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Dacomitinib Dacomitinib-oersjoch: Hoe NSCLC te behanneljen? - AASraw\nDacomitinib Review: Hoe NSCLC te behanneljen?\n/blog/Gallery/Dacomitinib Review: Hoe NSCLC te behanneljen?\nPosted op 04 / 28 / 2021 by 阿斯劳 skreau yn Gallery.\nFDA keurt dacomitinib goed foar metastatyske longkanker mei net in lytse sel\nWat is net-lytse sel longkanker?\nDacomitinib klinyske tapassing yn net-lytse-sel longkanker\nDacomitinib Mechanisme fan aksje\nSide-effekten fan Dacomitinib\nNet-lytse selle longkankerbehanneling: Dacomitinib VS Gefitinib\nHoe kinne jo Dacomitinib poeder online keapje?\nFDA keurt dacomitinib goed foar metastatyske net-lytse sel longkanker\nOp 27 septimber 2018 goedkarde de Food and Drug Administration dacomitinib-tabletten (VIZIMPRO, Pfizer Pharmaceutical Company) foar de earste line behanneling fan pasjinten mei metastatyk net-lytse sel longkanker (NSCLC) mei epidermale groeifaktorreceptor (EGFR) exon 19 wiskjen as exon 21 L858R-substitúsjemutaasjes lykas ûntdutsen troch in FDA-goedkarde test.\nGoedkarring wie basearre op in randomisearre, multisintrum, iepen-label, aktyf kontroleare proef (ARCHER 1050; NCT01774721) dy't de feiligens en effektiviteit fan dacomitinib fergelike mei gefitinib yn 452 pasjinten mei unresektabele, metastatyske NSCLC. Pasjinten waarden ferplicht gjin foarige terapy te hawwen foar metastatyske sykte of weromkommende sykte mei in minimum fan 12 moannen syktefrij nei ôfrin fan systemyske net-EGFR TKI-befetsjende terapy; in prestaasjestatus fan Eastern Cooperative Oncology Group fan 0 of 1; en EGFR exon 19-wiskjen as exon 21 L858R-substitúsjemutaasjes. Pasjinten waarden randomisearre (1: 1) om ien kear deistich dacomitinib 45 mg oraal te ûntfangen, of gefitinib 250 mg oraal ien kear deistich oant sykteprogresje of ûnakseptabele toksisiteit.\nDe proef toande in wichtige ferbettering yn fuortgongsfrije oerlibjen; gjin ferbettering yn algemiene antwurdrate as algemiene survival waard oantoand. It mediaan fuortgongsfrij oerlibjen, lykas bepaald troch in ûnôfhinklike beoardielingskommisje. wie respektivelik 14.7 en 9.2 moannen yn 'e earms dacomitinib en gefitinib (gefaarferhâlding 0.59; 95% CI: 0.47, 0.74; p <0.0001).\nDe foarskriuwende ynformaasje befettet warskôgingen en foarsoarchsmaatregels foar interstitiale longsykte (ILD), diarree, en dermatologyske neidielige reaksjes. Fan 394 pasjinten dy't dacomitinib krigen, kamen serieuze neidielige reaksjes foar by 27%. De meast foarkommende neidielige reaksjes dy't resulteare yn stopjen fan dacomitinib wiene diarree en ILD. De meast foarkommende (> 20%) neidielige reaksjes fan dacomitinib wiene diarree, útslach, paronychia, stomatitis, fermindere appetit, droege hûd, fermindere gewicht, alopecia, hoest en pruritus).\nLongkanker is de meast foarkommende kanker wrâldwiid, mei mear as twa miljoen nije gefallen wrâldwiid diagnostisearre yn 2018. Sawat 85 prosint fan alle longkankers wurdt identifisearre as net-lytse sel, en sawat 75 prosint dêrfan is metastatysk, of avansearre, by diagnoaze ,\nEgfr is in aaiwyt dat helpt sellen groeie en ferdiele. As it EGFR-gen wurdt mutearre, kin it it aaiwyt feroarsaakje te oerwurkjend, wêrtroch kankersellen ûntsteane. EGFR-mutaasjes kinne wrâldwiid foarkomme yn 10 oant 35 prosint fan NSCLC-tumors, en de meast foarkommende aktivearjende mutaasjes binne wiskjen yn exon 19 en exon 21 L858R-substitúsje, dy't tegearre goed binne foar mear as 80 prosint fan bekende aktivearjende EGFR-mutaasjes. De sykte wurdt assosjeare mei lege oerlibbenssifers en sykteprogresje bliuwt in útdaging.\nAASraw is de profesjonele fabrikant fan Dacomitinib.\nDacomitinib is in EGFR-tyrosine-kinase-ynhibitor (TKI) fan 'e twadde generaasje dy't irreversibel bindet oan en remt EGFR / Her1, Her2 en Her4 subtypen mei in effektiviteit te fergelykjen mei oare TKI's. Yn 'e ARCHER 1050-proef waard fuortgongsfrije oerlibjen ferbettere troch dacomitinib yn ferliking mei gefitinib, stipe dacomitinib as earste behannelingopsje foar avansearre net-lyts-sel longkanker mei gefoelige EGFR-mutaasje. Oangeande it taryf fan hegere neidielige barrens hawwe dosearreduksjes de effektiviteit fan dacomitinib net fermindere en koe de ynfal en hurdens fan adversive barrens effektyf fermindere. Sjoen it evoluerende lânskip fan EGFR-mutante longkanker mei net in lytse sel, soe takomstige kop-oan-kop-ferliking tusken dacomitinib en osimertinib wichtige ynformaasje kinne jaan om it optimale TKI-behannelingskema te bepalen.\nDe hege ynsidinsje fan longkanker fan net-lytse sel hat ús twongen nei te tinken oer behannele opsjes, hoe kinne wy effektive metoaden fine? Nei in protte klinyske stúdzjes, Dacomitinib behanneling sil foar elkenien wurde oanrikkemandearre. Folgjende, litte wy efkes sjen nei Dacomitinib:\nDacomitinib, ûntwurpen as (2E) -N-16-4- (piperidin-1-yl) but-2-enamide, is in mûnling heul selektyf quinazalone diel fan 'e tyrosine kinase-ynhibitoren fan' e twadde generaasje dy't wurde karakterisearre troch de ûnomkearbere bining by it ATP-domein fan 'e epidermale groeifaktor-receptor kinase-domeinen. Dacomitinib is in medikaasje foar de behanneling fan net-lyts-sel longkarsinoom (NSCLC). It is in selektive en ûnomkearbere remmer fan EGFR.\nDacomitinib waard ûntwikkele troch Pfizer Inc en goedkard troch de FDA op 27 septimber 2018. Guon bewiis yn 'e literatuer suggerearje it terapeutyske potensjeel fan dacomitinib yn it epitheliale eierstokkankermodel, hoewol fierdere ûndersiken binne nedich.\nFoar no is Dacomitinib poeder (CAS:1110813-31-4) kin wurde levere troch AASraw út Sina.\nDacomitinib is in ûnomkearbere remmer fan lytse molekulen fan 'e aktiviteit fan' e famylje fan 'e minsklike epidermale groeifaktor (EGFR) famylje (EGFR / HER1, HER2, en HER4) tyrosine kinases. It berikt ûnomkearbere remming fia kovalente bonding oan de cysteïne-residuen yn 'e katalytyske domeinen fan' e HER-receptors. De affiniteit fan dacomitinib is oantoand in IC50 fan 6 nmol / L te hawwen.\nDe famylje ErbB as epidermale groeifaktor (EGF) spilet in rol yn tumorgroei, metastase, en wjerstân tsjin behanneling troch aktivearjende paden fan sinjaltransduksje streamstream lykas Ras-Raf-MAPK, PLCgamma-PKC-NFkB en PI3K / AKT troch de tyrosine kinase-oandreaune fosforylaasje by de carboxy-terminus.1 Rûn 40% fan 'e gefallen toant amplifikaasje fan EGFR-gen en 50% fan' e gefallen presintearje de EGFRvIII-mutaasje dy't in wiskjen fertsjintwurdiget dy't in trochgeande aktivearring produseart fan it tyrosine kinase-domein fan 'e reseptor.\nDacomitinib is goedkard foar behanneling: Net-lytse sel longkanker (NSCLC) dy't metastasearre is (ferspraat nei oare dielen fan it lichem). It wurdt brûkt as earste-line behanneling by pasjinten waans tumors beskate EGFR-genmutaasjes hawwe.\nDacomitinib wurdt ek ûndersocht yn 'e behanneling fan oare soarten kanker.\nWichtige dingen om te ûnthâlden oer de side-effekten fan dacomitinib:\n▪ De measte minsken sille hjir net alle side-effekten fan dacomitinib ûnderfine.\n▪ Dacomitinib-side-effekten binne faaks foarsisber yn termen fan har begjin, doer en hurdens.\n▪ Side-effekten fan Dacomitinib sille ferbetterje neidat de terapy is foltôge.\n▪ Dacomitinib-side-effekten kinne heul beheare. D'r binne in protte opsjes om de side-effekten fan dacomitinib te minimalisearjen of te foarkommen.\nDe folgjende side-effekten binne faak (foarkommen yn mear dan 30%) foar pasjinten dy't dacomitinib nimme:\n▪ Bakteriële of skimmende neilynfeksje (paronychia)\n▪ Droege hûd\n▪ Leech albumine\n▪ Leech kalk\n▪ Hege bloedglukoaze-nivo's\n▪ Ferlytse appetit\n▪ Anemia (leech hemoglobine)\n▪ Leech oantal wite bloedsellen\n▪ Ferhege leverenzymen\nDit binne minder foarkommende side-effekten (foarkomme yn 10-29%) foar pasjinten dy't dacomitinib krije:\nRoodheid, swelling en pine op 'e palmen fan' e hannen en / of de soallen fan 'e fuotten\n▪ Leech kalium-, magnesium- en natriumnivo\n▪ Muskuloskeletale pine\n▪ Swakheid / gebrek oan enerzjy\n▪ Untstekking as ynfeksje fan 'e eagen\n▪ Ferhege serumkreatinine\nHoest, neustekens en symptomen, muoite mei sykheljen en ynfeksje yn 'e boppeste luchtwegen\nNet alle side-effekten wurde hjirboppe neamd. Side-effekten dy't heul seldsum binne - foarkomme yn minder dan sawat 10 persint fan pasjinten - wurde hjir net neamd. Mar jo moatte jo soarchfersekerder altyd ynformearje as jo ungewoane symptomen ûnderfine.\nUnder pasjinten mei EGFR-positive, net-harsemetastatyske net-lytse sel longkanker (NSCLC) ferbetteret earste-line dacomitinib progresjefrije survival (PFS) oer gefitinib, neffens in faze 3-stúdzje publisearre yn The Lancet Oncology.\nEarste generaasje EGFR – tyrosine kinase-ynhibitoren (TKI's), ynklusyf gefitinib, wurde yn 'e earste rigel brûkt foar pasjinten mei EGFR-posysjesykte, dy't tusken 10% en 44% fan alle longadenokarsinomen foarmje. Foarige stúdzje hat net bepaald oft EGFR-TKI's fan 'e twadde generaasje superieur binne oan it ferskaat fan' e earste generaasje.\nFoar dizze open-label, randomisearre stúdzje (ARCHER 1050; ClinicalTrials.gov Identifier: NCT01774721), dy't de auteurs opmurken is de earste fase 3-stúdzje om in EGFR-TKI fan 'e twadde generaasje te fergelykjen mei in EGFR-TKI fan' e earste generaasje yn dizze ynstelling , registrearden ûndersikers 452 pasjinten om dacomitinib te ûntfangen (227 pasjinten) of gefitinib (225 pasjinten). Pasjinten mei harsenmetastasen kamen net yn oanmerking.\nBy in mediaan follow-up fan 22.1 moannen wie mediaan PFS 14.7 moannen foar dacomitinib vs 9.2 moannen foar gefitinib; subgroepanalyses befoardere ek dacomitinib. Tolve folsleine antwurden waarden opnommen yn 'e dacomitinib-groep tsjin 4 yn' e gefitinib-groep. De objektive antwurdraten wiene lykwols gelyk (75% foar dacomitinib en 72% foar gefitinib; P = .4234).\nIenentweintich pasjinten dy't dacomitinib krigen hienen in serieuze behanneling-relatearre negatyf barren (AE); dit wie wier fan 10 pasjinten dy't gefitinib krigen. Behanneling-relatearre deaden waarden rapporteare foar 2 pasjinten dy't dacomitinib krigen tsjin 1 foar gefitinib.\nDe auteurs konkludearren dat "behanneling fan dacomitinib superieur wie gefitinib oangeande [PFS] en doer fan antwurd yn 'e earste-line behanneling fan pasjinten mei EGFR-mutaasje-posityf NSCLC en moat wurde beskôge as in nije behannelingopsje foar dizze populaasje. ”\nD'r binne in soad leveransier / fabrikanten fan dacomitinib poeder yn 'e merk, it finen fan echt is heul wichtich foar alle folken dy't dit produkt driuwend nedich binne. As wy beslute om dacomitinib poeder yn 'e merk te keapjen, moatte wy mear ynformaasje dêre leare, wite hoe't jo it brûke en it is meganisme fan aksje, alle risiko's as wy dacomitinib poeder nimme .... Derneist moatte priis en kwaliteit ús soargen wêze foardat it te keapjen.\nNei't wy gegevens fan 'e merk ûndersochten, in protte leveransiers fergelike, sjocht AASraw in goede kar foar minsken dy't in soad wolle keapje dacomitinib poeder, har produksje waard strikt kontroleare ûnder cGMP-betingst, de kwaliteit kin op elk momint folge wurde en se kinne alle testrapporten leverje as jo it bestelle. Wat de dacomitinib poederkosten / priis is, moat it yn myn eagen ridlik wêze. Om't ik in protte prizen ha fan ferskate leveransiers, yn fergeliking mei kwaliteit, tink ik dat aasraw gjin minne kar sil wêze.\n Kobayashi Y, Fujino T, Nishino M, et al. EGFR T790M en C797S mutaasjes as meganismen fan ferwurven wjerstân tsjin dacomitinib. J Thorac Oncol. 2018; 13 (5): 727-731. doi: 10.1016 / j.jtho.2018.01.009.\n Kris MG, Camidge DR, Giaccone G, et al. Targeting HER2-ôfwikingen as aksjeare bestjoerders yn longkankers: faze II-proef fan 'e pan-HER tyrosine kinase-ynhibitor dacomitinib by pasjinten mei HER2-mutante as fersterke tumors. Ann Oncol. 2015; 26 (7): 1421–1427. doi: 10.1093 / advert / mdv383.\n Park K, Tan EH, O'Byrne K, et al. Afatinib versus gefitinib as earste-line behanneling fan pasjinten mei EGFR-mutaasje-positive net-lytse-sel longkanker (LUX-Lung 7): in faze 2B, iepen-label, randomisearre kontroleare proef. Lancet Oncol. 2016; 17 (5): 577-589. doi: 10.1016 / S1470-2045 (16) 30033-X.\n Lacouture ME, Keefe DM, Sonis S, et al. In faze II-stúdzje (ARCHER 1042) om profylaktyske behanneling fan dacomitinib-induzearre dermatologyske en gastrointestinale neidielige barrens te evaluearjen yn avansearre net-lyts-sel longkanker. Ann Oncol. 2016; 27 (9): 1712–1718. doi: 10.1093 / advert / mdw227.\n Reckamp KL, Giaccone G, Camidge DR, et al. In faze 2-proef fan dacomitinib (PF-00299804), in orale, ûnomkearbere pan-HER (minsklike epidermale groeifaktor-receptor) remmer, by pasjinten mei avansearre net-lytse sel longkanker nei falen fan foarige gemoterapy en erlotinib. Kanker. 2014; 120 (8): 1145–1154. doi: 10.1002 / cncr.28561.\n Engelman JA, Zejnullahu K, Mitsudomi T, et al. MET-amplifikaasje liedt ta ferset tsjin gefitinib yn longkanker troch ERBB3-sinjalearring te aktivearjen. Wittenskip. 2007; 316 (5827): 1039-1043. doi: 10.1126 / science.1141478.\n Liu X, Wang P, Zhang C, et al. Epidermale groeifaktorreceptor (EGFR): In opkommende stjer yn it tiidrek fan presysjemedisy fan longkanker. Oncotarget. 2017; 8 (30): 50209–50220.\n Girard N. Optimalisearjen fan resultaten yn EGFR-mutaasje-positive NSCLC: hokker tyrosine kinase-ynhibitor en wannear? Takomst Oncol 2018.\n Inoue A, Kobayashi K, Maemondo M, et al. Finale resultaten foar algemiene oerlibjen fan NEJ002, in faze III-proef fergelykjen fan gefitinib mei carboplatin (CBDCA) plus paclitaxel (TXL) as earste behanneling foar avansearre net-lytse sel longkanker (NSCLC) mei EGFR mutaasjes. J Clin Oncol 2011; 29: 7519.\n Sequist LV, Yang JC, Yamamoto N, et al. Fase III-stúdzje fan afatinib of cisplatine plus pemetrexed by pasjinten mei metastatysk longadenokarsinoom mei EGFR-mutaasjes. J Clin Oncol 2013; 31: 3327-34.\n Lin JJ, Cardarella S, Lydon CA, et al. Fiifjierrich oerlibjen yn EGFR-mutant metastatysk longadokarcinoma behannele mei EGFR-TKI's. J Thorac Oncol 2016; 11: 556-65.\nGidsynformaasje oer Lenvatinib foar kanker - AASraw Hokker ynformaasje moatte jo witte foardat jo Erlotinib brûke


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20.09.2021 04:31:57\t Autoupdate\nToronto Stock Exchange CAD 19.79 0.00 0.00 512530\nNasdaq other OTC USD 15.51 -0.11 -0.68 3867\nTradegate EUR 13.24 -0.03 -0.19 185\nFrankfurt EUR 13.18 -0.07 -0.53 -\nMÃÂ¼nchen EUR 13.21 0.00 0.00 -\nDÃÂ¼sseldorf EUR 13.20 -0.01 -0.08 -\nBerlin EUR 13.24 -0.04 -0.26 -\nGeld 19.73 Vol. Geld 500\nBrief 19.85 Vol. Brief 2000\nSchlusskurs vom 16.09.2021 19.79 Volatilität in % 23.70\n2021 -9.05% 24.5 18.1\n1 Woche -1.40% 20.2 19.6\n1 Monat -3.32% 20.6 19.5\n3 Monate -2.70% 22.6 19.5\n6 Monate -0.10% 22.6 18.2\n1 Jahr 24.08% 24.5 15.6\n3 Jahre 67.29% 24.5 9.8\nKGV 2020 62.17


Kadowinkel Talant giet ticht | Omrop Fryslân (advertinsje)\nKadowinkel Talant giet ticht 16 jan 2008 - 15:03\nDe kadowinkel fan Talant yn Easterwâlde, de Flambouw, giet dizze simmer ticht. De winkel draait al jierren mei ferlies. De oarsaak is de sterke konkurrinsje fan oare winkels.\nDy ferkeapje guod tsjin prizen dêr't de Flambouw se net iens foar ynkeapje kin. De ôfrûne tiid is oan de hân fan aksjes besocht sluting foar te kommen. Dat die lykwols gjin fertuten. Foar de tsien Talant-meiwurkers yn de de Flambouw wurdt no oar wurk socht.\nKorreksje meldeTrefwurden: argyfLês foarView the discussion thread.Diel dit berjocht op: 16-01Wyldemerkers yn fakânsjehúskes 16-01Bankbus foarsjocht yn ferlet Besjoch ek...


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