ki? (Roseola Virus)\n– What are the staphylococcus?\n– dekhte kmn?? (Spherical.. Gram Positive)\n– ki ki disease kore?\n– Enterotoxin a ki ki disease hoy? (Food poisoning. Diarrhoea & Vomiting)\n-Kon type er vomit hoy? (Projectile Vomiting)\n– What is projectile vomiting? ( Projection akare gastric content ber hoye dure jeye porbe)\n– Keno projectile Vomiting hoy? (Ami Reverse Peristalsis bolechi, but madam answer thik niyechen kina janina. Bolenai kichu ei niye).\nProfessional MBBS viva Question Paper 25\nToxin def, diff b/w exotoxin & endotoxin\nessential components of bacteria, function of mesosome & ribosome, cell wall diff b/w gram positive & negative, spore def, process of germination of spore,\nSexually transmitted disease caused by bacteria, morphology of Treponema (movement ta shunte chay), lab dx of T. pallidum,\nSeromarkers of hepatitis virus, HbeAg er indication ki.\nDefine parasite, classify parasite, entamoeba histolytica’r ki ki form ache, kon ta infective form, extraintestinal lesions kothay kothay hoy, malaria kon mosquito diye hoy, sand fly diye ki hoy, encystation, kothay hoy, name some nematodes.\nCandida ki kore, cells of innate immunity, function of NK cell, extracellular or intracellular killing? Graft ki ki ache? Graft rejection prevent korar jonno ki ki test korbo?\n2nd board Rupom sir..question krsen external sir.\nDefine parasite, classify..then classify protozoa..common sporozoite..plasmodium er life cycle er stage..kon stage e fever hoy.. Common organism of skin infection..candida skin e infect korle ki sample nibo..skin slit kothay nite hoy..\nKalajar…k kore…r ki disease kore…pkdl er stage koita…dx… hypersensitivity classify.. cutaneous mycosis kara kore…kotha theke specimen nibo…oral thrush k kore…kotha theke specimen nibo…\n-name some protozoa\n-Entamoeba histolytica:site of encystation excystation\n-Cells of innate and acquired immunity\n-How to prevent Graft vs host reaction,which drugs r used\n-Tests done before transplantation\n-U cant take a graft from ur parents but cant take from ur siblings…cause in case of parents there is 50% similarity, u need 60% at least for transplant\nWhat is immunity , component of innate immunity,cells , apc ,antigen , immunogen , hapten , alternate pathway of complement activation ,type1 hypersensitivity er eg. cells -ig involved , ag-ab reaction in vitro , eg of agglutination ,\nDefine parasite & protozoa(motile bola jabe na) , classification, infective form of e.histolytica , giardia , t.vaginalis , plasmodium,filaria , hook worm , sexual contact e kon protozoa pass hoy , giardia er morphology , leishmania dx , tissue nematode ki ki ache , filaria dx ,\nMycosis ki , superficial cutaneous sub cutaneous systemic fungi er name , fungus er culture media ,time\nProfessional MBBS examination viva Question Paper 6\nParasite- Kalaazar er vector,kon system er disease. Kon blood cell effect kore.lab diagnosis.\nFilariasis.type,difference. infective form.\nIntestinal cestode er nam.E.granulosus er infective form.hydatid cyst rupture hole ki hobe.anaphylaxis hobe.kon type hypersensitivity\nImmunity-type 1 example.kon cell,immunoglobulin. IgA er type distribution. IgM keno placenta cross korte pare na.\nFungus-opportunistic fungus.oral thrus er lab diagnosis.\nsoil e ache emn protozoa?\nE histolytica and giardia\ner disease and site.\ndhakai kon malaria?\nkon ta kharap?\nrelapse na konta? kno relapse na.\ntenia sagina and solium\ninfective form, indermediate host.\nkonta kharap? kno kharap?\ndefine complement? koto dhoroner ache? complement pathway ki ki ache?\nalternative pathway k activate kore?\nmorphological type of fungus?\nopportunistic fungus name?\nskin srapping microscope e dekhar jonno ki dibo?\nProtozoa def,5ta protozoa r name,trichomonas motile na nonmotile, motile ki diye, infective form ki, kno 1ta form,ki disease, erokom disease kore r kon fungus r bacteria , cestodes ki ki, infective form, fish khle konta hoy, def host r intermediate host ki etar, kon diease kore, kothay thake,hypersensitivity r def, type 1ki ki cell thake +Ig ki?type 2+4 er xmple ,, contact dermatitis kise hoy?\nParasite def..host..protozoa..metazoa..oral imfection kre kara..entamoeba histolylica form..infective konta..kothay disease kre. Ki ki disease..giardia kothay kre..thread worm konta..ki kre..kivabe transmit hoy..kader beshi hoy..kno..cestod ki..tenia solium r saginata konta kharap..kno..larvar form of both..larva ki kre..cystecercosis ki diye hbe..\nSuperficial fungus..ki kre..dimorphic fungus.\nImmunity def..effect gulo..hypersensitivy..cell mediate konta..example.. Complement kothay hoy..pathway..konta age hoy..r kheyal nai,\n1.Define Host,Classify host.\n2.Intermediate host কাকে বলে(Larval stage+ asexual stage).\n3.Man এ asexual stage আছে এরকম ১ টা & larval stage আছে এরকম ১ টা parasite এর নাম বলো.\n4.Malaria এর Asexual stage গুলার নাম বলো.কোন stage এর জন্য Relaps হয়.Relaps হবে না কোনটাতে.\nPlasmodium falciparum কেনো Most dangerous.\n5.Complications of Falciparum Malaria. Types of Pernicious malaria.\n6.E.histolytica এর infective form.কোন route এ entry করে.কোথায় lesions produce করে.Invade করবে কী না.কোন Layer অবধি invade করে.Invade করবে না কিন্তু Lesion করবে কোনটা(G.intestinalis).\n7.Intestine এ পাবা এ রকম Nematode এর নাম বলো.A.Lumbricoides এর Food কী.\n8.Intestine এর কিছু Cestode এর নাম বলো.T.soleum & T.saginata এর মধ্যে কোনটা বেশী Dangerous.\n9.Liver এ Disease করবে এরকম Cestode এর নাম.Hydatid cyst diagnosis এর জন্য কী Liver থেকে Aspiration নিতে পারবো(পারবো না).যদি নেই তাহলে কী হবে.নেওয়া যাবে কোন ক্ষেত্রে(E.histolytica).\n10.Hydatid fluid এর composition.\n1.Fungal disease গুলাকে এক কথায় কী বলবা.কী কী আছে নাম বলো.\n2.Opportunistic infection করে এরকম Fungus এর নাম বলো.কী কী infection করবে.\n4.Opportunistic Infection কখন হয়.Infection ছাড়া কী Immunosuppression হইতে পারে.কখন হবে(Due to Broad spectrum Antibiotics +Corticosteroid).\n4.Moniliasis এর Cause.\n1.Antibody কী কী আছে.\n2.Monomeric IgM কই থাকে.\n3.Immunoglobulin কোথায় তৈরী হয়.\n4.Liver এ তৈরী হয় এরকম কিছু Protein নাম.\n6.Complement কোথায় তৈরী হয়.complement deficiency হইলে কী হবে.\n5.Autoimmunity কখন Develope করবে,immunosuppression হইলে নাকি immunocompetent এ\nParasitology : protozoa r definition, metazoa r classification, intestinal gula r nam kaj infective form, g. Instestainalis in (detalis)\nLeishmania r diseases, diphyllobothrium latum host + site, enterobias varmicularis (detalils)\nMycology: morphological classification, dimorphic er exmple, eta koi thake, ki kore?\nImmunology : all about ig (G,A,M,E,D)\nDistribution, function, defination of immunoglobulin, dfrnc btwn antibody and immunoglobulin.\n1) Define host\n2) মানুষ intermediate host, এমন কিছু parasite এর নাম বলো\n3) Plasmodium এর species গুলার নাম কি?\n4) কোনটা dangerous, কেনো?\n5) Malaria এর দুইটা স্টেজের নাম, আর কি নাম আছে? (shizogony, sporogony শুনতে চায়)\n6) Giardia কি disease করে, কোথায় করে, এটার trophozoite দেখতে কেমন? কয়টা nucleas থাকে? (Both trophozoite & cyst এ)\n7) Diphyllobothrium latum: এটা কি,কি disease করে?\n8) Echinococcus granulosus: কি disease করে, hydatid cyst rupture হলে কি হবে?\n9) Fungus এর morphological classification বলো, Mold এর নাম, Dimorphic fungus এর নাম, কেনো dimorphic বলা হয়, Opportunistic fungus এর নাম\n10) Acquired immunity এর cell গুলার নাম, B cell এর function, NK cell এর function, Ig কয়টা আছে? কোনটা placenta cross করতে পারে?\nprotozoar defination,E.histolytical er excystation kuthai hoi,giardia er encystation kuthai hoi,E.histolytic er infective form.Giardia er infective form,kunta kun intestine e infect kore,amoebic and bacillary dycentry er differance,e.histolytica ki ki disease kore,filariasis ke kore,types,elepanthiasis kun type er,ovo-viviparous ki, kar hoi,hyperinfection er name suncho,oita ki,fish tape worm er scientific name,aita ki kore,kibabe anemia kore.\nsuperficial fungus er name,malasezzia furfur ki kore,(ptyriasis versicolor),aitar lesion ta kmn,laboratory diagnosis,\nhypersensitivity er defination,type 2 ke ki bole,kune immuglobulin diye hoi,example,eryrhobaltosis fetalis e kun ig kore,complement er type,classical e activate kore ke,tolerance er defi,aita valo na kharap,na hale ki hoi,autoimmunity er cause gula\nparasite ki?non motile parasite? basa kothay?\nPlasmodium er species …falciparam er complications?kno beshi dangerous?kalaazar er staining r culture? Ki ki form?patient ki ki presentation niye asbe?rangpur e konta hote pare mosha diye?vector name?kn system er disease kre?infective form?intestinal nematodes?small intestine er cestodes?Mycology:\nMoniliasis k kre? Predisposing factors?oral thrust e ki specimen nibo?staining?\nImmunology: cells of innate immunity?NK cells er function?extracellular na intracellular mechanism?intracellular mechanism e kre kongula?hypersensitivity-def ..type 1 er arek nam ..koy dhoroner..local r systemic er 2 ta kre example..immune response sob..\nHafiza madam(1st board):\nBacteria ki?toxin ki? Ki ki toxin produce kre?endotoxin r exotoxin er difference?clostridium tetani er characteristics….clinical features? Kon mediay culture?\nHepatotrophic viruses?r k k hepatitis kre?hepatitis B er seromarkers?HBcAg kn thake na blood e? HBeAg er indication?\nParasite def,classification, protozoa def,(motile part bola jabe na).,name some protozoa,filaria infective form,dx, sample collection ( night+ day),malaria transmission mosquite bite sara r kise hoi( blood transfusion)..\nComplement koi toiri hoi(liver),alternative pathway r activator,\nSkin fungal infection ( dermatofyte)..\n2.protozoa? (Unicellular eukaryotic bolte bolche sir)\n3. একটা psuedopod movement protozoa name?\nKi disease korbe?\nKoi koi lession korbe?\n4.flazella আছে এমন protozoa\n5.hemoflajelet ki ki ache?\n6.Giardia infective form\nEr trohozoit dkhte kmn?\n7. Cestode ki ki ache\nAnemia knta korbe?\nD.latum kivabe macrocytic kore?\n8.E.granulous D host, I host,\nMnush e ki kore?\nHydatid cyst koi koi hy?\n9. Ascaris infctive form\nKi ki kore?\n10. Mycosis classification\n11.Cutaneous ki ki ache\n12.Dermarophyte ki ki ache?\nSuperficial ki ki?\n14.innate, acquired classify\n15.TT,TIG konta ki?\n17.CD 8+ r function?\nKidney transplant ki graft?\nbasha koi, dhakay ki hoite pare parasitic disease, hydatid cyst ki kore, E. histolytica ki kore, giardia ki kore, structure, koi thake, E. vermicularis koi thake\nHypersensitivity defi, type 2 er example, ki diye hoy, autoimmunity defi, mech, graft rejection , B cell function, secondary immunity er cell,Ig defi ,\nsir 2 rao choto choto besh ques kore. amar ja mone ase tai likhlam r mone porle edit kore dibo.\nKisu protozoa r nam bolo jara disease kore, ami E.histolytica r giardia bolsi, E. Histolytica transmission route( oral bolte hobe), eder moddhe konta kothay disease kore, konta invasive, noninvasive, liver e disease kore emon cestode er nam bolo, ki kore, hydatid cyst rupture hole ki hobe, large intestinal nematode er nam, enterobius er infective form, trasmission route. Morphological classification of fungus, mould er nam, dermatophytes gular nam.\nImmunology: (rupom sir dhorsen)\nHypersensitivity def, type 1 er arekta nam ki, ekhane kon antibody kaj kore, kon cell kaj kore, type 1 er type gular nam ki with examples, primary and secondary mediators of anaphylaxis, functions of NK cell.\n#Netrakona ki ki disease endemic hote pare?(Kala-azar,Malaria)..Durgapur border thakay bolche j kon maleria(hill area) hoy?\n..Falciform malaria kno dangerous?pernicious maleria type,Dx er jonno blood kokhon collect korte hobe?Relapse hoy kon maleria? Rangpur a konta endemic,,Filaria type, route,vector, E.granulosus ki disease kore,,eita ki dhoroner nematode,,hydatid cyst ki kore,,liver er baire ki kore,,complication ki,,Largest nematode er name, eita kothay thake,Eye a kon nematode thake,,\nskin a ki fungal disease hoy,,eitar jonno ki sample niba,(skin scraping), periphery na central theke?kno peripheri theke?\ncomplement activation er koyta pathway,alternate pathway kivabe activate hoy,endoxin dara activate hole ki hobe?\ncomplement deficiency diseases,,Type 1 hypersensitivity cell and Ig,,\nWhat is host?Types of host? Manush kon kon disease e intermediate host thake?\nDhaka te Malaria hoile sheta kon variant hobe? Hilly area te hoile kon variant? Konta beshi dangerous? Kno dangerous? Complication of falciparum malaria? Pernicious malaria er type ki ki?\nKon kon nematode skin penetrate kore dhukbe?\nAncylostoma duodenale er infective form ki?\nVector borne nematode konta? Ki disease kore? Filaria er infective form ki?\nWhat is Mycosis? Types? Cutaneous er example\nTypes of immunity? Types of immune response?\nKontay kon Immunoglobulin thake? Konta beshi time lage? Placenta kon immunoglobulin cross krte pare? Shochey beshi kon immunoglobulin ase? Shobchey boro kon immunoglobulin? Hypersensitivity reaction koy type er? Type 3 er example ki?\nProtozoa def,non motile protozoa ki ki ase,motile kongula, trichomonas er form ki,cyst kno hoy na,basha kothay jiggesh korlo,thn bollo j,dhakay baire giye fuchka khaile kon protozoal infection hoite pare, entamoeba r giardia er ki ki form ase,konta kothay lesion kore,konta invasive,nematodes er nam,rangpur e kon nematode diye disease hoite pare,filaria koy dhoroner,kontay microfilaria blood e paowa jay, specimen kokhn nite hobe,DEC kno khaowano hoy, microfilaria ki blood cell er vitore thake naki baire (baire thake) ,cell er vitore thake emn parasite kongula,kon cell e thake,kala-azar bd er kon kon jaygay beshi paowa jay,ekta female kala-azar er pt ki symptoms niye ashbe\nFungal disease gula ke eksathe ki bole,mycosis er classification,,,ekjon er bronchial asthma ase ,she steroid nicche onk din dhore,tar kon fungal infection hoite pare,,candida infection er predisposing factors ki ki\nComplement activation hoy kivabe, immunity classification,B cell er function,APC kongula,kar kase antigen present kore,kivabe kore, complement deficiency disorder ki ki\nOral route e diseases kore emn protozoa,Entamoeba te ekta amoeba theke koita amebule toiri hoy,cyst e koita nucleus thake,infective form ki,ki ki lesion kore,amoebic and bacillary dysentery er difference…Encystation kothai hoy..\nNematode er nam,large gut e konta paoa jai (Enterobius),Enterobius er egg dekhte kmn,kivabe transmission hoy,Nematode blood e kon cell count barai,Eosinophil ki dhoroner hypersensitivity kore,ekhane r kon cell kaj kore…type 3 hypersensitivity er example…kon Nematode Macrocytic anaemia kore..er intermediate host ki ki…\nYeast like fungus er nam.site of infection of C. albicans…oral thrush e ki sample nibo,Dermatophytosis e skin lesion hole ki specimen nibo.\nProtozoa defi, plasmodium ar species, kn ta dangerous, Keno.. Tartian fever mane ki quartan fever ar mane ki\nCerebral malaria ar pathogenesis, aikhan theke ki hobe,\nAkta nematode ar nam bolo je cerebral disease kore, (echinococcus granulosus)\nSmall cyst k produce kore-( teniea solium)\nLarge cyst k produce kore(echinococcus granulosus, hydratid cyst)\nCerebral disease kore amn fungus ar nam bolo,\nMorphological classification of fungus, common skin disease kore kn fungus..\nAnigen kake bole,\nProtein r lipopolysaccaride antigen ar moddhe knta dangerous\nAntigen body te ki produce kore,\nAntibody kn cell produce kore,\nB cell ar surface a kn immunoglobulin thake,\nMonomaric na pentamaric,\nPentamaric kothai pauya jai.\n– Definiton of Protozoa\n– What are the Common protozoas\n– Basha kothay(Rajshahi).. Rajshahi te tomar kon protozoal disease hote pare (Kala-azar)\n– Vector er nam\n– Oraganism er nam\n– Infective form of the organism (Promastigote)\n– Promastigote a ki flagella thake??\n– kala-azar kon system er disease? (Reticuloendothelial system)\n– Complications of kala-azar\n– akbar valo hoye jaoar por abar hote pare ki? Hole tkhn seitake ki bole\n– PKDL er diagnosis a specimen kotha theke nibo?? (Slit Skin Smear)\n– Kala-azar ki culture kora jay? Kon media? (NNN media)\n-What are the Intestinal Nematodes?\n– Which nematode can cause a common disease in Rangpur? (W. bancrofti – Filariasis)\n– Vector (Culex mosquito)\n– Infective form\n– W. bancrofti generally kothay thake? (Lymphnnode)\n– RBC te ki thake? (Plasmodium)\n– WBC te ki thake? (L. donovani)\n– Filariasis a blood sample kokhn neoa hoy?? (DEC deoar 45min por.. otherwise Raat a from 10PM-4AM in W. bancrofti)\n-What are the types of Hypersensitivity reactions?\n– Type-1 kon cell diye hoy??\n– Complement pathway ki ki ache?? Konta ki diye activate hoy??\nopy and paste this code on your site\nReplace your existing ad code by copying and pasting the code below into the HTML of your site.\n– Complement na thkle ki ki disease hoto??\nHost def, parasite classification, protozoa example,\nPlasmodium life cycle, stages of asexual phase, fever kon stage a hy, ki type fever: intermittent naki relapsing, koto ghonta por por hy,\nFungus morphological classification, example of yeast & yeast like, Crytococcus diye ki ki disease hy, Candida diye ki hy,\nImmunity def, cells of innate immunity, hypersensitivity def, types, function of cytotoxic T cell, eta kon type hypersensitivity kre.\n*define protozoa,name some protozoa\n-infective form,(mature quadrineucleated cyst)\n★name some intestinal nematode\n-er choto vai k(N.americana)\n– ki disease kre\n-ki type er anemia\n-Megalobastic anemia k kre\n-cystode na nematode\n*yeast like knta\n*prolong oral intubation e ki hoy? K kre?\n*Normal vaginal flora gulo ki\n– moniliasis e PH bare na kme?\n★innate immunity cell er nam\n*function of neutrofil,macrophage, B cell,T cell\n* APS cell er name,kake present kre\n*fate of plasma cell\n*autoimmune diseases gulor nam\n*SLE kader beshi hoy\nProfessional MBBS viva Question Paper 26\n-spore k activation k kre\n-spore forming bacteria er nam\n-s/s of tetanus\n-lab dx puro shunte chan\n-special test ki & kno kra hoy\n-why highest state of parasitism\n★Hepa-B er seromarker\nIt can be update as soon as possible.


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Q=100	C=54	SS=0	LS=54	PS=9	DC=0	LC=0	SD=0	BS=1	G0=4	G1=2	G2=1	DLID=bn_Latn	BODY=Golf API Data Feeds Description | Goalserve\nGolf API Data Feeds\nGoalserve Golf Data Feed API provide fixtures, live leaderboard, live statistics, hole-by-hole, golfer profiles, rankings, historical Data since 2010. We provide detailed PGA, LPGA, European coverage. Data availalbe in XML and JSON formats.\nPGA player stats\nPGA past season statistics\nplayer pos="1" name="Tiger Woods" tee_time="" par="-13" hole="68" today="70" po="" scoreToPar="-13" driveDistAvg="294.6" driveAccuracyPct="62.5" gir="80.6" puttsGirAvg="2.107" saves="25" eagles="0" birdies="22" pars="41" bogeys="9" doubles="0" total="275" id="1149">\n<round number="1" result="70"/>\n<round number="2" result="68"/>\n<round number="3" result="67"/>\n<round number="4" result="70"/>\n<hole number="1" par="4" score="4" eagle="False" birdie="False" bogey="False" dbl_bogey_worse="False"/>\n<hole number="2" par="5" score="4" eagle="False" birdie="True" bogey="False" dbl_bogey_worse="False"/>\n<hole number="3" par="4" score="4" eagle="False" birdie="False" bogey="False" dbl_bogey_worse="False"/>\n<hole number="4" par="3" score="3" eagle="False" birdie="False" bogey="False" dbl_bogey_worse="False"/>\n<hole number="5" par="4" score="5" eagle="False" birdie="False" bogey="True" dbl_bogey_worse="False"/>\n<hole number="6" par="3" score="3" eagle="False" birdie="False" bogey="False" dbl_bogey_worse="False"/>\n<hole number="7" par="4" score="4" eagle="False" birdie="False" bogey="False" dbl_bogey_worse="False"/>\n<hole number="8" par="5" score="5" eagle="False" birdie="False" bogey="False" dbl_bogey_worse="False"/>\n<hole number="9" par="4" score="3" eagle="False" birdie="True" bogey="False" dbl_bogey_worse="False"/>\n<hole number="OUT" par="36" score="35" eagle="False" birdie="False" bogey="False" dbl_bogey_worse="False"/>\n<hole number="10" par="4" score="4" eagle="False" birdie="False" bogey="False" dbl_bogey_worse="False"/>\n<hole number="11" par="4" score="4" eagle="False" birdie="False" bogey="False" dbl_bogey_worse="False"/>\n<hole number="12" par="3" score="3" eagle="False" birdie="False" bogey="False" dbl_bogey_worse="False"/>\n<hole number="13" par="5" score="4" eagle="False" birdie="True" bogey="False" dbl_bogey_worse="False"/>\n<hole number="14" par="4" score="3" eagle="False" birdie="True" bogey="False" dbl_bogey_worse="False"/>\n<hole number="15" par="5" score="5" eagle="False" birdie="False" bogey="False" dbl_bogey_worse="False"/>\n<hole number="16" par="3" score="3" eagle="False" birdie="False" bogey="False" dbl_bogey_worse="False"/>\n<hole number="17" par="4" score="5" eagle="False" birdie="False" bogey="True" dbl_bogey_worse="False"/>\n<hole number="18" par="4" score="4" eagle="False" birdie="False" bogey="False" dbl_bogey_worse="False"/>\n<hole number="IN" par="36" score="35" eagle="False" birdie="False" bogey="False" dbl_bogey_worse="False"/>\n<hole number="TOT" par="72" score="70" eagle="False" birdie="False" bogey="False" dbl_bogey_worse="False"/>\n<hole number="2" par="5" score="5" eagle="False" birdie="False" bogey="False" dbl_bogey_worse="False"/>\n<hole number="4" par="3" score="2" eagle="False" birdie="True" bogey="False" dbl_bogey_worse="False"/>\n<hole number="6" par="3" score="2" eagle="False" birdie="True" bogey="False" dbl_bogey_worse="False"/>\n<hole number="8" par="5" score="6" eagle="False" birdie="False" bogey="True" dbl_bogey_worse="False"/>\n<hole number="11" par="4" score="3" eagle="False" birdie="True" bogey="False" dbl_bogey_worse="False"/>\n<hole number="13" par="5" score="5" eagle="False" birdie="False" bogey="False" dbl_bogey_worse="False"/>\n<hole number="15" par="5" score="4" eagle="False" birdie="True" bogey="False" dbl_bogey_worse="False"/>\n<hole number="17" par="4" score="4" eagle="False" birdie="False" bogey="False" dbl_bogey_worse="False"/>\n<hole number="IN" par="36" score="33" eagle="False" birdie="False" bogey="False" dbl_bogey_worse="False"/>\n<hole number="TOT" par="72" score="68" eagle="False" birdie="False" bogey="False" dbl_bogey_worse="False"/>\n<hole number="7" par="4" score="3" eagle="False" birdie="True" bogey="False" dbl_bogey_worse="False"/>\n<hole number="8" par="5" score="4" eagle="False" birdie="True" bogey="False" dbl_bogey_worse="False"/>\n<hole number="9" par="4" score="4" eagle="False" birdie="False" bogey="False" dbl_bogey_worse="False"/>\n<hole number="OUT" par="36" score="34" eagle="False" birdie="False" bogey="False" dbl_bogey_worse="False"/>\n<hole number="14" par="4" score="4" eagle="False" birdie="False" bogey="False" dbl_bogey_worse="False"/>\n<hole number="16" par="3" score="2" eagle="False" birdie="True" bogey="False" dbl_bogey_worse="False"/>\n<hole number="TOT" par="72" score="67" eagle="False" birdie="False" bogey="False" dbl_bogey_worse="False"/>\n<hole number="3" par="4" score="3" eagle="False" birdie="True" bogey="False" dbl_bogey_worse="False"/>\n<hole number="4" par="3" score="4" eagle="False" birdie="False" bogey="True" dbl_bogey_worse="False"/>\n<hole number="10" par="4" score="5" eagle="False" birdie="False" bogey="True" dbl_bogey_worse="False"/>\n<hole number="18" par="4" score="5" eagle="False" birdie="False" bogey="True" dbl_bogey_worse="False"/>\nGolf Feed Coverage\nGoalserve golf data feed coverage include all major series in USA and worldwide. Our golf data feed can be used in different types of applications like livescore, fantasy applications, betting and more. Feed coverage include live score data, live fantasy stats data, bookmaker pregame and in-play odds.


Q=100	C=66	SS=0	LS=66	PS=33	DC=0	LC=0	SD=100	BS=1	G0=1	G1=0	G2=0	DLID=bn_Latn	BODY=Bona Fide Purchasers | Technology Trends\nSome articles on bona fide, bona, bona fide purchaser, bona fide purchasers:\n... and asks the foreign fiancé(e) questions to determine that a bona fide relationship exists ... Consular officer can issue the visa, if he is convinced of a bona fide relationship that meets all legal requirements ... officer does not think the relationship is bona fide or finds some legal impediment to issuing the visa, the petition will be returned to USCIS and recommended for revocation and no visa will be issued ...\nParliamentary Franchise In The United Kingdom 1885–1918 - The Parliamentary Franchise - Counties\n... who actually or bona fide occupy the premises or were seised of such estate on the 7th June, 1832, or have acquired such estate after that day by marriage, marriage settlement, devise, or promotion to a ... unless they have acquired the property by inheritance, marriage, or will, or are bona fide carrying on business thereon as partners, in which case all can ... can be registered, unless they derive the property by inheritance, marriage, or will, or are bona-fide engaged in carrying on business as partners ...\n... passed on trust property is not "equity's darling", the "bona fide purchaser" of the asset ... A bona fide purchaser of property, even if received after a breach of trust, has long been held to take free of any claims by prior owners, provided that they have committed no wrong, and ... If the recipient is not a bona fide purchaser, they must make restitution of the property to the former owner to avoid unjust enrichment ...\nNemo Dat Quod Non Habet - United States\n... In American law, a bona fide purchaser who unknowingly purchases and subsequently sells stolen goods will, at common law, be held liable in trover for the full market value of those goods as ... this is true even in a chain of successive bona fide purchasers (i.e ... the true owner can successfully sue the fifth bona fide purchaser in trover) ...\nFamous quotes containing the words bona fide, purchasers, bona and/or fide:\nTerms related to bona fide purchasers:\nWikipedia Purchasers (Creative Commons)


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