phagocytosis is the process by which phagocytic cells enter the tissues from capillaries.
phagocytosis is the process by which phagocytic cells enter the tissues from capillaries.
is the process by which phagocytic cells enter the tissues from capillaries?
phagocytosis
section::::function.









































section::::function.:phagocytosis.









































half-maximum stimulation is attained at about 100 nm. stimulation of phagocytosis is obtained with polymorphonuclear leukocyte (pmn) cells from human, dog, rabbit and cow as well as with macrophages from the lung and peritoneal cavity of mice, and guinea pig and mouse bone marrow cells. this effect is inhibited by peptide analogue thr-lys-pro-pro-arg. basal activity is not inhibited, so basal phagocytosis may follow a different pathway from that which follows stimulation. stimulation of pinocytosis is exerted only on phagocytic cells, not on cultured cell line mouse leukemia.









































section::::function.:motility and chemotaxis.
tuftsin
aiha can be caused by a number of different classes of antibody, with igg and igm antibodies being the main causative classes. depending on which is involved, the pathology will differ. igg is not very effective at activating complement and effectively binds the fc receptor (fcr) of phagocytic cells, aiha involving igg is generally characterized by phagocytosis of rbcs. igm is a potent activator of the classical complement pathway, thus, aiha involving igm is characterized by complement mediated lysis of rbcs. igm also leads to phagocytosis of rbcs however, because phagocytic cells have receptors for the bound complement (rather than fcrs as in igg aiha). in general, igg aiha takes place in the spleen, whereas igm aiha takes place in kupffer cells – phagocytic cells of the liver. phagocytic aiha is termed extravascular, whereas complement-mediated lysis of rbcs is termed intravascular aiha. in order for intravascular aiha to be recognizable, it requires overwhelming complement activation, therefore most aiha is extravascular – be it igg- or igm-mediated.
autoimmune hemolytic anemia