syncope
We hypothesize that increased endothelium-mediated vasodilatation may        contribute to hypotension and syncope in patients with left ventricular outflow       obstruction.
We hypothesize that increased endothelium-mediated vasodilatation may        contribute to hypotension and  @SUBJECT$   @PREDICAT$   @OBJECT$  with left ventricular outflow       obstruction.