Once the mutation has been codified and the protein is functional, there is no chance to turn back and we will speak about a mutated individual. However, if the mutant dally is codified but it is not performing its function yet, a chaperone can identify it and try to correct the mutation, or directly send it to a proteasome using ubiquitins and degrade it.
Notwithstanding, there is another possible solution when malformations have occurred as a result of Wg activity loss. Ectopic dally can potentiate Wg signaling but this effect is dependent on some Wg activity remaining at the cell surface. Moreover, ectopic expression of dally+ from hs-dally+ transgene, stimulates Wg signaling. Thus, naked larval cuticle   loss is recuperated and once the larva has become an adult, its tissues execute their normal function. Despite this fact, an intense expression of dally+ results in the death of most of the Drosophila melanogaster’s embryos.