the more common way that atp is produced in cells is oxidative phosphorylation.
the more common way that atp is produced in cells is oxidative phosphorylation.
the more common way that atp is produced in cells is?
oxidative phosphorylation
glycolysis, which means “sugar splitting,” is the initial process in the cellular respiration pathway. glycolysis can be either an aerobic or anaerobic process. when oxygen is present, glycolysis continues along the aerobic respiration pathway. if oxygen is not present, then atp production is restricted to anaerobic respiration. the location where glycolysis, aerobic or anaerobic, occurs is in the cytosol of the cell. in glycolysis, a six-carbon glucose molecule is split into two three-carbon molecules called pyruvate. these carbon molecules are oxidized into nadh and atp. for the glucose molecule to oxidize into pyruvate, an input of atp molecules is required. this is known as the investment phase, in which a total of two atp molecules are consumed. at the end of glycolysis, the total yield of atp is four molecules, but the net gain is two atp molecules. even though atp is synthesized, the two atp molecules produced are few compared to the second and third pathways, krebs cycle and oxidative phosphorylation.
carbohydrate catabolism
all cells require atp as an energy source for their metabolic activity. the kidney is damaged by anoxia when kidney cortical cells are unable to generate sufficient atp under anaerobic conditions to meet the needs of the cells. when excising a kidney some anoxia is inevitable in the interval between dividing the renal artery and cooling the kidney. it has been shown by bergstrom that 50% of a dog's kidney's cortical cells atp content is lost within 1 minute of clamping the renal artery, and similar results were found by warnick in whole mice kidneys, with a fall in cellular atp by 50% after about 30 seconds of warm anoxia. warnick and bergstrom also showed that cooling the kidney immediately after removal markedly reduced any further atp loss. when these non warm-injured kidneys were perfused with oxygenated hypothermic plasma, atp levels were reduced by 50% after 24-hour storage and, after 48 hours, mean tissue atp levels were a little higher than this indicating that synthesis of atp had occurred. pegg has shown that rabbit kidneys can resynthesize atp after a period of perfusion storage following warm injury, but no resynthesis occurred in non warm-injured kidneys.
machine perfusion